D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 62 Citations 15,168 133 World Ranking 4699 National Ranking 2307

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • DNA

His primary areas of study are Paraoxonase, PON1, Aryldialkylphosphatase, Biochemistry and Paraoxon. His studies in Paraoxonase integrate themes in fields like Molecular biology, Toxicity and Cholinesterase. Clement E. Furlong interconnects Enzyme activator and Environmental exposure in the investigation of issues within Toxicity.

He combines subjects such as Pharmacology, Haplotype and Coding region with his study of PON1. His Aryldialkylphosphatase research includes elements of Chlorpyrifos, Low-density lipoprotein, High-density lipoprotein, Parathion and In vivo. His Paraoxon study incorporates themes from Arylesterase, Diazinon and Organophosphate.

His most cited work include:

  • Mice lacking serum paraoxonase are susceptible to organophosphate toxicity and atherosclerosis (950 citations)
  • The molecular basis of the human serum paraoxonase activity polymorphism. (749 citations)
  • The effect of the human serum paraoxonase polymorphism is reversed with diazoxon, soman and sarin (525 citations)

What are the main themes of his work throughout his whole career to date?

Clement E. Furlong mainly focuses on Paraoxonase, PON1, Biochemistry, Aryldialkylphosphatase and Paraoxon. He has included themes like Arylesterase, Genetics and Organophosphate in his Paraoxonase study. His PON1 research is multidisciplinary, incorporating elements of Oxon, Toxicity, Molecular biology and Pharmacology.

His Molecular biology research is multidisciplinary, relying on both Cell culture and Gene expression. His research in Aryldialkylphosphatase intersects with topics in Lipoprotein and High-density lipoprotein. As a part of the same scientific study, Clement E. Furlong usually deals with the Paraoxon, concentrating on Soman and frequently concerns with Sarin.

He most often published in these fields:

  • Paraoxonase (46.89%)
  • PON1 (44.98%)
  • Biochemistry (30.14%)

What were the highlights of his more recent work (between 2011-2021)?

  • PON1 (44.98%)
  • Paraoxonase (46.89%)
  • Internal medicine (17.22%)

In recent papers he was focusing on the following fields of study:

Clement E. Furlong mostly deals with PON1, Paraoxonase, Internal medicine, Endocrinology and Genetics. His PON1 study is concerned with the field of Biochemistry as a whole. The concepts of his Paraoxonase study are interwoven with issues in Aryldialkylphosphatase, Exome, Disease, Genotype and Genetic variation.

His work in the fields of Cohort study overlaps with other areas such as Mesa. His Endocrinology research is multidisciplinary, incorporating elements of Stepwise regression, Case-control study, Cohort and Confounding. In his research on the topic of Genetics, Expression quantitative trait loci and Methylenetetrahydrofolate reductase is strongly related with Blood pressure.

Between 2011 and 2021, his most popular works were:

  • Large-Scale Gene-Centric Meta-Analysis across 39 Studies Identifies Type 2 Diabetes Loci (224 citations)
  • Large-Scale Gene-Centric Meta-analysis across 32 Studies Identifies Multiple Lipid Loci (180 citations)
  • Loci influencing blood pressure identified using a cardiovascular gene-centric array (115 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • DNA

PON1, Paraoxonase, Internal medicine, Endocrinology and Oxidative stress are his primary areas of study. His PON1 study integrates concerns from other disciplines, such as Lipid metabolism, Toxicity and Phylogenetic tree. His biological study deals with issues like Oxon, which deal with fields such as Genetics.

His Paraoxonase research incorporates themes from Zoology, Stepwise regression, Protein function and Aryldialkylphosphatase. His Aryldialkylphosphatase research includes elements of Immunology, Autoimmune disease, Case-control study and Lipoprotein. Clement E. Furlong has included themes like Mitochondrion, Knockout mouse and Neuroprotection in his Oxidative stress study.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Mice lacking serum paraoxonase are susceptible to organophosphate toxicity and atherosclerosis

Diana M. Shih;Lingjie Gu;Yu Rong Xia;Mohamad Navab.
Nature (1998)

1299 Citations

The molecular basis of the human serum paraoxonase activity polymorphism.

Richard Humbert;David A. Adler;Christine M. Disteche;Christopher Hassett.
Nature Genetics (1993)

1031 Citations

The effect of the human serum paraoxonase polymorphism is reversed with diazoxon, soman and sarin

Holly G. Davies;Rebecca J. Richter;Matthew Keifer;Clarence A. Broomfield.
Nature Genetics (1996)

816 Citations

Modulation of paraoxonase (PON1) activity

Lucio G. Costa;Annabella Vitalone;Toby B. Cole;Clement E. Furlong.
Biochemical Pharmacology (2005)

564 Citations

Mechanisms underlying adverse effects of HDL on eNOS-activating pathways in patients with coronary artery disease

Christian Besler;Kathrin Heinrich;Lucia Rohrer;Carola Doerries.
Journal of Clinical Investigation (2011)

556 Citations

Association of in Utero Organophosphate Pesticide Exposure and Fetal Growth and Length of Gestation in an Agricultural Population

Brenda Eskenazi;Kim Harley;Asa Bradman;Erin K Weltzien.
Environmental Health Perspectives (2004)

493 Citations

Effects of 5' regulatory-region polymorphisms on paraoxonase-gene (PON1) expression.

Victoria H. Brophy;Rachel L. Jampsa;James B. Clendenning;Laura A. McKinstry.
American Journal of Human Genetics (2001)

464 Citations

Yeast gene required for spindle pole body duplication: homology of its product with Ca2+-binding proteins

Peter Baum;Clement Furlong;Breck Byers.
Proceedings of the National Academy of Sciences of the United States of America (1986)

420 Citations

Paraoxonase (PON1) Phenotype Is a Better Predictor of Vascular Disease Than Is PON1192 or PON155 Genotype

Gail Pairitz Jarvik;Laura S. Rozek;Victoria H. Brophy;Thomas S. Hatsukami.
Arteriosclerosis, Thrombosis, and Vascular Biology (2000)

389 Citations

Determination of paraoxonase (PON1) status requires more than genotyping.

Rebecca J. Richter;Clement E. Furlong.
Pharmacogenetics (1999)

374 Citations

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