Mohamad Navab focuses on Internal medicine, Endocrinology, Biochemistry, Paraoxonase and Lipoprotein. His Internal medicine research is multidisciplinary, incorporating perspectives in Immunology and Cardiology. Mohamad Navab combines subjects such as Inflammation, Proinflammatory cytokine and Monocyte with his study of Endocrinology.
His research in Biochemistry focuses on subjects like Apolipoprotein E, which are connected to Autoxidation, Electrospray ionization, Mass spectrometry and Arteriosclerosis. His Paraoxonase research incorporates themes from Genetically modified mouse, Low-density lipoprotein and Acute-phase protein. He interconnects Oxidative stress, Meal, Lipid oxidation, Cell biology and Superoxide in the investigation of issues within Lipoprotein.
Internal medicine, Endocrinology, Apolipoprotein B, Biochemistry and Lipoprotein are his primary areas of study. Mohamad Navab combines topics linked to Immunology with his work on Internal medicine. In his study, Innate immune system is strongly linked to Inflammation, which falls under the umbrella field of Endocrinology.
His studies deal with areas such as Oral administration, Lesion, Anti-inflammatory, Reverse cholesterol transport and Efflux as well as Apolipoprotein B. His High-density lipoprotein study combines topics from a wide range of disciplines, such as Lipid oxidation and Low-density lipoprotein. His biological study spans a wide range of topics, including Endothelial stem cell and Cell biology.
Mohamad Navab focuses on Internal medicine, Endocrinology, Inflammation, LDL receptor and Apolipoprotein B. All of his Internal medicine and Lipoprotein, High-density lipoprotein, Low-density lipoprotein, Oxidative stress and Pulmonary hypertension investigations are sub-components of the entire Internal medicine study. His Lipoprotein research incorporates elements of Proinflammatory cytokine, Apolipoprotein E, Genetically modified mouse and Myocardial infarction.
The Endocrinology study combines topics in areas such as Serum amyloid A and Lysophosphatidic acid, Receptor. His Inflammation study combines topics in areas such as Tumor necrosis factor alpha, Biochemistry, Angiogenesis and Mechanism of action. His research in Apolipoprotein B tackles topics such as Computational biology which are related to areas like Combinatorial chemistry.
His primary areas of investigation include Internal medicine, Endocrinology, Apolipoprotein B, Inflammation and Lipoprotein. His work deals with themes such as Signal transduction, Hydroxyeicosatetraenoic acid and Ultrafine particle, which intersect with Internal medicine. His Endocrinology research incorporates themes from Serum amyloid A and Lysophosphatidic acid.
Mohamad Navab interconnects Pulmonary hypertension, Computational biology, Cell growth and High-density lipoprotein in the investigation of issues within Apolipoprotein B. His biological study focuses on Proinflammatory cytokine. His Lipoprotein research includes elements of Area under the curve and Pharmacodynamics.
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From vulnerable plaque to vulnerable patient: a call for new definitions and risk assessment strategies: Part II.
Morteza Naghavi;Peter Libby;Erling Falk;S. Ward Casscells;S. Ward Casscells.
Atherosclerosis: basic mechanisms. Oxidation, inflammation, and genetics.
Judith A. Berliner;Mohamad Navab;Alan M. Fogelman;Joy S. Frank.
Antiinflammatory Properties of HDL
Philip J. Barter;Stephen Nicholls;Kerry Anne Rye;G. M. Anantharamaiah.
Circulation Research (2004)
Protective effect of high density lipoprotein associated paraoxonase. Inhibition of the biological activity of minimally oxidized low density lipoprotein.
A. D. Watson;J. A. Berliner;S. Y. Hama;B. N. La Du.
Journal of Clinical Investigation (1995)
From Vulnerable Plaque to Vulnerable Patient
Morteza Naghavi;Peter Libby;Erling Falk;S. Ward Casscells.
Mice lacking serum paraoxonase are susceptible to organophosphate toxicity and atherosclerosis
Diana M. Shih;Lingjie Gu;Yu Rong Xia;Mohamad Navab.
Structural Identification by Mass Spectrometry of Oxidized Phospholipids in Minimally Oxidized Low Density Lipoprotein That Induce Monocyte/Endothelial Interactions and Evidence for Their Presence in Vivo
Andrew D. Watson;Norbert Leitinger;Mohamad Navab;Kym F. Faull.
Journal of Biological Chemistry (1997)
The Yin and Yang of Oxidation in the Development of the Fatty Streak: A Review Based on the 1994 George Lyman Duff Memorial Lecture
M Navab;J A Berliner;A D Watson;S Y Hama.
Arteriosclerosis, Thrombosis, and Vascular Biology (1996)
Anti-inflammatory HDL becomes pro-inflammatory during the acute phase response. Loss of protective effect of HDL against LDL oxidation in aortic wall cell cocultures.
B. J. Van Lenten;S. Y. Hama;F. C. De Beer;D. M. Stafforini.
Journal of Clinical Investigation (1995)
The oxidation hypothesis of atherogenesis: the role of oxidized phospholipids and HDL.
Mohamad Navab;G.M. Ananthramaiah;Srinivasa T. Reddy;Brian J. Van Lenten.
Journal of Lipid Research (2004)
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