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Bruce N. Cronstein

Bruce N. Cronstein

D-Index & Metrics

Medicine

D-Index
97
Citations
33209
World Ranking
9318
National Ranking
4807

Overview

Bruce N. Cronstein is affiliated with New York University in the United States and has a significant publication record across several fields of study, primarily in medicine and biochemistry, genetics, and molecular biology. Their research spans multiple subfields including physiology, molecular biology, rheumatology, biomedical engineering, and surgery.

The primary research topics addressed by Cronstein include adenosine and purinergic signaling, bone tissue engineering materials, dental implant techniques and outcomes, periodontal regeneration and treatments, osteoarthritis treatment and mechanisms, autoimmune and inflammatory disorders research, as well as cancer-related molecular mechanisms research.

Frequent coauthors collaborating with Cronstein are Lukasz Witek, Paulo G. Coelho, Roberto L. Flores, Bryan N. Becker, and Lars Berglund.

Common publication venues for their work include:

  • Translational Research
  • Purinergic Signalling
  • Plastic & Reconstructive Surgery Global Open
  • The FASEB Journal
  • Scientific Reports

Among recent papers authored or co-authored by Cronstein, notable examples include:

  • Methotrexate and its mechanisms of action in inflammatory arthritis, 2020, Nature Reviews Rheumatology
  • Effects of Acute Colchicine Administration Prior to Percutaneous Coronary Intervention, 2020, Circulation Cardiovascular Interventions
  • International Union of Basic and Clinical Pharmacology. CXII: Adenosine Receptors: A Further Update, 2022, Pharmacological Reviews
  • The effects of caffeine on bone mineral density and fracture risk, 2022, Osteoporosis International
  • Adenosine A2A receptor (A2AR) stimulation enhances mitochondrial metabolism and mitigates reactive oxygen species-mediated mitochondrial injury, 2020, The FASEB Journal

Best Publications

  • Adenosine receptors: therapeutic aspects for inflammatory and immune diseases

    György Haskó;Joel Linden;Bruce Cronstein;Pál Pacher

  • Adenosine 5'-triphosphate and adenosine as endogenous signaling molecules in immunity and inflammation

    M.J.L. Bours;E.L.R. Swennen;F. Di Virgilio;B.N. Cronstein

  • Adenosine: an endogenous regulator of innate immunity

    György Haskó;György Haskó;Bruce N. Cronstein

  • A Mechanism for the Antiinflammatory Effects of Corticosteroids: The Glucocorticoid Receptor Regulates Leukocyte Adhesion to Endothelial Cells and Expression of Endothelial-Leukocyte Adhesion Molecule 1 and Intercellular Adhesion Molecule 1

    Cronstein Bn;Kimmel Sc;Levin Ri;Martiniuk F

  • Adenosine, an endogenous anti-inflammatory agent.

    B. N. Cronstein

  • The antiinflammatory mechanism of methotrexate. Increased adenosine release at inflamed sites diminishes leukocyte accumulation in an in vivo model of inflammation.

    Bruce N. Cronstein;Dwight Naime;Edward Ostad

  • Low-Dose Methotrexate: A Mainstay in the Treatment of Rheumatoid Arthritis

    Bruce N. Cronstein

  • Adenosine: a physiological modulator of superoxide anion generation by human neutrophils.

    B N Cronstein;S B Kramer;G Weissmann;R Hirschhorn

  • Molecular action of methotrexate in inflammatory diseases

    Edwin S L Chan;Bruce N Cronstein

  • Adenosine: an endogenous inhibitor of neutrophil-mediated injury to endothelial cells.

    B N Cronstein;R I Levin;J Belanoff;G Weissmann

  • Neutrophil adherence to endothelium is enhanced via adenosine A1 receptors and inhibited via adenosine A2 receptors.

    B N Cronstein;R I Levin;M Philips;R Hirschhorn

  • The adenosine/neutrophil paradox resolved: human neutrophils possess both A1 and A2 receptors that promote chemotaxis and inhibit O2 generation, respectively.

    B N Cronstein;L Daguma;D Nichols;A J Hutchison

  • Methotrexate--how does it really work?

    Edwin S L Chan;Bruce N Cronstein

  • Adenosine; a physiologic modulator of superoxide anion generation by human neutrophils. Adenosine acts via an A2 receptor on human neutrophils.

    B N Cronstein;E D Rosenstein;S B Kramer;G Weissmann

  • Colchicine alters the quantitative and qualitative display of selectins on endothelial cells and neutrophils.

    B N Cronstein;Y Molad;J Reibman;E Balakhane

  • Methotrexate inhibits neutrophil function by stimulating adenosine release from connective tissue cells.

    Bruce N. Cronstein;Mark A. Eberle;Harry E. Gruber;Richard I. Levin

  • Methotrexate and its mechanisms of action in inflammatory arthritis.

    Bruce N. Cronstein;Thomas M. Aune

  • Regulation of inflammation by adenosine.

    György Haskó;Bruce N Cronstein

  • Understanding the mechanisms of action of methotrexate: implications for the treatment of rheumatoid arthritis.

    Henghe Tian;Bruce N Cronstein

  • The adhesion molecules of inflammation.

    Bruce N. Cronstein;Gerald Weissmann

Frequent Co-Authors

Gerald Weissmann
Gerald Weissmann New York University
Paulo G. Coelho
Paulo G. Coelho University of Miami
Rochelle Hirschhorn
Rochelle Hirschhorn New York University
György Haskó
György Haskó Columbia University
Jill P. Buyon
Jill P. Buyon New York University
Steven B. Abramson
Steven B. Abramson New York University
Kathryn J. Moore
Kathryn J. Moore New York University Langone Medical Center
Jan Vilcek
Jan Vilcek New York University
Michael A. Schwarzschild
Michael A. Schwarzschild Harvard University
David Fenyö
David Fenyö New York University

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