D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 46 Citations 13,350 105 World Ranking 3042 National Ranking 111

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Cancer
  • Cytokine

Suppressor of cytokine signaling 1, Immunology, SOCS3, Cell biology and Cytokine are his primary areas of study. His Suppressor of cytokine signaling 1 research is multidisciplinary, incorporating perspectives in Molecular biology, Cancer research and Suppressor of cytokine signalling. His Molecular biology study combines topics from a wide range of disciplines, such as STAT3 and Gene expression.

His Cancer research research is multidisciplinary, relying on both Natural killer T cell, Cytotoxic T cell, IL-2 receptor and Immunoprecipitation. His research investigates the connection between SOCS3 and topics such as STAT4 that intersect with issues in STAT protein and SH2 domain. The concepts of his Cytokine study are interwoven with issues in Programmed cell death and STAT1.

His most cited work include:

  • Structure and function of a new STAT-induced STAT inhibitor (1185 citations)
  • SOCS proteins, cytokine signalling and immune regulation. (1125 citations)
  • The immune responses in CD40-deficient mice: Impaired immunoglobulin class switching and germinal center formation (988 citations)

What are the main themes of his work throughout his whole career to date?

His main research concerns Cancer research, Immunology, Cytokine, Cancer and Molecular biology. Tetsuji Naka combines subjects such as Cell culture, Cell growth, Suppressor of cytokine signaling 1, Cancer cell and Signal transduction with his study of Cancer research. Tetsuji Naka usually deals with Suppressor of cytokine signaling 1 and limits it to topics linked to SOCS3 and STAT protein.

His Immunology study frequently involves adjacent topics like Biomarker. The Cancer study combines topics in areas such as Immunohistochemistry, Oncology and Antigen. His Molecular biology study incorporates themes from In vitro and In vivo.

He most often published in these fields:

  • Cancer research (38.75%)
  • Immunology (29.76%)
  • Cytokine (14.88%)

What were the highlights of his more recent work (between 2016-2021)?

  • Cancer research (38.75%)
  • Cancer (13.84%)
  • Antibody (11.42%)

In recent papers he was focusing on the following fields of study:

Tetsuji Naka mostly deals with Cancer research, Cancer, Antibody, Monoclonal antibody and Biomarker. His Cancer research study integrates concerns from other disciplines, such as Immunohistochemistry, Cell culture, Cell, Cell growth and Receptor. His studies deal with areas such as Molecular biology, Annexin and Janus kinase as well as Cancer.

The study incorporates disciplines such as Inflammation, Immunology, Inflammatory bowel disease and Tuberculosis in addition to Biomarker. He combines Immunology and In patient in his research. Tetsuji Naka interconnects Ubiquitin, Ubiquitin ligase, Cytokine, Cell biology and Interleukin 12 in the investigation of issues within Immune system.

Between 2016 and 2021, his most popular works were:

  • Leucine-rich Alpha-2 glycoprotein is a serum biomarker of mucosal healing in ulcerative colitis (37 citations)
  • Anti-glypican-1 antibody-drug conjugate exhibits potent preclinical antitumor activity against glypican-1 positive uterine cervical cancer. (25 citations)
  • Glypican-1 targeted antibody-based therapy induces preclinical antitumor activity against esophageal squamous cell carcinoma. (24 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Cancer
  • Immune system

Tetsuji Naka focuses on Cancer research, Antibody, Cancer, Monoclonal antibody and Immunology. His Cancer research research includes elements of Cancer cell, Transforming growth factor, Genetic enhancement and In vivo. His Genetic enhancement research incorporates themes from Signal transduction, Janus kinase, Suppressor of cytokine signaling 1, Gene silencing and STAT protein.

His Cancer research incorporates elements of Exome sequencing, Radioresistance, Radiation therapy and STAT3. His research integrates issues of Glutamine and Interstitial lung disease in his study of Immunology. His studies in Receptor integrate themes in fields like Interleukin 12, Ubiquitin ligase, Cytokine and Cell biology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

SOCS proteins, cytokine signalling and immune regulation.

Akihiko Yoshimura;Tetsuji Naka;Masato Kubo.
Nature Reviews Immunology (2007)

1636 Citations

Structure and function of a new STAT-induced STAT inhibitor

Tetsuji Naka;Masashi Narazaki;Moritoshi Hirata;Tomoshige Matsumoto.
Nature (1997)

1582 Citations

The immune responses in CD40-deficient mice: Impaired immunoglobulin class switching and germinal center formation

Tsutomu Kawabe;Tetsuji Naka;Kanji Yoshida;Takashi Tanaka.
Immunity (1994)

1548 Citations

SOCS-1 participates in negative regulation of LPS responses.

Reiko Nakagawa;Tetsuji Naka;Hiroko Tsutsui;Minoru Fujimoto.
Immunity (2002)

732 Citations

The paradigm of IL-6: from basic science to medicine

Tetsuji Naka;Norihiro Nishimoto;Tadamitsu Kishimoto.
Arthritis Research & Therapy (2002)

673 Citations

Aryl hydrocarbon receptor regulates Stat1 activation and participates in the development of Th17 cells

Akihiro Kimura;Tetsuji Naka;Keiko Nohara;Yoshiaki Fujii-Kuriyama.
Proceedings of the National Academy of Sciences of the United States of America (2008)

619 Citations

IL-6-dependent and -independent pathways in the development of interleukin 17-producing T helper cells

Akihiro Kimura;Tetsuji Naka;Tadamitsu Kishimoto.
Proceedings of the National Academy of Sciences of the United States of America (2007)

462 Citations

IL-6 blockade inhibits the induction of myelin antigen-specific Th17 cells and Th1 cells in experimental autoimmune encephalomyelitis.

Satoshi Serada;Minoru Fujimoto;Masahiko Mihara;Nobuo Koike.
Proceedings of the National Academy of Sciences of the United States of America (2008)

425 Citations

Aryl hydrocarbon receptor in combination with Stat1 regulates LPS-induced inflammatory responses

Akihiro Kimura;Tetsuji Naka;Taisuke Nakahama;Ichino Chinen.
Journal of Experimental Medicine (2009)

414 Citations

Accelerated apoptosis of lymphocytes by augmented induction of Bax in SSI-1 (STAT-induced STAT inhibitor-1) deficient mice

Tetsuji Naka;Tomoshige Matsumoto;Masashi Narazaki;Minoru Fujimoto.
Proceedings of the National Academy of Sciences of the United States of America (1998)

345 Citations

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