His primary scientific interests are in DNA methylation, Molecular biology, Epigenetics, Cancer research and Methylation. Bernard W. Futscher has included themes like Cancer, Breast cancer and Histone methyltransferase in his DNA methylation study. His Molecular biology research is multidisciplinary, incorporating perspectives in Chromatin, Gene, Mutation and RNA-Directed DNA Methylation.
His research in Epigenetics is mostly focused on Cancer epigenetics. The Cancer research study combines topics in areas such as Tumor suppressor gene and Carcinogenesis. His study looks at the relationship between Methylation and topics such as Gene expression, which overlap with Cell culture, CD19 and Bone marrow.
The scientist’s investigation covers issues in Cancer research, DNA methylation, Molecular biology, Epigenetics and Carcinogenesis. His work carried out in the field of Cancer research brings together such families of science as Chromatin immunoprecipitation, Maspin, Metastasis, Tumor suppressor gene and Gene silencing. His DNA methylation study combines topics in areas such as Chromatin, Cancer and Methylation.
His Molecular biology research includes themes of Cell culture, Gene expression, Gene, DNA and O-6-methylguanine-DNA methyltransferase. Bernard W. Futscher combines subjects such as Malignant transformation, Regulation of gene expression and Histone with his study of Epigenetics. The study incorporates disciplines such as Mutation, Cell cycle, Telomerase and Polymerase chain reaction in addition to Carcinogenesis.
Cancer research, DNA methylation, Cancer, Carcinogenesis and Epigenetics are his primary areas of study. His Cancer research research includes elements of Pancreatic cancer, Pathology, Metastasis, Tumor progression and Gene silencing. His studies in DNA methylation integrate themes in fields like Gene expression profiling, microRNA, Disease and Carcinoma.
His Cancer research incorporates themes from Lung cancer and Methylation, DNA. His study in Epigenetics is interdisciplinary in nature, drawing from both Malignant transformation, Epigenetics of physical exercise and RNA-Directed DNA Methylation. His Gene research is multidisciplinary, incorporating perspectives in Molecular biology and Inflammatory breast cancer.
Bernard W. Futscher focuses on Carcinogenesis, Cancer research, Epigenetics, Gene and DNA methylation. The various areas that Bernard W. Futscher examines in his Carcinogenesis study include Senescence, Cell aging, Small hairpin RNA, Telomerase and Transduction. The concepts of his Cancer research study are interwoven with issues in Tumor Virus, Semaphorin, Lymphangiogenesis, Tumor microenvironment and Transcriptome.
His Epigenetics research includes elements of Promoter, Gene silencing, microRNA and RNA-Directed DNA Methylation. His Gene study frequently draws connections between related disciplines such as Molecular biology. His research integrates issues of Mutation, Point mutation and DNA repair in his study of Molecular biology.
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Role for DNA methylation in the control of cell type specific maspin expression.
Bernard W. Futscher;Marc M. Oshiro;Ryan J. Wozniak;Nicholas Holtan.
Nature Genetics (2002)
P-glycoprotein expression and function in circulating blood cells from normal volunteers
Walter T. Klimecki;Bernard W. Futscher;Thomas M. Grogan;William S. Dalton.
Phase I/II trial of cyclosporine as a chemotherapy-resistance modifier in acute leukemia.
A F List;C Spier;J Greer;S Wolff.
Journal of Clinical Oncology (1993)
Role for DNA methylation in the regulation of miR-200c and miR-141 expression in normal and cancer cells.
Lukas Vrba;Taylor J. Jensen;James C. Garbe;Ronald L. Heimark.
PLOS ONE (2010)
Methylation of discrete regions of the O6-methylguanine DNA methyltransferase (MGMT) CpG island is associated with heterochromatinization of the MGMT transcription start site and silencing of the gene.
George S. Watts;Russell O. Pieper;Joseph F. Costello;Yei Mei Peng.
Molecular and Cellular Biology (1997)
Aberrant methylation of the BRCA1 CpG island promoter is associated with decreased BRCA1 mRNA in sporadic breast cancer cells
Judd C Rice;Kathy S Massey-Brown;Bernard W Futscher.
Methylation of the BRCA1 promoter is associated with decreased BRCA1 mRNA levels in clinical breast cancer specimens
Judd C. Rice;Hilmi Ozcelik;Hilmi Ozcelik;Patrick Maxeiner;Irene Andrulis;Irene Andrulis.
Graded methylation in the promoter and body of the O6-methylguanine DNA methyltransferase (MGMT) gene correlates with MGMT expression in human glioma cells
Joseph F. Costello;Bernard W. Futscher;Keizo Tano;Dawn M. Graunke.
Journal of Biological Chemistry (1994)
Epigenetic silencing of maspin gene expression in human breast cancers.
Frederick E. Domann;Judd C. Rice;Mary J.C. Hendrix;Bernard W. Futscher.
International Journal of Cancer (2000)
5-Aza-2′-deoxycytidine-mediated reductions in G9A histone methyltransferase and histone H3 K9 di-methylation levels are linked to tumor suppressor gene reactivation
R J Wozniak;W T Klimecki;S S Lau;Y Feinstein.
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