Member of the European Academy of Sciences and Arts
His primary areas of investigation include Immunology, Inflammation, Innate immune system, Immune system and Epitope. In Immunology, he works on issues like Receptor, which are connected to T cell. In the subject of general Inflammation, his work in Proinflammatory cytokine is often linked to Dna genetics, thereby combining diverse domains of study.
His Innate immune system research includes themes of Oxidative stress and Lung injury. The concepts of his Immune system study are interwoven with issues in DNA, Pathogenesis and Antigen. His Epitope research is multidisciplinary, relying on both Phosphorylcholine, Disease, Scavenger receptor and Immunity.
Christoph J. Binder focuses on Immunology, Internal medicine, Inflammation, Antibody and Epitope. Immune system, Antigen, Innate immune system, Immunoglobulin M and Autoantibody are the core of his Immunology study. His work deals with themes such as Gastroenterology, Endocrinology and Cardiology, which intersect with Internal medicine.
His studies deal with areas such as Context, Steatohepatitis, Scavenger receptor and Cell biology as well as Inflammation. His work carried out in the field of Antibody brings together such families of science as Receptor, Apolipoprotein E and Germline. His study in Epitope is interdisciplinary in nature, drawing from both Molecular biology, Tissue homeostasis and Immunity.
Christoph J. Binder spends much of his time researching Antibody, Immunology, Immune system, Inflammation and Internal medicine. His study in the fields of Epitope, Seroprevalence, Serology and Titer under the domain of Antibody overlaps with other disciplines such as Severe acute respiratory syndrome coronavirus 2. Christoph J. Binder has researched Immunology in several fields, including Apolipoprotein E, Coagulation and Kexin.
His primary area of study in Immune system is in the field of Innate immune system. Christoph J. Binder studies Proinflammatory cytokine which is a part of Inflammation. His Internal medicine research is multidisciplinary, incorporating elements of Gastroenterology, Endocrinology, Oncology and Cardiology.
Christoph J. Binder mainly focuses on Antibody, Seroprevalence, Internal medicine, Severe acute respiratory syndrome coronavirus 2 and B cell. His Antibody study combines topics in areas such as Tissue homeostasis and Alternative splicing. His studies in Seroprevalence integrate themes in fields like Cohort and Antigen.
His biological study spans a wide range of topics, including Titer and Neutralization. His Internal medicine research focuses on Epidemiology, Allograft inflammatory factor 1 and Inflammation. Christoph J. Binder interconnects Filamentous actin, Cytoskeleton, Cell growth, Immune system and Actin in the investigation of issues within B cell.
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Interleukin-4-dependent production of PPAR-γ ligands in macrophages by 12/15-lipoxygenase
Jannet T. Huang;John S. Welch;Mercedes Ricote;Christoph J. Binder.
Identification of Oxidative Stress and Toll-like Receptor 4 Signaling as a Key Pathway of Acute Lung Injury
Yumiko Imai;Keiji Kuba;Keiji Kuba;G. Greg Neely;Rubina Yaghubian-Malhami.
Innate and acquired immunity in atherogenesis.
Christoph J Binder;Mi-Kyung Chang;Peter X Shaw;Yury I Miller.
Nature Medicine (2002)
Pneumococcal vaccination decreases atherosclerotic lesion formation: molecular mimicry between Streptococcus pneumoniae and oxidized LDL.
Christoph J Binder;Sohvi Hörkkö;Sohvi Hörkkö;Asheesh Dewan;Mi-Kyung Chang.
Nature Medicine (2003)
Differential inhibition of macrophage foam-cell formation and atherosclerosis in mice by PPARα, β/δ, and γ
Andrew C. Li;Christoph J. Binder;Alejandra Gutierrez;Kathleen K. Brown.
Journal of Clinical Investigation (2004)
C-reactive protein binds to both oxidized LDL and apoptotic cells through recognition of a common ligand: Phosphorylcholine of oxidized phospholipids.
Mi-Kyung Chang;Christoph J. Binder;Michael Torzewski;Joseph L. Witztum.
Proceedings of the National Academy of Sciences of the United States of America (2002)
Oxidative damage in multiple sclerosis lesions
Lukas Haider;Marie T. Fischer;Josa M. Frischer;Jan Bauer.
Oxidation-Specific Epitopes Are Danger-Associated Molecular Patterns Recognized by Pattern Recognition Receptors of Innate Immunity
Yury I. Miller;Soo Ho Choi;Philipp Wiesner;Longhou Fang.
Circulation Research (2011)
IL-5 links adaptive and natural immunity specific for epitopes of oxidized LDL and protects from atherosclerosis
Christoph J. Binder;Karsten Hartvigsen;Mi-Kyung Chang;Marina Miller.
Journal of Clinical Investigation (2004)
Minimally modified LDL binds to CD14, induces macrophage spreading via TLR4/MD-2, and inhibits phagocytosis of apoptotic cells.
Yury I. Miller;Suganya Viriyakosol;Christoph J. Binder;James R. Feramisco.
Journal of Biological Chemistry (2003)
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