D-Index & Metrics Best Publications

Frederic J. Seidler

Duke University
United States

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name Citations Publications World Ranking National Ranking

Medicine D-index 98 Citations 25,723 374 World Ranking 5471 National Ranking 3025

Overview

What is he best known for?

The fields of study Frederic J. Seidler is best known for:

Neurotransmitter
Central nervous system
Dopamine

Internal medicine connects with themes related to Adenylyl cyclase in his study. His study brings together the fields of Internal medicine and Adenylyl cyclase. His Endocrinology study typically links adjacent topics like Stimulation. Stimulation is frequently linked to Endocrinology in his study. Frederic J. Seidler integrates Cholinergic with Neurotransmitter in his study. His work often combines Neurotransmitter and Central nervous system studies. He conducted interdisciplinary study in his works that combined Central nervous system and Neurotoxicity. He combines topics linked to Toxicity with his work on Neurotoxicity. He brings together Toxicity and Pharmacology to produce work in his papers.

His most cited work include:

Developmental Neurotoxicity of Chlorpyrifos: Cellular Mechanisms (268 citations)
Cellular Mechanisms for Developmental Toxicity of Chlorpyrifos: Targeting the Adenylyl Cyclase Signaling Cascade (240 citations)
Adolescent nicotine exposure causes persistent upregulation of nicotinic cholinergic receptors in rat brain regions (220 citations)

What are the main themes of his work throughout his whole career to date

His Pregnancy research includes a combination of various areas of study, such as Fetus and Gestation. Many of his studies on Fetus involve topics that are commonly interrelated, such as Genetics. In his work, he performs multidisciplinary research in Genetics and Offspring. He performs integrative Gestation and Pregnancy research in his work. As part of his studies on Internal medicine, Frederic J. Seidler frequently links adjacent subjects like Nicotine. The study of Endocrinology is intertwined with the study of Dexamethasone in a number of ways. His Dexamethasone study often links to related topics such as Endocrinology. Much of his study explores Receptor relationship to Agonist. Frederic J. Seidler combines topics linked to Receptor with his work on Agonist.

Frederic J. Seidler most often published in these fields:

Internal medicine (96.04%)
Endocrinology (95.05%)
Receptor (56.44%)

What were the highlights of his more recent work (between 2001-2007)?

Internal medicine (100.00%)
Endocrinology (80.00%)
Receptor (60.00%)

In recent works Frederic J. Seidler was focusing on the following fields of study:

His research ties Acetylcholine and Internal medicine together. Borrowing concepts from Central nervous system, he weaves in ideas under Acetylcholine. He combines Central nervous system and Stimulation in his research. His Stimulation study frequently intersects with other fields, such as Neuroscience. Neuroscience is closely attributed to Cerebral cortex in his study. He frequently studies issues relating to Endocrinology and Cerebral cortex. His Endocrinology study frequently draws connections to other fields, such as Catecholaminergic cell groups. His study brings together the fields of Internal medicine and Catecholaminergic cell groups. His research on Receptor frequently links to adjacent areas such as Adrenergic receptor.

Between 2001 and 2007, his most popular works were:

Functional alterations in CNS catecholamine systems in adolescence and adulthood after neonatal chlorpyrifos exposure (113 citations)
Perinatal exposure to environmental tobacco smoke upregulates nicotinic cholinergic receptors in monkey brain (71 citations)
Neuroinflammation and Behavioral Abnormalities after Neonatal Terbutaline Treatment in Rats: Implications for Autism (71 citations)

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Is the PentaBDE replacement, tris (1,3-dichloro-2-propyl) phosphate (TDCPP), a developmental neurotoxicant? Studies in PC12 cells.

Laura V. Dishaw;Christina M. Powers;Ian T. Ryde;Simon C. Roberts.
Toxicology and Applied Pharmacology (2011)

366 Citations

Developmental neurotoxicity of chlorpyrifos: cellular mechanisms.

Karl Whitney;F. J. Seidler;T. A. Slotkin.
Toxicology and Applied Pharmacology (1995)

342 Citations

Adolescent nicotine exposure causes persistent upregulation of nicotinic cholinergic receptors in rat brain regions

J.A Trauth;F.J Seidler;E.C McCook;T.A Slotkin.
Brain Research (1999)

292 Citations

Cellular Mechanisms for Developmental Toxicity of Chlorpyrifos: Targeting the Adenylyl Cyclase Signaling Cascade☆

X. Song;F.J. Seidler;J.L. Saleh;J. Zhang.
Toxicology and Applied Pharmacology (1997)

286 Citations

Comparative Developmental Neurotoxicity of Organophosphates In Vivo: Transcriptional Responses of Pathways for Brain Cell Development, Cell Signaling, Cytotoxicity and Neurotransmitter Systems

Theodore A. Slotkin;Frederic J. Seidler.
Brain Research Bulletin (2007)

282 Citations

Effects of prenatal nicotine exposure on development of central and peripheral cholinergic neurotransmitter systems. Evidence for cholinergic trophic influences in developing brain.

Hernan Navarro;FJ Seidler;JP Eylers;FE Baker.
Journal of Pharmacology and Experimental Therapeutics (1989)

281 Citations

Persistent behavioral consequences of neonatal chlorpyrifos exposure in rats.

Edward D Levin;Nii Addy;Aya Nakajima;N.Channelle Christopher.
Developmental Brain Research (2001)

279 Citations

Adrenomedullary function in the neonatal rat: responses to acute hypoxia.

F J Seidler;T A Slotkin.
The Journal of Physiology (1985)

261 Citations

Prenatal chlorpyrifos exposure in rats causes persistent behavioral alterations.

Edward D Levin;Nii Addy;Avanti Baruah;Alana Elias.
Neurotoxicology and Teratology (2002)

259 Citations

Developmental exposure of rats to chlorpyrifos leads to behavioral alterations in adulthood, involving serotonergic mechanisms and resembling animal models of depression.

Justin E. Aldridge;Edward D. Levin;Frederic J. Seidler;Theodore A. Slotkin.
Environmental Health Perspectives (2005)

257 Citations

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