His primary areas of study are Paraoxonase, PON1, Aryldialkylphosphatase, Biochemistry and Paraoxon. His studies in Paraoxonase integrate themes in fields like Molecular biology, Toxicity and Cholinesterase. Clement E. Furlong interconnects Enzyme activator and Environmental exposure in the investigation of issues within Toxicity.
He combines subjects such as Pharmacology, Haplotype and Coding region with his study of PON1. His Aryldialkylphosphatase research includes elements of Chlorpyrifos, Low-density lipoprotein, High-density lipoprotein, Parathion and In vivo. His Paraoxon study incorporates themes from Arylesterase, Diazinon and Organophosphate.
Clement E. Furlong mainly focuses on Paraoxonase, PON1, Biochemistry, Aryldialkylphosphatase and Paraoxon. He has included themes like Arylesterase, Genetics and Organophosphate in his Paraoxonase study. His PON1 research is multidisciplinary, incorporating elements of Oxon, Toxicity, Molecular biology and Pharmacology.
His Molecular biology research is multidisciplinary, relying on both Cell culture and Gene expression. His research in Aryldialkylphosphatase intersects with topics in Lipoprotein and High-density lipoprotein. As a part of the same scientific study, Clement E. Furlong usually deals with the Paraoxon, concentrating on Soman and frequently concerns with Sarin.
Clement E. Furlong mostly deals with PON1, Paraoxonase, Internal medicine, Endocrinology and Genetics. His PON1 study is concerned with the field of Biochemistry as a whole. The concepts of his Paraoxonase study are interwoven with issues in Aryldialkylphosphatase, Exome, Disease, Genotype and Genetic variation.
His work in the fields of Cohort study overlaps with other areas such as Mesa. His Endocrinology research is multidisciplinary, incorporating elements of Stepwise regression, Case-control study, Cohort and Confounding. In his research on the topic of Genetics, Expression quantitative trait loci and Methylenetetrahydrofolate reductase is strongly related with Blood pressure.
PON1, Paraoxonase, Internal medicine, Endocrinology and Oxidative stress are his primary areas of study. His PON1 study integrates concerns from other disciplines, such as Lipid metabolism, Toxicity and Phylogenetic tree. His biological study deals with issues like Oxon, which deal with fields such as Genetics.
His Paraoxonase research incorporates themes from Zoology, Stepwise regression, Protein function and Aryldialkylphosphatase. His Aryldialkylphosphatase research includes elements of Immunology, Autoimmune disease, Case-control study and Lipoprotein. Clement E. Furlong has included themes like Mitochondrion, Knockout mouse and Neuroprotection in his Oxidative stress study.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Mice lacking serum paraoxonase are susceptible to organophosphate toxicity and atherosclerosis
Diana M. Shih;Lingjie Gu;Yu Rong Xia;Mohamad Navab.
Nature (1998)
The molecular basis of the human serum paraoxonase activity polymorphism.
Richard Humbert;David A. Adler;Christine M. Disteche;Christopher Hassett.
Nature Genetics (1993)
The effect of the human serum paraoxonase polymorphism is reversed with diazoxon, soman and sarin
Holly G. Davies;Rebecca J. Richter;Matthew Keifer;Clarence A. Broomfield.
Nature Genetics (1996)
Modulation of paraoxonase (PON1) activity
Lucio G. Costa;Annabella Vitalone;Toby B. Cole;Clement E. Furlong.
Biochemical Pharmacology (2005)
Mechanisms underlying adverse effects of HDL on eNOS-activating pathways in patients with coronary artery disease
Christian Besler;Kathrin Heinrich;Lucia Rohrer;Carola Doerries.
Journal of Clinical Investigation (2011)
Association of in Utero Organophosphate Pesticide Exposure and Fetal Growth and Length of Gestation in an Agricultural Population
Brenda Eskenazi;Kim Harley;Asa Bradman;Erin K Weltzien.
Environmental Health Perspectives (2004)
Effects of 5' regulatory-region polymorphisms on paraoxonase-gene (PON1) expression.
Victoria H. Brophy;Rachel L. Jampsa;James B. Clendenning;Laura A. McKinstry.
American Journal of Human Genetics (2001)
Yeast gene required for spindle pole body duplication: homology of its product with Ca2+-binding proteins
Peter Baum;Clement Furlong;Breck Byers.
Proceedings of the National Academy of Sciences of the United States of America (1986)
Paraoxonase (PON1) Phenotype Is a Better Predictor of Vascular Disease Than Is PON1192 or PON155 Genotype
Gail Pairitz Jarvik;Laura S. Rozek;Victoria H. Brophy;Thomas S. Hatsukami.
Arteriosclerosis, Thrombosis, and Vascular Biology (2000)
Determination of paraoxonase (PON1) status requires more than genotyping.
Rebecca J. Richter;Clement E. Furlong.
Pharmacogenetics (1999)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
University of Washington
University of Washington
Utrecht University
University of Leicester
University of Maryland, Baltimore
University of Pennsylvania
Utrecht University
University of Pennsylvania
University of California, Los Angeles
University of Washington
Princeton University
Florida State University
Daimler (Germany)
University of Tsukuba
Lanzhou University
Northwestern University
University of Messina
Chang Gung University
Finnish Environment Institute
Universität Hamburg
Rush University Medical Center
Beth Israel Deaconess Medical Center
Beth Israel Deaconess Medical Center
Erasmus University Rotterdam
Complutense University of Madrid
Leiden University