Roger A. Greenberg mostly deals with Molecular biology, Cancer research, Carcinogenesis, Telomere and Telomerase. His Molecular biology research is multidisciplinary, incorporating elements of Ubiquitin, Gene expression, DNA methylation and Chromatin, DNA. The various areas that Roger A. Greenberg examines in his Cancer research study include Immunology, Protein kinase B, DNA damage and Cell growth.
His study explores the link between DNA damage and topics such as DNA repair that cross with problems in DNA replication, DNA polymerase and Telomere Homeostasis. Roger A. Greenberg interconnects Breast cancer and Gene expression profiling in the investigation of issues within Carcinogenesis. His work deals with themes such as RAD51 and Homologous recombination, which intersect with Telomerase.
Roger A. Greenberg mainly investigates DNA repair, DNA damage, Cell biology, Cancer research and Genetics. His DNA repair study combines topics from a wide range of disciplines, such as Chromatin, Molecular biology, Genome instability and Homologous recombination. Roger A. Greenberg focuses mostly in the field of Molecular biology, narrowing it down to matters related to Telomerase reverse transcriptase and, in some cases, Cell growth.
His research in DNA damage focuses on subjects like Cell cycle checkpoint, which are connected to Immune system. He has researched Cell biology in several fields, including Cell, Cell cycle, SUMO protein, Ubiquitin and Telomere. His study in Cancer research is interdisciplinary in nature, drawing from both Cancer, Ovarian cancer, Fanconi anemia, PARP inhibitor and In vivo.
Roger A. Greenberg spends much of his time researching Cell biology, DNA repair, DNA damage, Cancer cell and Cancer research. His biological study spans a wide range of topics, including Telomere, Cell cycle and Active site. His Telomere study combines topics in areas such as SUMO protein and Telomere elongation.
In DNA repair, Roger A. Greenberg works on issues like Homologous recombination, which are connected to CHD1L. His research integrates issues of Chromatin and Cell cycle checkpoint in his study of DNA damage. The concepts of his Cancer research study are interwoven with issues in Immune checkpoint, Cancer, Ovarian cancer, Immunotherapy and Drug resistance.
His primary areas of investigation include Cell biology, DNA repair, DNA damage, Cancer cell and DNA. His research in Cell biology intersects with topics in Mutation, Interferon, Cytokine and Active site. His work investigates the relationship between DNA repair and topics such as Telomere that intersect with problems in Telomere Recombination.
His DNA damage research integrates issues from Cell cycle and Lamin. His work carried out in the field of Cancer cell brings together such families of science as Chromatin, Immunotherapy, SUMO protein, Protein sumoylation and Chemically induced dimerization. His DNA research incorporates themes from Nuclear protein and Mitosis.
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Mitotic progression following DNA damage enables pattern recognition within micronuclei
Shane M. Harding;Joseph L. Benci;Jerome Irianto;Dennis E. Discher.
RAP80 targets BRCA1 to specific ubiquitin structures at DNA damage sites.
Bijan Sobhian;Genze Shao;Dana R. Lilli;Aedín C. Culhane.
Network modeling links breast cancer susceptibility and centrosome dysfunction.
Miguel Angel Pujana;Jing Dong J Han;Lea M. Starita;Kristen N. Stevens.
Nature Genetics (2007)
ATM-Dependent Chromatin Changes Silence Transcription In cis to DNA Double-Strand Breaks
Niraj M. Shanbhag;Ilona U. Rafalska-Metcalf;Carlo Balane-Bolivar;Susan M. Janicki.
Telomerase reverse transcriptase gene is a direct target of c-Myc but is not functionally equivalent in cellular transformation
Roger A. Greenberg;Rónán C. O'Hagan;Hongyu Deng;Qiurong Xiao.
HIF-α Effects on c-Myc Distinguish Two Subtypes of Sporadic VHL-Deficient Clear Cell Renal Carcinoma
John D. Gordan;Priti Lal;Vijay R. Dondeti;Richard Letrero.
Cancer Cell (2008)
Short dysfunctional telomeres impair tumorigenesis in the INK4a(delta2/3) cancer-prone mouse.
Roger A. Greenberg;Lynda Chin;Andrea M. Femino;Kee-Ho Lee.
Acetylation limits 53BP1 association with damaged chromatin to promote homologous recombination
Jiangbo Tang;Nam Woo Cho;Gaofeng Cui;Erica M Manion.
Nature Structural & Molecular Biology (2013)
Expression of mouse telomerase reverse transcriptase during development, differentiation and proliferation.
Roger A. Greenberg;Richard C. Allsopp;Richard C. Allsopp;Lynda Chin;Gregg B. Morin.
Constitutive telomerase expression promotes mammary carcinomas in aging mice
Steven E. Artandi;Steven E. Artandi;Scott Alson;Maja K. Tietze;Maja K. Tietze;Norman E. Sharpless.
Proceedings of the National Academy of Sciences of the United States of America (2002)
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