Her scientific interests lie mostly in Cell biology, Immunology, Receptor, Cholesterol and Scavenger receptor. As part of one scientific family, Kathryn J. Moore deals mainly with the area of Cell biology, narrowing it down to issues related to the Microglia, and often LYN, FYN, Protein kinase A, Proto-oncogene tyrosine-protein kinase Src and Senile plaques. Her study in Immunology focuses on Inflammation, NALP3, AIM2 and Proinflammatory cytokine.
Her work focuses on many connections between Receptor and other disciplines, such as Macrophage, that overlap with her field of interest in Cytokine and Immune system. Kathryn J. Moore interconnects miR-33 and ABCA1 in the investigation of issues within Cholesterol. Kathryn J. Moore has researched Scavenger receptor in several fields, including Phagocytosis, CD36 and Signal transduction.
Kathryn J. Moore focuses on Cell biology, Inflammation, Immunology, Internal medicine and Macrophage. Her biological study deals with issues like Lipid metabolism, which deal with fields such as Lipid droplet. Kathryn J. Moore combines subjects such as Adipose tissue macrophages, Insulin resistance, Receptor and Phenotype with her study of Inflammation.
In her research on the topic of Immunology, Cancer research is strongly related with Netrin. Kathryn J. Moore focuses mostly in the field of Internal medicine, narrowing it down to topics relating to Endocrinology and, in certain cases, Apolipoprotein E and Cell. As a member of one scientific family, Kathryn J. Moore mostly works in the field of Cholesterol, focusing on ABCA1 and, on occasion, Liver X receptor.
Kathryn J. Moore spends much of her time researching Inflammation, Cell biology, Macrophage, Internal medicine and Immune system. Her Inflammation study combines topics in areas such as Diabetes mellitus, Cytokine and Bioinformatics. Her studies in Cell biology integrate themes in fields like Oxidative stress, Psychological repression, microRNA, Long non-coding RNA and Co activator.
Her study in Macrophage is interdisciplinary in nature, drawing from both Glycolysis, Tumor necrosis factor alpha, Immunology and Myeloid cells. Her work deals with themes such as Endocrinology, Oncology and MEDLINE, which intersect with Internal medicine. Her Endocrinology research integrates issues from Proinflammatory cytokine, Cell and Lipid localization.
Her main research concerns Inflammation, Macrophage, Immunology, Innate immune system and Cell biology. Her biological study spans a wide range of topics, including Adipose tissue, Cell and Immune system. Her Macrophage research incorporates themes from Platelet activation, Platelet and Cytokine.
Her Immunology study frequently draws connections between adjacent fields such as Atherosclerosis regression. Her research integrates issues of Cancer, Breast cancer, Cancer research, Myocardial infarction and Monocyte in her study of Innate immune system. Her Cell biology study incorporates themes from Gene knockdown, Glycolysis, RNA, Transcription factor and ABCA1.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals
Peter Duewell;Hajime Kono;Katey J. Rayner;Katey J. Rayner;Cherilyn M. Sirois.
Nature (2010)
Macrophages in the pathogenesis of atherosclerosis.
Kathryn J. Moore;Ira Tabas.
Cell (2011)
PPARγ Is Required for the Differentiation of Adipose Tissue In Vivo and In Vitro
Evan D Rosen;Pasha Sarraf;Amy E Troy;Gary Bradwin.
Molecular Cell (1999)
The NALP3 inflammasome is involved in the innate immune response to amyloid-beta.
Annett Halle;Veit Hornung;Gabor C Petzold;Cameron R Stewart.
Nature Immunology (2008)
Macrophages in atherosclerosis: a dynamic balance
Kathryn J. Moore;Frederick J. Sheedy;Edward A. Fisher.
Nature Reviews Immunology (2013)
CD36 ligands promote sterile inflammation through assembly of a Toll-like receptor 4 and 6 heterodimer
Cameron R Stewart;Lynda M Stuart;Kim Wilkinson;Janine M van Gils.
Nature Immunology (2010)
MiR-33 Contributes to the Regulation of Cholesterol Homeostasis
Katey J. Rayner;Yajaira Suárez;Alberto Dávalos;Saj Parathath.
Science (2010)
Scavenger Receptors Class A-I/II and CD36 Are the Principal Receptors Responsible for the Uptake of Modified Low Density Lipoprotein Leading to Lipid Loading in Macrophages
Vidya V. Kunjathoor;Maria Febbraio;Eugene A. Podrez;Kathryn J. Moore.
Journal of Biological Chemistry (2002)
Reduced atherosclerosis in MyD88-null mice links elevated serum cholesterol levels to activation of innate immunity signaling pathways.
Harry Björkbacka;Vidya V Kunjathoor;Kathryn J Moore;Stephanie Koehn.
Nature Medicine (2004)
Inhibition of miR-33a/b in non-human primates raises plasma HDL and lowers VLDL triglycerides
Katey J. Rayner;Christine C. Esau;Farah N. Hussain;Allison L. McDaniel.
Nature (2011)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
New York University
Harvard University
Yale University
Columbia University
Harvard University
University of Bonn
National Institutes of Health
University of Massachusetts Medical School
University of Massachusetts Medical School
University of Melbourne
University of British Columbia
Tokyo University of Science
Fudan University
Queen's University Belfast
Shanxi University
Korea Institute for Advanced Study
Estonian University of Life Sciences
Università Campus Bio-Medico
The University of Texas at Austin
Institut Universitaire de France
Chinese Academy of Sciences
Boston Children's Hospital
University of Salzburg
Baylor College of Medicine
Karolinska Institute
University of Cape Town