James J. Lee mainly investigates Immunology, Eosinophil, Immune system, Eosinophilia and Interleukin 5. In most of his Immunology studies, his work intersects topics such as Lung. The concepts of his Eosinophil study are interwoven with issues in Bronchoalveolar lavage and Pathology.
His Immune system research integrates issues from Cytotoxic T cell, Trichinella spiralis and Effector. His Interleukin 5 research includes themes of Infiltration, Granuloma, Fibrosis, Innate immune system and Helminthiasis. The concepts of his Cell biology study are interwoven with issues in RNA, Extracellular RNA, In situ hybridization, Microvesicles and Molecular biology.
Immunology, Eosinophil, Eosinophil peroxidase, Inflammation and Pathology are his primary areas of study. In his research on the topic of Immunology, Ovalbumin is strongly related with Lung. Eosinophil is frequently linked to Cell biology in his study.
His Eosinophil peroxidase research incorporates elements of Eosinophil Granule Proteins and Molecular biology. James J. Lee is involved in the study of Pathology that focuses on Eosinophilic esophagitis in particular. His research integrates issues of Gastroenterology, GERD and Esophagus in his study of Eosinophilic esophagitis.
His primary scientific interests are in Immunology, Eosinophil, Eosinophil peroxidase, Eosinophilic esophagitis and Eosinophilia. His Immunology research is multidisciplinary, incorporating perspectives in Lung and Pathology. His research in Eosinophil intersects with topics in Cytokine, Chemokine, Immune system, Cell biology and Allergic inflammation.
His work in Eosinophil peroxidase addresses subjects such as Eosinophil cationic protein, which are connected to disciplines such as Disease activity. The study incorporates disciplines such as Receptor, In vitro and Colitis in addition to Inflammation. His Major basic protein research incorporates themes from Eotaxin and Interleukin 5.
James J. Lee mainly focuses on Eosinophil, Immunology, Eosinophil peroxidase, Eosinophilia and Major basic protein. His Eosinophil study combines topics in areas such as Cytokine, Pathology, Cell biology, Colorectal cancer and Allergic inflammation. In Cell biology, James J. Lee works on issues like Gene knockdown, which are connected to Eosinophilic esophagitis.
His study in Asthma, Innate immune system, Immune system, Inflammation and Hemostatic function falls under the purview of Immunology. His Eosinophil peroxidase study combines topics from a wide range of disciplines, such as Eosinophil Granule Proteins, Eosinophil cationic protein and Autoantibody. His Major basic protein research is multidisciplinary, incorporating elements of Eotaxin and Interleukin 5.
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Exosome-mediated transfer of mRNAs and microRNAs is a novel mechanism of genetic exchange between cells
Hadi Valadi;Karin Ekström;Apostolos Bossios;Margareta Sjöstrand.
Nature Cell Biology (2007)
Primary structure, gene organization and polypeptide expression of poliovirus RNA
Naomi Kitamura;Bert L. Semler;Paul G. Rothberg;Glenn R. Larsen.
Catapult-like release of mitochondrial DNA by eosinophils contributes to antibacterial defense
Shida Yousefi;Jeffrey A. Gold;Nicola Andina;James J. Lee.
Nature Medicine (2008)
Defining a link with asthma in mice congenitally deficient in eosinophils.
James J. Lee;Dawn Dimina;Mi Mi P. Macias;Sergei I. Ochkur.
Neutrophil and B cell expansion in mice that lack the murine IL-8 receptor homolog
Grace Cacalano;James Lee;Kristine Kikly;Ann M. Ryan.
Eosinophils are required for the maintenance of plasma cells in the bone marrow
Van Trung Chu;Anja Fröhlich;Gudrun Steinhauser;Tobias Scheel.
Nature Immunology (2011)
Interleukin-5 expression in the lung epithelium of transgenic mice leads to pulmonary changes pathognomonic of asthma.
James J. Lee;Michael P. McGarry;Steven C. Farmer;Karen L. Denzler.
Journal of Experimental Medicine (1997)
Corrigendum: Genetic variants associated with subjective well-being, depressive symptoms, and neuroticism identified through genome-wide analyses (vol 48, pg 624, 2016)
Aysu Okbay;Bart M. L. Baselmans;Jan-Emmanuel De Neve;Patrick Turley.
Nature Genetics (2016)
A tale of two controversies: defining both the role of peroxidases in nitrotyrosine formation in vivo using eosinophil peroxidase and myeloperoxidase-deficient mice, and the nature of peroxidase-generated reactive nitrogen species.
Marie Luise Brennan;Weijia Wu;Xiaoming Fu;Zhongzhu Shen.
Journal of Biological Chemistry (2002)
Fundamental signals that regulate eosinophil homing to the gastrointestinal tract
Anil Mishra;Simon P. Hogan;James J. Lee;Paul S. Foster.
Journal of Clinical Investigation (1999)
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