Internal medicine connects with themes related to Adenylyl cyclase in his study. His study brings together the fields of Internal medicine and Adenylyl cyclase. His Endocrinology study typically links adjacent topics like Stimulation. Stimulation is frequently linked to Endocrinology in his study. Frederic J. Seidler integrates Cholinergic with Neurotransmitter in his study. His work often combines Neurotransmitter and Central nervous system studies. He conducted interdisciplinary study in his works that combined Central nervous system and Neurotoxicity. He combines topics linked to Toxicity with his work on Neurotoxicity. He brings together Toxicity and Pharmacology to produce work in his papers.
His Pregnancy research includes a combination of various areas of study, such as Fetus and Gestation. Many of his studies on Fetus involve topics that are commonly interrelated, such as Genetics. In his work, he performs multidisciplinary research in Genetics and Offspring. He performs integrative Gestation and Pregnancy research in his work. As part of his studies on Internal medicine, Frederic J. Seidler frequently links adjacent subjects like Nicotine. The study of Endocrinology is intertwined with the study of Dexamethasone in a number of ways. His Dexamethasone study often links to related topics such as Endocrinology. Much of his study explores Receptor relationship to Agonist. Frederic J. Seidler combines topics linked to Receptor with his work on Agonist.
His research ties Acetylcholine and Internal medicine together. Borrowing concepts from Central nervous system, he weaves in ideas under Acetylcholine. He combines Central nervous system and Stimulation in his research. His Stimulation study frequently intersects with other fields, such as Neuroscience. Neuroscience is closely attributed to Cerebral cortex in his study. He frequently studies issues relating to Endocrinology and Cerebral cortex. His Endocrinology study frequently draws connections to other fields, such as Catecholaminergic cell groups. His study brings together the fields of Internal medicine and Catecholaminergic cell groups. His research on Receptor frequently links to adjacent areas such as Adrenergic receptor.
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Is the PentaBDE replacement, tris (1,3-dichloro-2-propyl) phosphate (TDCPP), a developmental neurotoxicant? Studies in PC12 cells.
Laura V. Dishaw;Christina M. Powers;Ian T. Ryde;Simon C. Roberts.
Toxicology and Applied Pharmacology (2011)
Developmental neurotoxicity of chlorpyrifos: cellular mechanisms.
Karl Whitney;F. J. Seidler;T. A. Slotkin.
Toxicology and Applied Pharmacology (1995)
Adolescent nicotine exposure causes persistent upregulation of nicotinic cholinergic receptors in rat brain regions
J.A Trauth;F.J Seidler;E.C McCook;T.A Slotkin.
Brain Research (1999)
Cellular Mechanisms for Developmental Toxicity of Chlorpyrifos: Targeting the Adenylyl Cyclase Signaling Cascade☆
X. Song;F.J. Seidler;J.L. Saleh;J. Zhang.
Toxicology and Applied Pharmacology (1997)
Comparative Developmental Neurotoxicity of Organophosphates In Vivo: Transcriptional Responses of Pathways for Brain Cell Development, Cell Signaling, Cytotoxicity and Neurotransmitter Systems
Theodore A. Slotkin;Frederic J. Seidler.
Brain Research Bulletin (2007)
Effects of prenatal nicotine exposure on development of central and peripheral cholinergic neurotransmitter systems. Evidence for cholinergic trophic influences in developing brain.
Hernan Navarro;FJ Seidler;JP Eylers;FE Baker.
Journal of Pharmacology and Experimental Therapeutics (1989)
Persistent behavioral consequences of neonatal chlorpyrifos exposure in rats.
Edward D Levin;Nii Addy;Aya Nakajima;N.Channelle Christopher.
Developmental Brain Research (2001)
Adrenomedullary function in the neonatal rat: responses to acute hypoxia.
F J Seidler;T A Slotkin.
The Journal of Physiology (1985)
Prenatal chlorpyrifos exposure in rats causes persistent behavioral alterations.
Edward D Levin;Nii Addy;Avanti Baruah;Alana Elias.
Neurotoxicology and Teratology (2002)
Developmental exposure of rats to chlorpyrifos leads to behavioral alterations in adulthood, involving serotonergic mechanisms and resembling animal models of depression.
Justin E. Aldridge;Edward D. Levin;Frederic J. Seidler;Theodore A. Slotkin.
Environmental Health Perspectives (2005)
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