What is he best known for?
The fields of study he is best known for:
- Gene
- Immune system
- Internal medicine
His primary scientific interests are in Immunology, Inflammation, Interleukin 5, Eosinophil and Eosinophilia.
Immunology is a component of his Eotaxin, Cytokine, Interleukin 13, Allergic inflammation and Interleukin studies.
His biological study spans a wide range of topics, including Pathogenesis, Pathology, Ovalbumin, Methacholine and Asthma.
In his research, Hypereosinophilia, Tryptase and CCR3 is intimately related to Lung, which falls under the overarching field of Interleukin 5.
His Eosinophil research includes elements of Fibrosis and Innate immune system, Immune system.
His studies deal with areas such as Endocrinology, Degranulation and Aeroallergen as well as Eosinophilia.
His most cited work include:
- Interleukin 5 deficiency abolishes eosinophilia, airways hyperreactivity, and lung damage in a mouse asthma model. (1264 citations)
- Eosinophils: Biological Properties and Role in Health and Disease (604 citations)
- Eosinophils: changing perspectives in health and disease (485 citations)
What are the main themes of his work throughout his whole career to date?
Immunology, Inflammation, Eosinophil, Asthma and Interleukin 5 are his primary areas of study.
Immune system, Cytokine, Interleukin 13, Eosinophilia and Allergic inflammation are the core of his Immunology study.
His Inflammation study also includes fields such as
- Pathogenesis which connect with Airway,
- Immunoglobulin E that intertwine with fields like Pathology.
His studies in Eosinophil integrate themes in fields like Chemokine and Bronchoalveolar lavage.
His study explores the link between Asthma and topics such as Lung that cross with problems in Respiratory system.
His work on Interleukin 5 is being expanded to include thematically relevant topics such as Interleukin 4.
He most often published in these fields:
- Immunology (86.07%)
- Inflammation (34.83%)
- Eosinophil (25.87%)
What were the highlights of his more recent work (between 2015-2021)?
- Immunology (86.07%)
- Inflammation (34.83%)
- Asthma (21.39%)
In recent papers he was focusing on the following fields of study:
His scientific interests lie mostly in Immunology, Inflammation, Asthma, Lung and Tumor necrosis factor alpha.
His Immunology study often links to related topics such as COPD.
His Inflammation research is multidisciplinary, relying on both Fibrosis, Immune system and In vivo.
His research in Asthma intersects with topics in Virus, Gene expression, Disease and Eosinophilic.
In the subject of general Lung, his work in Bronchoalveolar lavage is often linked to Coronavirus, thereby combining diverse domains of study.
He combines subjects such as Colitis, Inflammatory bowel disease, CXCL1, Neutrophilia and Proinflammatory cytokine with his study of Tumor necrosis factor alpha.
Between 2015 and 2021, his most popular works were:
- MicroRNA-21 drives severe, steroid-insensitive experimental asthma by amplifying phosphoinositide 3-kinase–mediated suppression of histone deacetylase 2 (110 citations)
- Mechanisms and treatments for severe, steroid-resistant allergic airway disease and asthma (65 citations)
- MicroRNA-125a and -b inhibit A20 and MAVS to promote inflammation and impair antiviral response in COPD. (62 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Immune system
- Internal medicine
Paul S. Foster spends much of his time researching Immunology, Inflammation, Pathogenesis, Disease and Asthma.
His Immunology study combines topics from a wide range of disciplines, such as Bronchoalveolar lavage, Antagomir and Chronic gvhd.
His Inflammation study integrates concerns from other disciplines, such as Tumor necrosis factor alpha, Plasminogen activator and Necrosis.
The study incorporates disciplines such as Bioinformatics, Donor derived, microRNA, Lung and Interleukin in addition to Disease.
His research integrates issues of Pathological and Intensive care medicine in his study of Asthma.
His Cytokine research includes themes of Eosinophil and Innate immune system.
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