2017 - Fellow of the Royal Society, United Kingdom
Andrew N. J. McKenzie mainly investigates Immunology, Interleukin 13, Innate lymphoid cell, Cytokine and Immune system. Immunology is represented through his Interleukin 33, Interleukin 4, Inflammation, Nippostrongylus brasiliensis and Immunity research. The Interleukin 33 study combines topics in areas such as IL-2 receptor, Receptor, Degranulation, Adoptive cell transfer and Cell biology.
In general Interleukin 13 study, his work on Lebrikizumab often relates to the realm of Population, thereby connecting several areas of interest. The various areas that Andrew N. J. McKenzie examines in his Innate lymphoid cell study include Tissue homeostasis and Cellular differentiation. His Cytokine research incorporates themes from T cell and Pathogenesis.
His primary areas of study are Immunology, Innate lymphoid cell, Interleukin 13, Immune system and Cytokine. His work in Inflammation, Immunity, Interleukin, Interleukin 33 and Interleukin 4 are all subfields of Immunology research. His Innate lymphoid cell research integrates issues from Thymic stromal lymphopoietin, Cellular differentiation and Allergic inflammation.
His Interleukin 13 research is multidisciplinary, incorporating perspectives in Eosinophil, Interleukin 5, Eosinophilia, Molecular biology and Immunoglobulin E. His research investigates the connection between Immune system and topics such as Cell biology that intersect with problems in Cell and Transcription factor. Andrew N. J. McKenzie interconnects Proinflammatory cytokine, Receptor and T cell, Adoptive cell transfer in the investigation of issues within Cytokine.
Andrew N. J. McKenzie mostly deals with Innate lymphoid cell, Immunology, Immune system, Inflammation and Cell biology. Andrew N. J. McKenzie combines subjects such as Interleukin 13 and Cytokine with his study of Innate lymphoid cell. His study in Thymic stromal lymphopoietin, Interleukin 33, Atopic dermatitis, Antigen and Immunoglobulin E falls within the category of Immunology.
His Immune system research includes elements of Phenotype, Downregulation and upregulation, Lung and Allergic inflammation. His research integrates issues of Endocrinology, Human skin and Pathogenesis in his study of Inflammation. His Cell biology research is multidisciplinary, relying on both Nippostrongylus brasiliensis, Cell, Transcription factor and Cellular differentiation.
Innate lymphoid cell, Immunology, Immune system, Inflammation and Immunity are his primary areas of study. His studies in Innate lymphoid cell integrate themes in fields like Interleukin, Interleukin 13, Cytokine and Cell biology. The study incorporates disciplines such as Nippostrongylus brasiliensis and Cellular differentiation in addition to Cell biology.
His study involves Interleukin 33, Filaggrin, Atopic dermatitis, Thymic stromal lymphopoietin and Interleukin 5, a branch of Immunology. His Immune system study combines topics in areas such as Tissue homeostasis and Downregulation and upregulation. His Acquired immune system research is multidisciplinary, incorporating elements of Cell and Innate immune system.
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Innate lymphoid cells — how did we miss them?
Jennifer A. Walker;Jillian L. Barlow;Andrew N. J. McKenzie.
Nature Reviews Immunology (2013)
Identification of an interleukin (IL)-25–dependent cell population that provides IL-4, IL-5, and IL-13 at the onset of helminth expulsion
Padraic G. Fallon;Sarah J. Ballantyne;Niamh E. Mangan;Jillian L. Barlow.
Journal of Experimental Medicine (2006)
Group 2 innate lymphoid cells are critical for the initiation of adaptive T helper 2 cell-mediated allergic lung inflammation.
Timotheus Y.F. Halim;Catherine A. Steer;Laura Mathä;Matthew J. Gold.
A role for IL-25 and IL-33-driven type-2 innate lymphoid cells in atopic dermatitis.
Maryam Salimi;Jillian L. Barlow;Sean P. Saunders;Luzheng Xue.
Journal of Experimental Medicine (2013)
ST2 is an inhibitor of interleukin 1 receptor and Toll-like receptor 4 signaling and maintains endotoxin tolerance.
Elizabeth K Brint;Damo Xu;Haiying Liu;Aisling Dunne.
Nature Immunology (2004)
Periostin: A novel component of subepithelial fibrosis of bronchial asthma downstream of IL-4 and IL-13 signals
Go Takayama;Kazuhiko Arima;Taisuke Kanaji;Shuji Toda.
The Journal of Allergy and Clinical Immunology (2006)
An Mll–AF9 Fusion Gene Made by Homologous Recombination Causes Acute Leukemia in Chimeric Mice: A Method to Create Fusion Oncogenes
Javier Corral;Isabelle Lavenir;Helen Impey;Alan J Warren.
Innate IL-13–producing nuocytes arise during allergic lung inflammation and contribute to airways hyperreactivity
Jillian L. Barlow;Agustin Bellosi;Clare S. Hardman;Lesley F. Drynan.
The Journal of Allergy and Clinical Immunology (2012)
Innate Lymphoid Cells: 10 Years On.
Eric Vivier;David Artis;Marco Colonna;Andreas Diefenbach.
IL-33 exacerbates antigen-induced arthritis by activating mast cells
Damo Xu;Hui-Rong Jiang;Peter Kewin;Yubin Li.
Proceedings of the National Academy of Sciences of the United States of America (2008)
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