What is he best known for?
The fields of study he is best known for:
- Gene
- Enzyme
- Internal medicine
His main research concerns Toxicity, Biochemistry, Internal medicine, Endocrinology and Neurotoxicity.
His work deals with themes such as Paraoxonase, Pharmacology, Cholinesterase and Aryldialkylphosphatase, PON1, which intersect with Toxicity.
His Biochemistry research is multidisciplinary, incorporating perspectives in Parathion, Neuroglia and Neuron.
His studies in Muscarinic acetylcholine receptor and Central nervous system are all subfields of Internal medicine research.
His Neurotoxicity research is multidisciplinary, relying on both Oxidative stress, Polybrominated diphenyl ethers, Glutathione, Environmental chemistry and Physiology.
Lucio G. Costa interconnects Developmental neurotoxicity, Toxicology and Diphenyl ether in the investigation of issues within Polybrominated diphenyl ethers.
His most cited work include:
- Mice lacking serum paraoxonase are susceptible to organophosphate toxicity and atherosclerosis (950 citations)
- Xestospongins: Potent Membrane Permeable Blockers of the Inositol 1,4,5-Trisphosphate Receptor (507 citations)
- Developmental neurotoxicity of polybrominated diphenyl ether (PBDE) flame retardants (468 citations)
What are the main themes of his work throughout his whole career to date?
Lucio G. Costa focuses on Internal medicine, Endocrinology, Neurotoxicity, Biochemistry and Toxicity.
The study incorporates disciplines such as Ethanol, Fetal alcohol syndrome and Monoamine oxidase B in addition to Internal medicine.
His Endocrinology research incorporates themes from Muscarinic acetylcholine receptor and In vivo.
His Neurotoxicity research integrates issues from Oxidative stress, In vitro, Neuroscience and Physiology.
As a part of the same scientific family, he mostly works in the field of Biochemistry, focusing on Cell biology and, on occasion, Astrocyte, Neurite and DNA synthesis.
His Toxicity research is multidisciplinary, relying on both Paraoxonase, Parathion, Toxicology, PON1 and Pharmacology.
He most often published in these fields:
- Internal medicine (38.14%)
- Endocrinology (37.92%)
- Neurotoxicity (33.05%)
What were the highlights of his more recent work (between 2013-2021)?
- Neurotoxicity (33.05%)
- Oxidative stress (17.16%)
- Food science (4.87%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in Neurotoxicity, Oxidative stress, Food science, Fermentation and Biochemistry.
His Neurotoxicity study improves the overall literature in Toxicity.
The Oxidative stress study combines topics in areas such as Reactive oxygen species, Glutathione and Neuroprotection.
His Biochemistry study frequently links to other fields, such as Cell biology.
His PON1 research includes elements of Internal medicine, Endoplasmic reticulum and Endocrinology.
His work carried out in the field of Internal medicine brings together such families of science as Fetus and Physiology.
Between 2013 and 2021, his most popular works were:
- Mechanisms of Neuroprotection by Quercetin: Counteracting Oxidative Stress and More (165 citations)
- A mechanistic view of polybrominated diphenyl ether (PBDE) developmental neurotoxicity. (149 citations)
- A mechanistic view of polybrominated diphenyl ether (PBDE) developmental neurotoxicity. (149 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Enzyme
- Internal medicine
Lucio G. Costa spends much of his time researching Neurotoxicity, Oxidative stress, Internal medicine, Endocrinology and Developmental neurotoxicity.
The various areas that Lucio G. Costa examines in his Neurotoxicity study include Air pollution, Toxicology, Physiology and Pregnancy.
His Pregnancy study deals with Paraoxonase intersecting with PON1.
Oxidative stress is a primary field of his research addressed under Biochemistry.
His biological study spans a wide range of topics, including Offspring, Mitochondrion and Cortex.
His research investigates the connection with Polybrominated diphenyl ethers and areas like Congener which intersect with concerns in Toxicity.
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