The scientist’s investigation covers issues in Immunology, Immune system, IL-2 receptor, Molecular biology and Cytotoxic T cell. His studies link Transplantation with Immunology. His study looks at the relationship between IL-2 receptor and topics such as CD40, which overlap with Interleukin 12 and Interleukin 17.
His Molecular biology research is multidisciplinary, incorporating elements of Cytokine and Cell biology. His Cytokine research incorporates themes from Tumor necrosis factor alpha, In vitro and Borrelia burgdorferi. His Cytotoxic T cell study deals with CD8 intersecting with RAR-related orphan receptor gamma.
His primary scientific interests are in Immunology, Arthritis, Immune system, Cell biology and Cytokine. His work on Immunology deals in particular with Antibody, Inflammation, Autoimmunity, T cell and Antigen. In his research, Molecular biology is intimately related to Cytotoxic T cell, which falls under the overarching field of T cell.
His work focuses on many connections between Arthritis and other disciplines, such as Rheumatoid arthritis, that overlap with his field of interest in Polyarthritis. His work carried out in the field of Cell biology brings together such families of science as Interleukin 33, In vitro and Interleukin 10. His study in Cytokine is interdisciplinary in nature, drawing from both Tumor necrosis factor alpha, Receptor, Kinase and Mast cell differentiation.
His primary areas of investigation include Immunology, Interleukin 33, Cell biology, Cytokine and Interleukin 6. His study in the fields of T cell, Interleukin and Antibody under the domain of Immunology overlaps with other disciplines such as Pandemic. He works mostly in the field of Interleukin, limiting it down to topics relating to Interleukin 18 and, in certain cases, Inflammation.
His Homeostasis study in the realm of Cell biology connects with subjects such as Eosinophil. In his study, which falls under the umbrella issue of Cytokine, Tumor necrosis factor alpha is strongly linked to p38 mitogen-activated protein kinases. His research investigates the connection between Allergic inflammation and topics such as Immune system that intersect with problems in Osteoclast.
Thomas Kamradt mostly deals with Immunology, Flow cytometry, Antibody, Osteoclast and Cytokine. His research related to Secondary infection, Fungal antigen, T lymphocyte, Sepsis and T cell might be considered part of Immunology. His work on Cell separation and Cell sorting as part of general Flow cytometry study is frequently linked to Cell selection and Data science, bridging the gap between disciplines.
His research in Antibody intersects with topics in Outbreak, Internal medicine, Cohort study and Immunity. His Osteoclast study integrates concerns from other disciplines, such as Proinflammatory cytokine, Inflammation, Cortical bone and Monocyte. His Cytokine research is multidisciplinary, incorporating perspectives in Tumor necrosis factor alpha and Signal transduction.
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Microbial Lipopeptides Induce the Production of IL-17 in Th Cells
Carmen Infante-Duarte;Heidi F. Horton;Michael C. Byrne;Thomas Kamradt.
Journal of Immunology (2000)
T1/ST2 is preferentially expressed on murine Th2 cells, independent of interleukin 4, interleukin 5, and interleukin 10, and important for Th2 effector function
Max Löhning;Arne Stroehmann;Anthony J. Coyle;Jane L. Grogan.
Proceedings of the National Academy of Sciences of the United States of America (1998)
The development of inflammatory T(H)-17 cells requires interferon-regulatory factor 4.
Anne Brüstle;Sylvia Heink;Magdalena Huber;Christine Rosenplänter.
Nature Immunology (2007)
Tim-3 inhibits T helper type 1-mediated auto- and alloimmune responses and promotes immunological tolerance
Alberto Sánchez-Fueyo;Jane Tian;Dominic Picarella;Christoph Domenig.
Nature Immunology (2003)
Crucial role of the interleukin 1 receptor family member T1/ST2 in T helper cell type 2-mediated lung mucosal immune responses.
Anthony J. Coyle;Clare Lloyd;Jane Tian;Trang Nguyen.
Journal of Experimental Medicine (1999)
Cutting Edge: Regulatory T Cells Prevent Efficient Clearance of Mycobacterium tuberculosis
Mischo Kursar;Markus Koch;Hans-Willi Mittrücker;Geraldine Nouailles.
Journal of Immunology (2007)
Interleukin (IL)-23 mediates Toxoplasma gondii-induced immunopathology in the gut via matrixmetalloproteinase-2 and IL-22 but independent of IL-17.
Melba Muñoz;Markus M. Heimesaat;Kerstin Danker;Daniela Struck.
Journal of Experimental Medicine (2009)
Regulatory CD4+CD25+ T cells restrict memory CD8+ T cell responses.
Mischo Kursar;Kerstin Bonhagen;Joachim Fensterle;Anne Köhler.
Journal of Experimental Medicine (2002)
A Th17-like developmental process leads to CD8(+) Tc17 cells with reduced cytotoxic activity.
Magdalena Huber;Sylvia Heink;Henrike Grothe;Anna Guralnik.
European Journal of Immunology (2009)
IL-17A secretion by CD8+ T cells supports Th17-mediated autoimmune encephalomyelitis
Magdalena Huber;Sylvia Heink;Axel Pagenstecher;Katharina Reinhard.
Journal of Clinical Investigation (2013)
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