World's Best Scientists 2026 revealed!

D-Index & Metrics

Biology and Biochemistry

D-Index
91
Citations
49941
World Ranking
2270
National Ranking
1217

Medicine

D-Index
91
Citations
50007
World Ranking
11521
National Ranking
5909

Overview

Kathryn J. Moore is affiliated with the New York University Langone Medical Center in the United States. Their research spans multiple fields within biomedical science, including Medicine, Biochemistry, Genetics and Molecular Biology, and Immunology and Microbiology. The subfields they focus on include Immunology, Molecular Biology, Cancer Research, Cardiology and Cardiovascular Medicine, and Oncology.

The main topics of their work cover a range of areas related to cardiovascular and cancer biology, such as:

  • Atherosclerosis and Cardiovascular Diseases
  • Immune cells in cancer
  • Cancer-related molecular mechanisms research
  • Adipokines, Inflammation, and Metabolic Diseases
  • RNA modifications and cancer
  • RNA Research and Splicing
  • MicroRNA in disease regulation

Among their recent papers are several significant contributions to various scientific journals:

  • "Long non-coding RNAs: definitions, functions, challenges and recommendations," published in 2023 in Nature Reviews Molecular Cell Biology
  • "Myocardial infarction accelerates breast cancer via innate immune reprogramming," published in 2020 in Nature Medicine
  • "Regulatory T Cells License Macrophage Pro-Resolving Functions During Atherosclerosis Regression," published in 2020 in Circulation Research
  • "Mycobacterium tuberculosis Limits Host Glycolysis and IL-1β by Restriction of PFK-M via MicroRNA-21," published in 2020 in Cell Reports
  • "Chronic stress primes innate immune responses in mice and humans," published in 2021 in Cell Reports

The scientist frequently publishes in notable venues such as:

  • Arteriosclerosis Thrombosis and Vascular Biology
  • Circulation Research
  • Nature Cardiovascular Research
  • Circulation
  • Proceedings of the National Academy of Sciences

Collaboration has been a consistent aspect of their career, with frequent co-authors including:

  • Coen van Solingen
  • Alexandra Newman
  • Morgane Gourvest
  • Chiara Giannarelli
  • Edward A. Fisher

Best Publications

  • NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals

    Peter Duewell;Hajime Kono;Katey J. Rayner;Katey J. Rayner;Cherilyn M. Sirois

  • Macrophages in the pathogenesis of atherosclerosis.

    Kathryn J. Moore;Ira Tabas

  • The NALP3 inflammasome is involved in the innate immune response to amyloid-beta.

    Annett Halle;Veit Hornung;Gabor C Petzold;Cameron R Stewart

  • Macrophages in atherosclerosis: a dynamic balance

    Kathryn J. Moore;Frederick J. Sheedy;Edward A. Fisher

  • PPARγ Is Required for the Differentiation of Adipose Tissue In Vivo and In Vitro

    Evan D Rosen;Pasha Sarraf;Amy E Troy;Gary Bradwin

  • CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer

    Cameron R Stewart;Lynda M Stuart;Kim Wilkinson;Janine M van Gils

  • MiR-33 Contributes to the Regulation of Cholesterol Homeostasis

    Katey J. Rayner;Yajaira Suárez;Alberto Dávalos;Saj Parathath

  • Scavenger Receptors Class A-I/II and CD36 Are the Principal Receptors Responsible for the Uptake of Modified Low Density Lipoprotein Leading to Lipid Loading in Macrophages

    Vidya V. Kunjathoor;Maria Febbraio;Eugene A. Podrez;Kathryn J. Moore

  • CD36 coordinates NLRP3 inflammasome activation by facilitating intracellular nucleation of soluble ligands into particulate ligands in sterile inflammation

    Frederick J Sheedy;Alena Grebe;Katey J Rayner;Parisa Kalantari

  • Inhibition of miR-33a/b in non-human primates raises plasma HDL and lowers VLDL triglycerides

    Katey J. Rayner;Christine C. Esau;Farah N. Hussain;Allison L. McDaniel

  • Reduced atherosclerosis in MyD88-null mice links elevated serum cholesterol levels to activation of innate immunity signaling pathways.

    Harry Björkbacka;Vidya V Kunjathoor;Kathryn J Moore;Stephanie Koehn

  • Antagonism of miR-33 in mice promotes reverse cholesterol transport and regression of atherosclerosis

    Katey J. Rayner;Frederick J. Sheedy;Christine C. Esau;Farah N. Hussain

  • miR-33a/b contribute to the regulation of fatty acid metabolism and insulin signaling.

    Alberto Dávalos;Leigh Goedeke;Peter Smibert;Cristina M. Ramírez

  • Scavenger Receptors in Atherosclerosis: Beyond Lipid Uptake

    Kathryn J. Moore;Mason W. Freeman

  • The role of PPAR-γ in macrophage differentiation and cholesterol uptake

    Kathryn J. Moore;Evan D. Rosen;Michael L. Fitzgerald;Felix Randow

  • MicroRNA Regulation of Atherosclerosis.

    Mark W. Feinberg;Kathryn J. Moore

  • CD36 Mediates the Innate Host Response to β-Amyloid

    Joseph B. El Khoury;Kathryn J. Moore;Terry K. Means;Josephine Leung

  • Atherogenic Lipids and Lipoproteins Trigger CD36-TLR2-Dependent Apoptosis in Macrophages Undergoing Endoplasmic Reticulum Stress

    Tracie A. Seimon;Marissa J. Nadolski;Xianghai Liao;Jorge Magallon

  • Response to Staphylococcus aureus requires CD36-mediated phagocytosis triggered by the COOH-terminal cytoplasmic domain

    Lynda M. Stuart;Jiusheng Deng;Jessica M. Silver;Kazue Takahashi

  • Erratum: NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals (Nature (2010) 464 (1357-1361))

    Peter Duewell;Hajime Kono;Katey J. Rayner;Cherilyn M. Sirois

Frequent Co-Authors

Edward A. Fisher
Edward A. Fisher New York University
Mason W. Freeman
Mason W. Freeman Harvard University
Yajaira Suárez
Yajaira Suárez Yale University
Andrew D. Luster
Andrew D. Luster Harvard University
Eicke Latz
Eicke Latz German Rheumatism Research Centre
Ira Tabas
Ira Tabas Columbia University
Douglas T. Golenbock
Douglas T. Golenbock University of Massachusetts Chan Medical School
P'ng Loke
P'ng Loke National Institutes of Health
Katherine A. Fitzgerald
Katherine A. Fitzgerald University of Massachusetts Chan Medical School

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