What is he best known for?
The fields of study he is best known for:
His primary scientific interests are in Senescence, Cell biology, Telomere, Senolytic and Mitochondrion.
His Senescence study incorporates themes from Phenotype, Chromatin, Transgene and Immunology.
His work deals with themes such as Cell and Ageing, which intersect with Cell biology.
His Telomere study incorporates themes from DNA damage, Telomerase and Mitochondrial DNA.
His study in Senolytic is interdisciplinary in nature, drawing from both Adipose tissue macrophages, Cancer research, Adipocyte and Progenitor cell.
The study incorporates disciplines such as Proinflammatory cytokine and Steatosis in addition to Mitochondrion.
His most cited work include:
- Feedback between p21 and reactive oxygen production is necessary for cell senescence (522 citations)
- Mitochondrial dysfunction accounts for the stochastic heterogeneity in telomere-dependent senescence. (469 citations)
- Cellular senescence mediates fibrotic pulmonary disease (465 citations)
What are the main themes of his work throughout his whole career to date?
His scientific interests lie mostly in Senescence, Cell biology, Mitochondrion, Telomere and Ageing.
He has included themes like Phenotype, Oxidative stress, Senolytic and Cell cycle checkpoint in his Senescence study.
His Phenotype research is multidisciplinary, incorporating elements of Cancer research, PI3K/AKT/mTOR pathway and Homeostasis.
His work carried out in the field of Cell biology brings together such families of science as Cellular senescence, Cell, Genetics and DNA damage.
João F. Passos combines subjects such as Cell aging, Mitochondrial DNA and Cytosol with his study of Mitochondrion.
His studies deal with areas such as Paracrine signalling, Immunology, Mitochondrial ROS, Stem cell and Telomerase as well as Telomere.
He most often published in these fields:
- Senescence (81.88%)
- Cell biology (71.01%)
- Mitochondrion (33.33%)
What were the highlights of his more recent work (between 2019-2021)?
- Senescence (81.88%)
- Inflammation (15.94%)
- Cell biology (71.01%)
In recent papers he was focusing on the following fields of study:
His primary areas of study are Senescence, Inflammation, Cell biology, Senolytic and Mitochondrion.
The various areas that João F. Passos examines in his Senescence study include Telomere, Oxidative stress, Endocrinology and Cell cycle checkpoint.
His biological study spans a wide range of topics, including DNA damage, Senescence-Associated Secretory Phenotype, Cellular senescence, Stem cell and Tissue architecture.
João F. Passos integrates many fields in his works, including Cell biology and Arginine.
In his work, Clinical trial, Bioinformatics and Senescent cell is strongly intertwined with Disease, which is a subfield of Senolytic.
Many of his studies on Mitochondrion apply to Mitochondrial DNA as well.
Between 2019 and 2021, his most popular works were:
- Mitochondria-to-nucleus retrograde signaling drives formation of cytoplasmic chromatin and inflammation in senescence (40 citations)
- Mitochondria-to-nucleus retrograde signaling drives formation of cytoplasmic chromatin and inflammation in senescence (40 citations)
- Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition) (38 citations)
In his most recent research, the most cited papers focused on:
The scientist’s investigation covers issues in Cancer research, Phenotype, Senescence, Inflammation and Autophagy.
His work in Cancer research addresses issues such as Senolytic, which are connected to fields such as DNA damage, Apoptosis, Osteoporosis, Telomere and Bone marrow.
While the research belongs to areas of Phenotype, João F. Passos spends his time largely on the problem of Disease, intersecting his research to questions surrounding Bioinformatics.
His Senescence research is multidisciplinary, incorporating perspectives in Oxidative stress and Retrograde signaling.
His Oxidative stress research incorporates themes from Oxidative phosphorylation, Cytosol, Mitochondrion, Cell biology and Proinflammatory cytokine.
His Inflammation research integrates issues from Cancer, Cancer risk, ATG5, Sequestosome-1 Protein and Ageing.
This overview was generated by a machine learning system which analysed the scientist’s
body of work. If you have any feedback, you can
contact us
here.
