What is he best known for?
The fields of study he is best known for:
Peter D. Adams mostly deals with Cell biology, Senescence, Chromatin, Autophagy and Cell.
His work deals with themes such as Lamin, Cancer cell, Cancer, Bisulfite sequencing and Heterochromatin, which intersect with Cell biology.
His Senescence research includes elements of Inflammation, Cell cycle, Regulation of gene expression and Chromatin remodeling.
He combines topics linked to Histone with his work on Chromatin.
His Autophagy research is multidisciplinary, incorporating elements of Suppressor and KRAS, Pancreatic Intraepithelial Neoplasia.
The various areas that Peter D. Adams examines in his Computational biology study include BECN1, MAP1LC3B, Sequestosome 1, Autophagosome and Physiology.
His most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4170 citations)
- Deregulated transcription factor E2F-1 expression leads to S-phase entry and p53-mediated apoptosis. (697 citations)
- Formation of MacroH2A-Containing Senescence-Associated Heterochromatin Foci and Senescence Driven by ASF1a and HIRA (532 citations)
What are the main themes of his work throughout his whole career to date?
His primary scientific interests are in Cell biology, Senescence, Chromatin, Cancer research and Epigenetics.
Peter D. Adams mostly deals with Cell aging in his studies of Cell biology.
As part of the same scientific family, he usually focuses on Senescence, concentrating on Mitosis and intersecting with Fibrosis.
His Chromatin study deals with Histone intersecting with Molecular biology, Chaperone and Viral replication.
His Cancer research research incorporates themes from Carcinogenesis, Cancer, Suppressor, Immunology and Mdm2.
The Epigenetics study combines topics in areas such as Epigenomics, Epigenesis, DNA methylation, Epigenome and Longevity.
He most often published in these fields:
- Cell biology (55.70%)
- Senescence (39.60%)
- Chromatin (30.20%)
What were the highlights of his more recent work (between 2018-2021)?
- Senescence (39.60%)
- Cancer research (29.53%)
- Cell biology (55.70%)
In recent papers he was focusing on the following fields of study:
The scientist’s investigation covers issues in Senescence, Cancer research, Cell biology, Phenotype and Cancer.
His study in Senescence is interdisciplinary in nature, drawing from both Chromatin, Senolytic and Mitochondrion.
His Chromatin study combines topics from a wide range of disciplines, such as Enhancer, Gene silencing, Histone Acetyltransferase p300 and Epigenome.
The concepts of his Cancer research study are interwoven with issues in Cell culture, Suppressor, Immune system, Orphan receptor and Metastasis.
Peter D. Adams combines subjects such as Telomere, Epigenetics and Oncogene-induced senescence with his study of Cell biology.
His research in Phenotype intersects with topics in Oncogene Activation, Transcriptome, CEBPB and Notch signaling pathway.
Between 2018 and 2021, his most popular works were:
- Cellular Senescence: Defining a Path Forward (329 citations)
- DNA methylation aging clocks: challenges and recommendations (142 citations)
- DNA methylation aging clocks: challenges and recommendations (142 citations)
In his most recent research, the most cited papers focused on:
Peter D. Adams spends much of his time researching Senescence, Cell biology, Mitochondrion, Computational biology and Nuclear protein.
His work carried out in the field of Senescence brings together such families of science as Phenotype, Regulation of gene expression and DNA damage.
His Cell biology research focuses on Mitosis in particular.
His biological study spans a wide range of topics, including Cell, Cell cycle checkpoint, Senolytic, Chromatin and Developmental biology.
Peter D. Adams has researched Computational biology in several fields, including Epigenesis, DNA methylation, CpG site, Epigenetics and Human genetics.
Peter D. Adams interconnects Proinflammatory cytokine, Oxidative stress, Oxidative phosphorylation and Retrograde signaling in the investigation of issues within Nuclear protein.
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