2017 - Fellow of the American Association for the Advancement of Science (AAAS)
Michael R. Blackburn spends much of his time researching Immunology, Adenosine, Adenosine receptor, Adenosine deaminase and Lung. His studies in Immunology integrate themes in fields like Fibrosis and Mucin. Michael R. Blackburn combines subjects such as Signal transduction, Purinergic signalling and Pathogenesis with his study of Adenosine.
His Purinergic signalling research integrates issues from Adenosine A3 receptor and Cell biology. His biological study spans a wide range of topics, including Molecular biology and Cellular differentiation. His biological study deals with issues like Immunodeficiency, which deal with fields such as Spleen and Deficient mouse.
His main research concerns Adenosine, Immunology, Adenosine deaminase, Adenosine receptor and Internal medicine. His study in Adenosine is interdisciplinary in nature, drawing from both Adenosine A3 receptor, Receptor, Signal transduction, Purinergic signalling and Adenosine A2B receptor. His research in Immunology focuses on subjects like Lung, which are connected to Fibrosis and Pathology.
As part of the same scientific family, Michael R. Blackburn usually focuses on Adenosine deaminase, concentrating on Molecular biology and intersecting with Thymocyte. The study incorporates disciplines such as Pharmacology and Nucleoside in addition to Adenosine receptor. His study looks at the intersection of Internal medicine and topics like Endocrinology with Decidua.
Michael R. Blackburn mainly investigates Adenosine, Fibrosis, Cancer research, Adenosine A2B receptor and Lung. Michael R. Blackburn works in the field of Adenosine, focusing on Adenosine deaminase in particular. His work deals with themes such as Idiopathic pulmonary fibrosis and Pathogenesis, which intersect with Fibrosis.
His Adenosine A2B receptor research includes themes of Hypoxia and Pulmonary edema. The Lung study combines topics in areas such as Nucleotidase, Immunology and Pathology. Michael R. Blackburn specializes in Immunology, namely Inflammation.
The scientist’s investigation covers issues in Adenosine, Lung, Inflammation, Adenosine A2B receptor and Pulmonary fibrosis. His work on Adenosine deaminase as part of his general Adenosine study is frequently connected to Nucleoside transporter, thereby bridging the divide between different branches of science. The subject of his Inflammation research is within the realm of Immunology.
His Immunology study combines topics from a wide range of disciplines, such as Cell signaling and Inosine. The concepts of his Adenosine A2B receptor study are interwoven with issues in Adenosine A3 receptor, Endocrinology, Hypoxia and Signal transduction. In his work, Cancer research and Fibroblast is strongly intertwined with Idiopathic pulmonary fibrosis, which is a subfield of Pulmonary fibrosis.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Muc5b is required for airway defence
Michelle G. Roy;Alessandra Livraghi-Butrico;Ashley A. Fletcher;Melissa M. McElwee.
Adenosine receptors in regulation of dendritic cell differentiation and function
Sergey V. Novitskiy;Sergey Ryzhov;Rinat Zaynagetdinov;Anna E. Goldstein.
Mucin Is Produced by Clara Cells in the Proximal Airways of Antigen-Challenged Mice
Christopher M. Evans;Olatunji W. Williams;Michael J. Tuvim;Rupesh Nigam.
American Journal of Respiratory Cell and Molecular Biology (2004)
Role of A2B adenosine receptor signaling in adenosine-dependent pulmonary inflammation and injury
Chun Xiao Sun;Hongyan Zhong;Amir Mohsenin;Eva Morschl.
Journal of Clinical Investigation (2006)
Metabolic Consequences of Adenosine Deaminase Deficiency in Mice Are Associated with Defects in Alveogenesis, Pulmonary Inflammation, and Airway Obstruction
Michael R. Blackburn;Jonathan B. Volmer;Janci L. Thrasher;Hongyan Zhong.
Journal of Experimental Medicine (2000)
Constant darkness is a circadian metabolic signal in mammals
Jianfa Zhang;Krista Kaasik;Michael R Blackburn;Cheng Chi Lee.
Adenosine mediates IL-13-induced inflammation and remodeling in the lung and interacts in an IL-13-adenosine amplification pathway
Michael R. Blackburn;Chun G. Lee;Hays W.J. Young;Zhou Zhu.
Journal of Clinical Investigation (2003)
Adenosine-dependent airway inflammation and hyperresponsiveness in partially adenosine deaminase-deficient mice.
Janci L. Chunn;Hays W. J. Young;Suman K. Banerjee;Giuseppe N. Colasurdo.
Journal of Immunology (2001)
Blockade of IL-6 Trans Signaling Attenuates Pulmonary Fibrosis
Thanh-Thuy T. Le;Harry Karmouty-Quintana;Ernestina Melicoff;Thanh-Truc T. Le.
Journal of Immunology (2014)
Adenosine Deaminase-deficient Mice Generated Using a Two-stage Genetic Engineering Strategy Exhibit a Combined Immunodeficiency
Michael R. Blackburn;Surjit K. Datta;Rodney E. Kellems.
Journal of Biological Chemistry (1998)
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