His primary scientific interests are in Internal medicine, Endocrinology, Biochemistry, Lipoprotein and High-density lipoprotein. Alan M. Fogelman has researched Endocrinology in several fields, including Inflammation, Proinflammatory cytokine and Coronary artery disease. His biological study spans a wide range of topics, including Apolipoprotein E, In vivo and Monocyte.
His Monocyte research incorporates elements of Endothelial stem cell, Macrophage colony-stimulating factor, Dose–response relationship, Biological activity and Endothelium. The concepts of his High-density lipoprotein study are interwoven with issues in Lipid oxidation, Statin, Simvastatin, Lipoprotein and Rheumatology. His study in the field of Apolipoprotein B is also linked to topics like Ultra rapid freezing.
His main research concerns Internal medicine, Endocrinology, Biochemistry, Apolipoprotein B and Lipoprotein. His is doing research in Cholesterol, High-density lipoprotein, LDL receptor, Monocyte and Paraoxonase, both of which are found in Internal medicine. In his study, Endothelial stem cell is strongly linked to Endothelium, which falls under the umbrella field of Monocyte.
His work carried out in the field of Endocrinology brings together such families of science as Lesion, Inflammation, Proinflammatory cytokine, Serum amyloid A and Apolipoprotein E. The Inflammation study combines topics in areas such as Oxidative stress and Small intestine. Alan M. Fogelman interconnects Molecular biology and In vivo in the investigation of issues within Biochemistry.
Alan M. Fogelman mostly deals with Internal medicine, Endocrinology, Inflammation, Apolipoprotein B and Lipoprotein. His Internal medicine research includes elements of Peptide mimetic and Hydroxyeicosatetraenoic acid. His Endocrinology research is multidisciplinary, relying on both Serum amyloid A, Lysophosphatidic acid and Oxidative phosphorylation.
His study in Inflammation is interdisciplinary in nature, drawing from both Tumor necrosis factor alpha, Small intestine, Inflammatory bowel disease and Mechanism of action. Lipoprotein is the subject of his research, which falls under Biochemistry. He works mostly in the field of Biochemistry, limiting it down to concerns involving In vivo and, occasionally, Angiogenesis.
His primary areas of investigation include Internal medicine, Endocrinology, Apolipoprotein B, Inflammation and Lipoprotein. His research combines Cell growth and Internal medicine. His research in Endocrinology intersects with topics in Serum amyloid A and Lysophosphatidic acid.
His Apolipoprotein B research integrates issues from Hypoxia, Inflammatory bowel disease, Chromosomal translocation, Myeloid and Computational biology. The study incorporates disciplines such as Cyclooxygenase, Barrier function and Intestinal epithelium in addition to Inflammation. His research integrates issues of Reactive oxygen species, Hemopexin, Molecular biology, Pharmacodynamics and Apolipoprotein E in his study of Lipoprotein.
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Atherosclerosis: basic mechanisms. Oxidation, inflammation, and genetics.
Judith A. Berliner;Mohamad Navab;Alan M. Fogelman;Joy S. Frank.
Minimally modified low density lipoprotein induces monocyte chemotactic protein 1 in human endothelial cells and smooth muscle cells
Susan D. Cushing;Judith A. Berliner;Anthony J. Valente;Mary C. Territo.
Proceedings of the National Academy of Sciences of the United States of America (1990)
Antiinflammatory Properties of HDL
Philip J. Barter;Stephen Nicholls;Kerry Anne Rye;G. M. Anantharamaiah.
Circulation Research (2004)
Minimally modified low density lipoprotein stimulates monocyte endothelial interactions
J A Berliner;M C Territo;A Sevanian;S Ramin.
Journal of Clinical Investigation (1990)
Mice lacking serum paraoxonase are susceptible to organophosphate toxicity and atherosclerosis
Diana M. Shih;Lingjie Gu;Yu Rong Xia;Mohamad Navab.
Malondialdehyde alteration of low density lipoproteins leads to cholesteryl ester accumulation in human monocyte-macrophages
Alan M. Fogelman;Ishaiahu Shechter;Janet Seager;Martha Hokom.
Proceedings of the National Academy of Sciences of the United States of America (1980)
Structural Identification by Mass Spectrometry of Oxidized Phospholipids in Minimally Oxidized Low Density Lipoprotein That Induce Monocyte/Endothelial Interactions and Evidence for Their Presence in Vivo
Andrew D. Watson;Norbert Leitinger;Mohamad Navab;Kym F. Faull.
Journal of Biological Chemistry (1997)
Monocyte transmigration induced by modification of low density lipoprotein in cocultures of human aortic wall cells is due to induction of monocyte chemotactic protein 1 synthesis and is abolished by high density lipoprotein
M Navab;S S Imes;S Y Hama;G P Hough.
Journal of Clinical Investigation (1991)
Induction of endothelial cell expression of granulocyte and macrophage colony-stimulating factors by modified low-density lipoproteins.
T. B. Rajavashisth;A. Andalibi;M. C. Territo;J. A. Berliner.
Anti-inflammatory HDL becomes pro-inflammatory during the acute phase response. Loss of protective effect of HDL against LDL oxidation in aortic wall cell cocultures.
B. J. Van Lenten;S. Y. Hama;F. C. De Beer;D. M. Stafforini.
Journal of Clinical Investigation (1995)
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