Harris Bernstein mainly investigates DNA damage, Oxidative stress, Biochemistry, Apoptosis and Bile acid. His research in DNA damage intersects with topics in Cancer research, Gene expression, DNA repair and Cell biology. Harris Bernstein interconnects Base excision repair, DNA mismatch repair, DNA repair protein XRCC4, Carcinogenesis and Genome instability in the investigation of issues within Cancer research.
Harris Bernstein studied Oxidative stress and Unfolded protein response that intersect with Hepatocyte, Nicotine, Response element and Genotoxic Stress. The Apoptosis study combines topics in areas such as Molecular biology, Reactive oxygen species and Cell culture. The study incorporates disciplines such as Gastroenterology, Cancer, Colorectal cancer and Pathology in addition to Bile acid.
His primary scientific interests are in Genetics, Apoptosis, DNA damage, Molecular biology and Colorectal cancer. His research integrates issues of Oxidative stress, Unfolded protein response and Gene expression in his study of DNA damage. The subject of his Oxidative stress research is within the realm of Biochemistry.
His work in Molecular biology tackles topics such as Thymidine which are related to areas like Escherichia coli. His studies in Colorectal cancer integrate themes in fields like Adenocarcinoma, Cancer research and Pathology. His work in Cancer research addresses subjects such as Small intestine, which are connected to disciplines such as Gastrointestinal cancer.
Harris Bernstein spends much of his time researching Bile acid, Colorectal cancer, Cancer research, Apoptosis and Genetics. His Bile acid research incorporates elements of Oxidative stress, Molecular biology and Secretion. The concepts of his Colorectal cancer study are interwoven with issues in Immunohistochemistry, Carcinogenesis and Carcinogen.
The various areas that Harris Bernstein examines in his Cancer research study include ERCC1 and Small intestine. Apoptosis is closely attributed to Pathology in his research. His biological study spans a wide range of topics, including DNA damage, Genome instability and Bioinformatics.
His primary areas of investigation include Apoptosis, Bile acid, Oxidative stress, Cancer and Cancer research. His Apoptosis research is multidisciplinary, incorporating elements of Cell culture, Western blot, Molecular biology and Pathology. In his study, Mutation, Antioxidant, Endocrinology and Deoxycholic acid is strongly linked to Carcinogen, which falls under the umbrella field of Bile acid.
His DNA damage research extends to Oxidative stress, which is thematically connected. His DNA damage study integrates concerns from other disciplines, such as Endoplasmic reticulum, Gene, DNA repair and Programmed cell death. His Cell biology research includes themes of Caspase and Genetics.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
DNA repair/pro-apoptotic dual-role proteins in five major DNA repair pathways: fail-safe protection against carcinogenesis.
Carol Bernstein;Harris Bernstein;Claire M. Payne;Harinder Garewal.
Mutation Research (2002)
Bile acids as carcinogens in human gastrointestinal cancers.
H. Bernstein;C. Bernstein;C.M. Payne;K. Dvorakova.
Mutation Research (2005)
An E. coli Ribonucleoprotein Containing 4.5S RNA Resembles Mammalian Signal Recognition Particle
Mark A. Poritz;Harris D. Bernstein;Katharina Strub;Dieter Zopf.
Carcinogenicity of deoxycholate, a secondary bile acid.
Carol Bernstein;Hana Holubec;Achyut K. Bhattacharyya;Huy Nguyen.
Archives of Toxicology (2011)
DNA Damage as the Primary Cause of Aging
Helen L. Gensler;Harris Bernstein.
The Quarterly Review of Biology (1981)
Bile acids as endogenous etiologic agents in gastrointestinal cancer
Harris Bernstein;Carol Bernstein;Claire M Payne;Katerina Dvorak.
World Journal of Gastroenterology (2009)
The targeting pathway of Escherichia coli presecretory and integral membrane proteins is specified by the hydrophobicity of the targeting signal
Hin C. Lee;Harris D. Bernstein.
Proceedings of the National Academy of Sciences of the United States of America (2001)
Bile acids in combination with low pH induce oxidative stress and oxidative DNA damage: relevance to the pathogenesis of Barrett’s oesophagus
Katerina Dvorak;Claire M Payne;Melissa Chavarria;Lois Ramsey.
Genetic damage, mutation, and the evolution of sex
Harris Bernstein;Henry C. Byerly;Frederic A. Hopf;Richard E. Michod.
Apoptosis Overview Emphasizing the Role of Oxidative Stress, DNA Damage and Signal- Transduction Pathways
Claire M. Payne;Carol Bernstein;Harris Bernstein.
Leukemia & Lymphoma (1995)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: