The scientist’s investigation covers issues in Immunology, Cell biology, Immune system, Cytotoxic T cell and Interleukin 33. His Cell biology research includes themes of Interleukin 3, Cell, T cell, T-cell receptor and Interleukin 4. The study incorporates disciplines such as CTL*, CD8 and Cellular differentiation in addition to T cell.
His T-cell receptor study which covers T cell differentiation that intersects with Virology, Lymphocytic choriomeningitis and Virus. His research integrates issues of Inflammation, Transcription factor and Cytokine in his study of Immune system. Max Löhning does research in Cytotoxic T cell, focusing on IL-2 receptor specifically.
Max Löhning focuses on Immunology, Cell biology, Cytotoxic T cell, Immune system and Cytokine. The Immunology study combines topics in areas such as Cellular differentiation and Virology. He has included themes like Cell, Transcription factor, Molecular biology and Antigen in his Cell biology study.
His Cytotoxic T cell research integrates issues from Cancer research and CD8. His Immune system study combines topics from a wide range of disciplines, such as Inflammation and Interleukin 33. In his work, Cirrhosis and Hepatocellular carcinoma is strongly intertwined with Virus, which is a subfield of Lymphocytic choriomeningitis.
Max Löhning spends much of his time researching Immunology, Cell biology, Cytotoxic T cell, Immune system and Immunity. His research on Immunology often connects related topics like Cellular differentiation. His Cell biology research incorporates elements of Cell, Bone marrow and Secretion.
His Cytotoxic T cell research is multidisciplinary, incorporating perspectives in Cancer research and CD8. Many of his studies on Immune system involve topics that are commonly interrelated, such as Inflammation. His work carried out in the field of Lymphocytic choriomeningitis brings together such families of science as Virus and Viral replication.
His primary areas of study are Immunology, Cytotoxic T cell, T cell, Cell biology and Signal transduction. His research brings together the fields of Small intestine and Immunology. His study in Small intestine is interdisciplinary in nature, drawing from both Homeostasis, Inflammation, Secretion, Immune system and Interleukin 33.
His studies deal with areas such as Interleukin 21, CD8, Cellular differentiation and T cell differentiation as well as IL-2 receptor. The concepts of his Priming study are interwoven with issues in Cancer immunotherapy, Immunotherapy, CTL* and Effector T-Lymphocyte. His Interleukin 10 research incorporates themes from Antigen-presenting cell, Transforming growth factor beta, Interleukin 13, Chemokine receptor and FOXP3.
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P- and E-selectin mediate recruitment of T-helper-1 but not T-helper-2 cells into inflamed tissues
Frank Austrup;Dietmar Vestweber;Eric Borges;Max Löhning.
Stat6-independent GATA-3 autoactivation directs IL-4-independent Th2 development and commitment.
Wenjun Ouyang;Max Löhning;Zhiguang Gao;Mario Assenmacher.
T1/ST2 is preferentially expressed on murine Th2 cells, independent of interleukin 4, interleukin 5, and interleukin 10, and important for Th2 effector function
Max Löhning;Arne Stroehmann;Anthony J. Coyle;Jane L. Grogan.
Proceedings of the National Academy of Sciences of the United States of America (1998)
The alarmin IL-33 promotes regulatory T-cell function in the intestine
Chris Schiering;Thomas Krausgruber;Agnieszka Chomka;Anja Fröhlich.
Eosinophils are required for the maintenance of plasma cells in the bone marrow
Van Trung Chu;Anja Fröhlich;Gudrun Steinhauser;Tobias Scheel.
Nature Immunology (2011)
Survival of long-lived plasma cells is independent of antigen
R A Manz;M Löhning;G Cassese;A Thiel.
International Immunology (1998)
Interferons Direct Th2 Cell Reprogramming to Generate a Stable GATA-3+T-bet+ Cell Subset with Combined Th2 and Th1 Cell Functions
Ahmed N. Hegazy;Ahmed N. Hegazy;Michael Peine;Michael Peine;Caroline Helmstetter;Caroline Helmstetter;Isabel Panse;Isabel Panse.
The Alarmin Interleukin-33 Drives Protective Antiviral CD8+ T Cell Responses
Weldy V. Bonilla;Anja Fröhlich;Anja Fröhlich;Karin Senn;Sandra Kallert.
Expression of ICOS In Vivo Defines CD4+ Effector T Cells with High Inflammatory Potential and a Strong Bias for Secretion of Interleukin 10
Max Löhning;Andreas Hutloff;Tilmann Kallinich;Tilmann Kallinich;Hans Werner Mages.
Journal of Experimental Medicine (2003)
Professional memory CD4+ T lymphocytes preferentially reside and rest in the bone marrow.
Koji Tokoyoda;Sandra Zehentmeier;Ahmed N. Hegazy;Inka Albrecht.
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