2023 - Research.com Medicine in Canada Leader Award
2020 - Fellow, National Academy of Inventors
His primary areas of study are Prostate cancer, Cancer research, Internal medicine, Endocrinology and Prostate. His Prostate cancer research is multidisciplinary, incorporating perspectives in Androgen and Docetaxel. The various areas that Martin E. Gleave examines in his Cancer research study include Cancer cell, Apoptosis, Clusterin, Paclitaxel and Adenocarcinoma.
His biological study spans a wide range of topics, including Hsp27 and Oncology. The concepts of his Endocrinology study are interwoven with issues in Oblimersen, Tumor progression and Regulation of gene expression. His research integrates issues of Testosterone, Pathology and Urology in his study of Prostate.
Martin E. Gleave mainly investigates Prostate cancer, Cancer research, Internal medicine, Oncology and Cancer. His Prostate cancer study incorporates themes from Androgen and Prostate. Martin E. Gleave interconnects Apoptosis, Clusterin, Gene knockdown, Cancer cell and In vivo in the investigation of issues within Cancer research.
His research in Internal medicine tackles topics such as Endocrinology which are related to areas like Tumor progression and Growth factor. The study incorporates disciplines such as Biopsy, Disease, Pathology, Castration resistant and Docetaxel in addition to Oncology. His LNCaP research is multidisciplinary, incorporating perspectives in Protein kinase B and Cell growth.
Martin E. Gleave mostly deals with Prostate cancer, Cancer research, Internal medicine, Oncology and Cancer. His work investigates the relationship between Prostate cancer and topics such as Prostate that intersect with problems in Surveillance study. His Cancer research study deals with Androgen intersecting with Degarelix.
Internal medicine and Endocrinology are frequently intertwined in his study. His work is dedicated to discovering how Oncology, Disease are connected with Germ cell and other disciplines. His research in Cancer is mostly concerned with LNCaP.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Autophagy (2016)
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Parasites & Vectors (2016)
Androgen Levels Increase by Intratumoral De novo Steroidogenesis during Progression of Castration-Resistant Prostate Cancer
Jennifer A. Locke;Emma S. Guns;Amy A. Lubik;Hans H. Adomat.
Cancer Research (2008)
Molecular Characterization of Neuroendocrine Prostate Cancer and Identification of New Drug Targets
Himisha Beltran;David S. Rickman;Kyung Park;Sung Suk Chae.
Cancer Discovery (2011)
Tumor protein 53-induced nuclear protein 1 expression is repressed by miR-155, and its restoration inhibits pancreatic tumor development.
Meritxell Gironella;Mylène Seux;Min-Jue Xie;Carla Cano.
Proceedings of the National Academy of Sciences of the United States of America (2007)
The androgen receptor fuels prostate cancer by regulating central metabolism and biosynthesis
Charles E Massie;Andy Lynch;Antonio Ramos-Montoya;Joan Boren.
The EMBO Journal (2011)
Intraprostatic androgens and androgen-regulated gene expression persist after testosterone suppression: therapeutic implications for castration-resistant prostate cancer.
Elahe A. Mostaghel;Stephanie T. Page;Daniel W. Lin;Ladan Fazli.
Cancer Research (2007)
Derivation of androgen-independent human LNCaP prostatic cancer cell sublines: Role of bone stromal cells
Hsi‐Chin ‐C Wu;Jer‐Tsong ‐T Hsieh;Martin E. Gleave;Nicholas M. Brown.
International Journal of Cancer (1994)
Acceleration of human prostate cancer growth in vivo by factors produced by prostate and bone fibroblasts.
Martin Gleave;Jer Tsong Hsieh;Chuan Gao;Andrew C. Von Eschenbach.
Cancer Research (1991)
Pten Loss and RAS/MAPK Activation Cooperate to Promote EMT and Metastasis Initiated from Prostate Cancer Stem/Progenitor Cells
David J. Mulholland;Naoko Kobayashi;Marcus Ruscetti;Allen Zhi.
Cancer Research (2012)
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