D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Medicine D-index 78 Citations 36,057 211 World Ranking 12628 National Ranking 6576
Biology and Biochemistry D-index 81 Citations 36,641 222 World Ranking 2394 National Ranking 1295

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • Internal medicine
  • Inflammation

Her scientific interests lie mostly in Cell biology, Immunology, Receptor, Cholesterol and Scavenger receptor. As part of one scientific family, Kathryn J. Moore deals mainly with the area of Cell biology, narrowing it down to issues related to the Microglia, and often LYN, FYN, Protein kinase A, Proto-oncogene tyrosine-protein kinase Src and Senile plaques. Her study in Immunology focuses on Inflammation, NALP3, AIM2 and Proinflammatory cytokine.

Her work focuses on many connections between Receptor and other disciplines, such as Macrophage, that overlap with her field of interest in Cytokine and Immune system. Kathryn J. Moore interconnects miR-33 and ABCA1 in the investigation of issues within Cholesterol. Kathryn J. Moore has researched Scavenger receptor in several fields, including Phagocytosis, CD36 and Signal transduction.

Her most cited work include:

  • NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals (2205 citations)
  • NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals (2205 citations)
  • PPARγ Is Required for the Differentiation of Adipose Tissue In Vivo and In Vitro (1656 citations)

What are the main themes of her work throughout her whole career to date?

Kathryn J. Moore focuses on Cell biology, Inflammation, Immunology, Internal medicine and Macrophage. Her biological study deals with issues like Lipid metabolism, which deal with fields such as Lipid droplet. Kathryn J. Moore combines subjects such as Adipose tissue macrophages, Insulin resistance, Receptor and Phenotype with her study of Inflammation.

In her research on the topic of Immunology, Cancer research is strongly related with Netrin. Kathryn J. Moore focuses mostly in the field of Internal medicine, narrowing it down to topics relating to Endocrinology and, in certain cases, Apolipoprotein E and Cell. As a member of one scientific family, Kathryn J. Moore mostly works in the field of Cholesterol, focusing on ABCA1 and, on occasion, Liver X receptor.

She most often published in these fields:

  • Cell biology (35.45%)
  • Inflammation (29.09%)
  • Immunology (27.27%)

What were the highlights of her more recent work (between 2018-2021)?

  • Inflammation (29.09%)
  • Cell biology (35.45%)
  • Macrophage (24.09%)

In recent papers she was focusing on the following fields of study:

Kathryn J. Moore spends much of her time researching Inflammation, Cell biology, Macrophage, Internal medicine and Immune system. Her Inflammation study combines topics in areas such as Diabetes mellitus, Cytokine and Bioinformatics. Her studies in Cell biology integrate themes in fields like Oxidative stress, Psychological repression, microRNA, Long non-coding RNA and Co activator.

Her study in Macrophage is interdisciplinary in nature, drawing from both Glycolysis, Tumor necrosis factor alpha, Immunology and Myeloid cells. Her work deals with themes such as Endocrinology, Oncology and MEDLINE, which intersect with Internal medicine. Her Endocrinology research integrates issues from Proinflammatory cytokine, Cell and Lipid localization.

Between 2018 and 2021, her most popular works were:

  • The long noncoding RNA CHROME regulates cholesterol homeostasis in primate. (53 citations)
  • Mycobacterium tuberculosis Limits Host Glycolysis and IL-1β by Restriction of PFK-M via MicroRNA-21. (29 citations)
  • Single-Cell RNA Sequencing of Visceral Adipose Tissue Leukocytes Reveals that Caloric Restriction Following Obesity Promotes the Accumulation of a Distinct Macrophage Population with Features of Phagocytic Cells. (26 citations)

In her most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Inflammation

Her main research concerns Inflammation, Macrophage, Immunology, Innate immune system and Cell biology. Her biological study spans a wide range of topics, including Adipose tissue, Cell and Immune system. Her Macrophage research incorporates themes from Platelet activation, Platelet and Cytokine.

Her Immunology study frequently draws connections between adjacent fields such as Atherosclerosis regression. Her research integrates issues of Cancer, Breast cancer, Cancer research, Myocardial infarction and Monocyte in her study of Innate immune system. Her Cell biology study incorporates themes from Gene knockdown, Glycolysis, RNA, Transcription factor and ABCA1.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals

Peter Duewell;Hajime Kono;Katey J. Rayner;Katey J. Rayner;Cherilyn M. Sirois.
Nature (2010)

3360 Citations

Macrophages in the pathogenesis of atherosclerosis.

Kathryn J. Moore;Ira Tabas.
Cell (2011)

2487 Citations

PPARγ Is Required for the Differentiation of Adipose Tissue In Vivo and In Vitro

Evan D Rosen;Pasha Sarraf;Amy E Troy;Gary Bradwin.
Molecular Cell (1999)

2409 Citations

The NALP3 inflammasome is involved in the innate immune response to amyloid-beta.

Annett Halle;Veit Hornung;Gabor C Petzold;Cameron R Stewart.
Nature Immunology (2008)

2321 Citations

Macrophages in atherosclerosis: a dynamic balance

Kathryn J. Moore;Frederick J. Sheedy;Edward A. Fisher.
Nature Reviews Immunology (2013)

2013 Citations

CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer

Cameron R Stewart;Lynda M Stuart;Kim Wilkinson;Janine M van Gils.
Nature Immunology (2010)

1460 Citations

MiR-33 Contributes to the Regulation of Cholesterol Homeostasis

Katey J. Rayner;Yajaira Suárez;Alberto Dávalos;Saj Parathath.
Science (2010)

1307 Citations

Scavenger Receptors Class A-I/II and CD36 Are the Principal Receptors Responsible for the Uptake of Modified Low Density Lipoprotein Leading to Lipid Loading in Macrophages

Vidya V. Kunjathoor;Maria Febbraio;Eugene A. Podrez;Kathryn J. Moore.
Journal of Biological Chemistry (2002)

1271 Citations

Reduced atherosclerosis in MyD88-null mice links elevated serum cholesterol levels to activation of innate immunity signaling pathways.

Harry Björkbacka;Vidya V Kunjathoor;Kathryn J Moore;Stephanie Koehn.
Nature Medicine (2004)

784 Citations

Inhibition of miR-33a/b in non-human primates raises plasma HDL and lowers VLDL triglycerides

Katey J. Rayner;Christine C. Esau;Farah N. Hussain;Allison L. McDaniel.
Nature (2011)

778 Citations

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