Suppressor of cytokine signaling 1, Immunology, SOCS3, Cell biology and Cytokine are his primary areas of study. His Suppressor of cytokine signaling 1 research is multidisciplinary, incorporating perspectives in Molecular biology, Cancer research and Suppressor of cytokine signalling. His Molecular biology study combines topics from a wide range of disciplines, such as STAT3 and Gene expression.
His Cancer research research is multidisciplinary, relying on both Natural killer T cell, Cytotoxic T cell, IL-2 receptor and Immunoprecipitation. His research investigates the connection between SOCS3 and topics such as STAT4 that intersect with issues in STAT protein and SH2 domain. The concepts of his Cytokine study are interwoven with issues in Programmed cell death and STAT1.
His main research concerns Cancer research, Immunology, Cytokine, Cancer and Molecular biology. Tetsuji Naka combines subjects such as Cell culture, Cell growth, Suppressor of cytokine signaling 1, Cancer cell and Signal transduction with his study of Cancer research. Tetsuji Naka usually deals with Suppressor of cytokine signaling 1 and limits it to topics linked to SOCS3 and STAT protein.
His Immunology study frequently involves adjacent topics like Biomarker. The Cancer study combines topics in areas such as Immunohistochemistry, Oncology and Antigen. His Molecular biology study incorporates themes from In vitro and In vivo.
Tetsuji Naka mostly deals with Cancer research, Cancer, Antibody, Monoclonal antibody and Biomarker. His Cancer research study integrates concerns from other disciplines, such as Immunohistochemistry, Cell culture, Cell, Cell growth and Receptor. His studies deal with areas such as Molecular biology, Annexin and Janus kinase as well as Cancer.
The study incorporates disciplines such as Inflammation, Immunology, Inflammatory bowel disease and Tuberculosis in addition to Biomarker. He combines Immunology and In patient in his research. Tetsuji Naka interconnects Ubiquitin, Ubiquitin ligase, Cytokine, Cell biology and Interleukin 12 in the investigation of issues within Immune system.
Tetsuji Naka focuses on Cancer research, Antibody, Cancer, Monoclonal antibody and Immunology. His Cancer research research includes elements of Cancer cell, Transforming growth factor, Genetic enhancement and In vivo. His Genetic enhancement research incorporates themes from Signal transduction, Janus kinase, Suppressor of cytokine signaling 1, Gene silencing and STAT protein.
His Cancer research incorporates elements of Exome sequencing, Radioresistance, Radiation therapy and STAT3. His research integrates issues of Glutamine and Interstitial lung disease in his study of Immunology. His studies in Receptor integrate themes in fields like Interleukin 12, Ubiquitin ligase, Cytokine and Cell biology.
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SOCS proteins, cytokine signalling and immune regulation.
Akihiko Yoshimura;Tetsuji Naka;Masato Kubo.
Nature Reviews Immunology (2007)
Structure and function of a new STAT-induced STAT inhibitor
Tetsuji Naka;Masashi Narazaki;Moritoshi Hirata;Tomoshige Matsumoto.
Nature (1997)
The immune responses in CD40-deficient mice: Impaired immunoglobulin class switching and germinal center formation
Tsutomu Kawabe;Tetsuji Naka;Kanji Yoshida;Takashi Tanaka.
Immunity (1994)
SOCS-1 participates in negative regulation of LPS responses.
Reiko Nakagawa;Tetsuji Naka;Hiroko Tsutsui;Minoru Fujimoto.
Immunity (2002)
The paradigm of IL-6: from basic science to medicine
Tetsuji Naka;Norihiro Nishimoto;Tadamitsu Kishimoto.
Arthritis Research & Therapy (2002)
Aryl hydrocarbon receptor regulates Stat1 activation and participates in the development of Th17 cells
Akihiro Kimura;Tetsuji Naka;Keiko Nohara;Yoshiaki Fujii-Kuriyama.
Proceedings of the National Academy of Sciences of the United States of America (2008)
IL-6-dependent and -independent pathways in the development of interleukin 17-producing T helper cells
Akihiro Kimura;Tetsuji Naka;Tadamitsu Kishimoto.
Proceedings of the National Academy of Sciences of the United States of America (2007)
IL-6 blockade inhibits the induction of myelin antigen-specific Th17 cells and Th1 cells in experimental autoimmune encephalomyelitis.
Satoshi Serada;Minoru Fujimoto;Masahiko Mihara;Nobuo Koike.
Proceedings of the National Academy of Sciences of the United States of America (2008)
Aryl hydrocarbon receptor in combination with Stat1 regulates LPS-induced inflammatory responses
Akihiro Kimura;Tetsuji Naka;Taisuke Nakahama;Ichino Chinen.
Journal of Experimental Medicine (2009)
Accelerated apoptosis of lymphocytes by augmented induction of Bax in SSI-1 (STAT-induced STAT inhibitor-1) deficient mice
Tetsuji Naka;Tomoshige Matsumoto;Masashi Narazaki;Minoru Fujimoto.
Proceedings of the National Academy of Sciences of the United States of America (1998)
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