Her primary areas of investigation include Adenosine, Biochemistry, Nucleotidase, Adenosine receptor and 5'-nucleotidase. Many of her research projects under Adenosine are closely connected to Adenine nucleotide with Adenine nucleotide, tying the diverse disciplines of science together. The concepts of her Adenosine receptor study are interwoven with issues in Immunology and Cell biology.
Her studies in 5'-nucleotidase integrate themes in fields like Immune system, Pharmacology and Ischemic preconditioning. Her study looks at the relationship between Adenosine A3 receptor and fields such as Purinergic signalling, as well as how they intersect with chemical problems. Her work deals with themes such as Cell, Cell type and Function, which intersect with Extracellular.
Linda F. Thompson mainly focuses on Immunology, Molecular biology, Adenosine, Adenosine deaminase and 5'-nucleotidase. Her research in Immunology intersects with topics in Receptor and Disease. Her Molecular biology research is multidisciplinary, relying on both T cell, Thymocyte, T-cell receptor, CD8 and Gene.
Her Adenosine study deals with the bigger picture of Biochemistry. As part of one scientific family, Linda F. Thompson deals mainly with the area of Biochemistry, narrowing it down to issues related to the Function, and often Cell type. Her biological study spans a wide range of topics, including Cell culture and Pharmacology.
The scientist’s investigation covers issues in Immunology, Adenosine, Adenosine receptor, T-cell receptor and Cancer research. Her work in Immunology tackles topics such as Disease which are related to areas like Mediator. Linda F. Thompson works in the field of Adenosine, focusing on Nucleotidase in particular.
Her research integrates issues of Host disease, 5'-nucleotidase and Allogeneic graft in her study of Adenosine receptor. As a part of the same scientific family, she mostly works in the field of T-cell receptor, focusing on Saliva and, on occasion, Gene and Single-cell analysis. Her research investigates the link between T cell and topics such as Germinal center that cross with problems in CD8, Tumor necrosis factor alpha and Molecular biology.
Linda F. Thompson mainly focuses on Cell biology, Immunology, Signal transduction, Kinase and Cell adhesion. Her Cell biology research incorporates elements of Ligand and T-cell receptor. Her Immunology study frequently intersects with other fields, such as Disease.
Her Signal transduction research is multidisciplinary, incorporating elements of Endocrinology, Cellular differentiation, Adenosine receptor, Osteoblast and Receptor. Her Kinase study combines topics from a wide range of disciplines, such as Tumor necrosis factor alpha and Integrin.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Ecto-5′-nucleotidase (CD73) regulation by hypoxia-inducible factor-1 mediates permeability changes in intestinal epithelia
Kristin Synnestvedt;Glenn T. Furuta;Katrina M. Comerford;Nancy Louis.
Journal of Clinical Investigation (2002)
Crucial Role for Ecto-5′-Nucleotidase (CD73) in Vascular Leakage during Hypoxia
Linda F. Thompson;Holger K. Eltzschig;Juan C. Ibla;Juan C. Ibla;C. Justin Van De Wiele.
Journal of Experimental Medicine (2004)
Physiological roles for ecto-5'-nucleotidase (CD73).
Sean P. Colgan;Holger K. Eltzschig;Holger K. Eltzschig;Tobias Eckle;Linda F. Thompson;Linda F. Thompson;Linda F. Thompson.
Purinergic Signalling (2006)
Cardioprotection by Ecto-5′-Nucleotidase (CD73) and A2B Adenosine Receptors
Tobias Eckle;Thomas Krahn;Almut Grenz;David Köhler.
Circulation (2007)
Ecto‐enzyme and signaling functions of lymphocyte CD 7 3
Regina Resta;Yoshio Yamashita;Linda F. Thompson.
Immunological Reviews (1998)
Endogenous adenosine produced during hypoxia attenuates neutrophil accumulation: coordination by extracellular nucleotide metabolism.
Holger K. Eltzschig;Linda F. Thompson;Jorn Karhausen;Richard J. Cotta.
Blood (2004)
CD73 on Tumor Cells Impairs Antitumor T-Cell Responses: A Novel Mechanism of Tumor-Induced Immune Suppression
Dachuan Jin;Jie Fan;Long Wang;Linda F. Thompson.
Cancer Research (2010)
Methotrexate and sulfasalazine promote adenosine release by a mechanism that requires ecto-5'-nucleotidase-mediated conversion of adenine nucleotides.
L Morabito;M C Montesinos;D M Schreibman;L Balter.
Journal of Clinical Investigation (1998)
Identification of very early lymphoid precursors in bone marrow and their regulation by estrogen.
Kay L. Medina;Karla P. Garrett;Linda F. Thompson;Maria Isabel D. Rossi.
Nature Immunology (2001)
A2B adenosine receptor dampens hypoxia-induced vascular leak
Tobias Eckle;Marion Faigle;Almut Grenz;Stefanie Laucher.
Blood (2008)
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