What is he best known for?
The fields of study he is best known for:
Roger A. Greenberg mostly deals with Molecular biology, Cancer research, Carcinogenesis, Telomere and Telomerase.
His Molecular biology research is multidisciplinary, incorporating elements of Ubiquitin, Gene expression, DNA methylation and Chromatin, DNA.
The various areas that Roger A. Greenberg examines in his Cancer research study include Immunology, Protein kinase B, DNA damage and Cell growth.
His study explores the link between DNA damage and topics such as DNA repair that cross with problems in DNA replication, DNA polymerase and Telomere Homeostasis.
Roger A. Greenberg interconnects Breast cancer and Gene expression profiling in the investigation of issues within Carcinogenesis.
His work deals with themes such as RAD51 and Homologous recombination, which intersect with Telomerase.
His most cited work include:
- RAP80 targets BRCA1 to specific ubiquitin structures at DNA damage sites. (557 citations)
- Network modeling links breast cancer susceptibility and centrosome dysfunction. (531 citations)
- Mitotic progression following DNA damage enables pattern recognition within micronuclei (509 citations)
What are the main themes of his work throughout his whole career to date?
Roger A. Greenberg mainly investigates DNA repair, DNA damage, Cell biology, Cancer research and Genetics.
His DNA repair study combines topics from a wide range of disciplines, such as Chromatin, Molecular biology, Genome instability and Homologous recombination.
Roger A. Greenberg focuses mostly in the field of Molecular biology, narrowing it down to matters related to Telomerase reverse transcriptase and, in some cases, Cell growth.
His research in DNA damage focuses on subjects like Cell cycle checkpoint, which are connected to Immune system.
He has researched Cell biology in several fields, including Cell, Cell cycle, SUMO protein, Ubiquitin and Telomere.
His study in Cancer research is interdisciplinary in nature, drawing from both Cancer, Ovarian cancer, Fanconi anemia, PARP inhibitor and In vivo.
He most often published in these fields:
- DNA repair (43.41%)
- DNA damage (38.76%)
- Cell biology (37.21%)
What were the highlights of his more recent work (between 2018-2021)?
- Cell biology (37.21%)
- DNA repair (43.41%)
- DNA damage (38.76%)
In recent papers he was focusing on the following fields of study:
Roger A. Greenberg spends much of his time researching Cell biology, DNA repair, DNA damage, Cancer cell and Cancer research.
His biological study spans a wide range of topics, including Telomere, Cell cycle and Active site.
His Telomere study combines topics in areas such as SUMO protein and Telomere elongation.
In DNA repair, Roger A. Greenberg works on issues like Homologous recombination, which are connected to CHD1L.
His research integrates issues of Chromatin and Cell cycle checkpoint in his study of DNA damage.
The concepts of his Cancer research study are interwoven with issues in Immune checkpoint, Cancer, Ovarian cancer, Immunotherapy and Drug resistance.
Between 2018 and 2021, his most popular works were:
- Mechanosensing by the Lamina Protects against Nuclear Rupture, DNA Damage, and Cell-Cycle Arrest. (88 citations)
- RAD52 and SLX4 act nonepistatically to ensure telomere stability during alternative telomere lengthening (36 citations)
- Rescue of DNA damage after constricted migration reveals a mechano-regulated threshold for cell cycle. (28 citations)
In his most recent research, the most cited papers focused on:
His primary areas of investigation include Cell biology, DNA repair, DNA damage, Cancer cell and DNA.
His research in Cell biology intersects with topics in Mutation, Interferon, Cytokine and Active site.
His work investigates the relationship between DNA repair and topics such as Telomere that intersect with problems in Telomere Recombination.
His DNA damage research integrates issues from Cell cycle and Lamin.
His work carried out in the field of Cancer cell brings together such families of science as Chromatin, Immunotherapy, SUMO protein, Protein sumoylation and Chemically induced dimerization.
His DNA research incorporates themes from Nuclear protein and Mitosis.
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