D-Index & Metrics Best Publications
Immunology
Germany
2023

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 81 Citations 36,339 168 World Ranking 954 National Ranking 59

Research.com Recognitions

Awards & Achievements

2023 - Research.com Immunology in Germany Leader Award

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Cytokine

His main research concerns Immunology, Cell biology, Receptor, Cytokine and Signal transduction. His work deals with themes such as Macrophage colony-stimulating factor, Macrophage and Macrophage polarization, which intersect with Immunology. His Macrophage research is multidisciplinary, relying on both Inflammation, T cell, Ontogeny and Homeostasis.

His Cell biology research includes elements of SOCS6 and NOD2. The study incorporates disciplines such as Transcription factor, Adenosine and Cellular differentiation in addition to Receptor. His Interleukin 10 study deals with Molecular biology intersecting with Gene expression.

His most cited work include:

  • Protective and pathogenic functions of macrophage subsets (2863 citations)
  • Macrophage Activation and Polarization: Nomenclature and Experimental Guidelines (2761 citations)
  • Recommendations for myeloid-derived suppressor cell nomenclature and characterization standards (1088 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of investigation include Immunology, Cell biology, Immune system, Inflammation and Macrophage. His Immunology research incorporates elements of Microbiology and Arginase. His research integrates issues of Suppressor of cytokine signaling 1, Receptor, Cytokine and NOD2 in his study of Cell biology.

His Inflammation research is multidisciplinary, incorporating perspectives in Wound healing and Cancer research. His Macrophage study integrates concerns from other disciplines, such as Fibrosis and Cancer. His studies deal with areas such as Molecular biology and STAT3 as well as Interleukin 10.

He most often published in these fields:

  • Immunology (38.50%)
  • Cell biology (30.00%)
  • Immune system (24.00%)

What were the highlights of his more recent work (between 2015-2021)?

  • Cancer research (18.00%)
  • Immune system (24.00%)
  • Immunology (38.50%)

In recent papers he was focusing on the following fields of study:

Cancer research, Immune system, Immunology, T cell and Inflammation are his primary areas of study. His Immune system research is multidisciplinary, incorporating elements of Tryptophan, Antigen, Myeloid-derived Suppressor Cell and Serotonin. The various areas that Peter J. Murray examines in his Immunology study include Haematopoiesis and Macrophage.

His T cell study combines topics from a wide range of disciplines, such as Cell growth, Tumor microenvironment, Cell biology, Cytotoxic T cell and Immunotherapy. The Cell biology study combines topics in areas such as Amino acid and Biochemistry. His Inflammation research incorporates themes from Interferon and Lung injury.

Between 2015 and 2021, his most popular works were:

  • Recommendations for myeloid-derived suppressor cell nomenclature and characterization standards (1088 citations)
  • New insights into the multidimensional concept of macrophage ontogeny, activation and function (363 citations)
  • Inhibition of arginase by CB-1158 blocks myeloid cell-mediated immune suppression in the tumor microenvironment (122 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Enzyme

Peter J. Murray mostly deals with T cell, Immunology, Immune system, Cancer research and Immunotherapy. His T cell study also includes

  • Cytotoxic T cell which is related to area like Cell biology,
  • CD8 that intertwine with fields like T-cell receptor, Kinase, Translation and Signal transduction. Peter J. Murray works in the field of Immunology, focusing on Immunity in particular.

His study in Immunity is interdisciplinary in nature, drawing from both Inflammation, Bone marrow and Homeostasis. In the field of Immune system, his study on Immune tolerance overlaps with subjects such as Confusion. His studies deal with areas such as Myeloid and Tumor microenvironment as well as Immunotherapy.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Protective and pathogenic functions of macrophage subsets

Peter J. Murray;Thomas A. Wynn.
Nature Reviews Immunology (2011)

4729 Citations

Macrophage Activation and Polarization: Nomenclature and Experimental Guidelines

Peter J Murray;Judith E Allen;Subhra K Biswas;Edward A Fisher.
Immunity (2014)

4506 Citations

Recommendations for myeloid-derived suppressor cell nomenclature and characterization standards

Vincenzo Bronte;Sven Brandau;Shu Hsia Chen;Mario P. Colombo.
Nature Communications (2016)

1829 Citations

The JAK-STAT Signaling Pathway: Input and Output Integration

Peter J. Murray.
Journal of Immunology (2007)

1343 Citations

Oxidative metabolism and PGC-1β attenuate macrophage-mediated inflammation

Divya Vats;Lata Mukundan;Justin I. Odegaard;Lina Zhang.
Cell Metabolism (2006)

1200 Citations

Signalling pathways and molecular interactions of NOD1 and NOD2

Warren Strober;Peter J. Murray;Atsushi Kitani;Tomohiro Watanabe.
Nature Reviews Immunology (2006)

1035 Citations

NOD2 is a negative regulator of Toll-like receptor 2-mediated T helper type 1 responses.

To mohiro Watanabe;Atsushi Kitani;Peter J Murray;Warren Strober.
Nature Immunology (2004)

1025 Citations

Arginase-1–Expressing Macrophages Suppress Th2 Cytokine–Driven Inflammation and Fibrosis

John T. Pesce;Thirumalai R. Ramalingam;Margaret M. Mentink-Kane;Mark S. Wilson.
PLOS Pathogens (2009)

817 Citations

Cytokine Signaling Modules in Inflammatory Responses

John J. O'Shea;Peter J. Murray.
Immunity (2008)

804 Citations

Shaping Gene Expression in Activated and Resting Primary Macrophages by IL-10

Roland Lang;Divyen Patel;John J. Morris;Robert L. Rutschman.
Journal of Immunology (2002)

658 Citations

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