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Molecular Biology
Hungary
2026
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Genetics and Molecular Biology
Hungary
2024

D-Index & Metrics

Molecular Biology

D-Index
68
Citations
26583
World Ranking
1496
National Ranking
3

Research.com Recognitions

  • 2026 - Research.com Molecular Biology in Hungary Leader Award
  • 2025 - Research.com Molecular Biology in Hungary Leader Award
  • 2024 - Research.com Genetics and Molecular Biology in Hungary Leader Award
  • 2023 - Research.com Genetics and Molecular Biology in Hungary Leader Award
  • 2022 - Research.com Genetics and Molecular Biology in Hungary Leader Award
  • 2012 - Member of Academia Europaea
  • Member of the European Molecular Biology Organization (EMBO)
  • Member of the European Molecular Biology Organization (EMBO)

Overview

Laszlo Nagy is affiliated with the University of Debrecen in Hungary. Their research spans multiple fields, primarily focusing on medicine and biochemistry, genetics, and molecular biology. Within these, main subfields include molecular biology, cardiology and cardiovascular medicine, immunology, genetics, and physiology.

Their scholarly work covers a range of topics such as:

  • Immune cells in cancer
  • Epigenetics and DNA Methylation
  • Cardiac Arrhythmias and Treatments
  • Atrial Fibrillation Management and Outcomes
  • Cardiac pacing and defibrillation studies
  • Cardiac electrophysiology and arrhythmias
  • Immune Cell Function and Interaction

Frequent co-authors contributing to Nagy's research include Zoltán Csanádi, László Halász, Andreas Patsalos, Gergely Nagy, and Zsolt Czimmerer.

Their work is regularly published in several research venues, with multiple contributions to:

  • Journal of Biological Chemistry
  • Journal of Clinical Investigation
  • Frontiers in Endocrinology
  • The Journal of Immunology
  • Cardiologia Hungarica

Notable recent papers authored or co-authored by Laszlo Nagy include:

  • "A growth factor-expressing macrophage subpopulation orchestrates regenerative inflammation via GDF-15" (2021) in The Journal of Experimental Medicine
  • "The epigenetic state of IL-4-polarized macrophages enables inflammatory cistromic expansion and extended synergistic response to TLR ligands" (2022) in Immunity
  • "Regenerative inflammation: When immune cells help to re-build tissues" (2022) in FEBS Journal
  • "The transcription factor EGR2 is the molecular linchpin connecting STAT6 activation to the late, stable epigenomic program of alternative macrophage polarization" (2020) in Genes & Development
  • "Myeloid ALX/FPR2 regulates vascularization following tissue injury" (2020) in Proceedings of the National Academy of Sciences

Laszlo Nagy has been recognized by election to prestigious bodies including membership in the Academia Europaea since 2012 and the European Molecular Biology Organization (EMBO).

Best Publications

  • PPARγ Promotes Monocyte/Macrophage Differentiation and Uptake of Oxidized LDL

    Peter Tontonoz;Laszlo Nagy;Laszlo Nagy;Jacqueline G.A Alvarez;Vilmos A Thomazy

  • Oxidized LDL Regulates Macrophage Gene Expression through Ligand Activation of PPARγ

    Laszlo Nagy;Laszlo Nagy;Peter Tontonoz;Peter Tontonoz;Jacqueline G.A Alvarez;Hongwu Chen

  • Nuclear Receptor Coactivator ACTR Is a Novel Histone Acetyltransferase and Forms a Multimeric Activation Complex with P/CAF and CBP/p300

    Hongwu Chen;Richard J Lin;Richard J Lin;R.Louis Schiltz;Debabrata Chakravarti

  • A PPARγ-LXR-ABCA1 Pathway in Macrophages Is Involved in Cholesterol Efflux and Atherogenesis

    Ajay Chawla;William A. Boisvert;Chih Hao Lee;Bryan A. Laffitte

  • Nuclear receptor repression mediated by a complex containing SMRT, mSin3A, and histone deacetylase.

    Laszlo Nagy;Hung Ying Kao;Debabrata Chakravarti;Richard J. Lin

  • PPAR-gamma dependent and independent effects on macrophage-gene expression in lipid metabolism and inflammation.

    Ajay Chawla;Yaacov Barak;Laszlo Nagy;Debbie Liao

  • PPARs are a unique set of fatty acid regulated transcription factors controlling both lipid metabolism and inflammation.

    Tamas Varga;Zsolt Czimmerer;Laszlo Nagy;Laszlo Nagy

  • Mechanism of corepressor binding and release from nuclear hormone receptors.

    Laszlo Nagy;Laszlo Nagy;Hung Ying Kao;James D. Love;James D. Love;Chuan Li

  • Structural basis for the activation of PPARγ by oxidized fatty acids

    Toshimasa Itoh;Louise Fairall;Kush Amin;Yuka Inaba

  • Mechanism of the nuclear receptor molecular switch

    Laszlo Nagy;John W.R. Schwabe

  • STAT6 Transcription Factor Is a Facilitator of the Nuclear Receptor PPARγ-Regulated Gene Expression in Macrophages and Dendritic Cells

    Attila Szanto;Balint L. Balint;Zsuzsanna S. Nagy;Zsuzsanna S. Nagy;Endre Barta;Endre Barta

  • Role for Peroxisome Proliferator-Activated Receptor α in Oxidized Phospholipid–Induced Synthesis of Monocyte Chemotactic Protein-1 and Interleukin-8 by Endothelial Cells

    Hans Lee;Weibin Shi;Peter Tontonoz;Peter Tontonoz;Shirley Wang

  • Retinoid X receptors: X-ploring their (patho)physiological functions

    A. Szanto;V. Narkar;Q. Shen;Ivan Peter Uray

  • Structural Basis for the Activation of Pparg by Oxidised Fatty Acids

    T. Itoh;L. Fairall;K. Amin;Y. Inaba

  • ACTIVATION OF RETINOID X RECEPTORS INDUCES APOPTOSIS IN HL-60 CELL LINES

    László Nagy;Vilmos A. Thomázy;Gregory L. Shipley;László Fésüs

  • 1,25-Dihydroxyvitamin D3 Is an autonomous regulator of the transcriptional changes leading to a tolerogenic dendritic cell phenotype

    Lajos Széles;Gábor Keresztes;Dániel Töröcsik;Zoltán Balajthy

  • PPARγ controls CD1d expression by turning on retinoic acid synthesis in developing human dendritic cells

    Istvan Szatmari;Attila Pap;Ralphz Rühl;Jiang Xing Ma

  • Nuclear Hormone Receptors Enable Macrophages and Dendritic Cells to Sense Their Lipid Environment and Shape Their Immune Response

    Laszlo Nagy;Attila Szanto;Istvan Szatmari;Lajos Széles

  • Peroxisome Proliferator-activated Receptor γ-regulated ABCG2 Expression Confers Cytoprotection to Human Dendritic Cells

    Istvan Szatmari;György Vámosi;Peter Brazda;Balint L. Balint

  • The Transcription Factor STAT6 Mediates Direct Repression of Inflammatory Enhancers and Limits Activation of Alternatively Polarized Macrophages

    Zsolt Czimmerer;Bence Daniel;Attila Horvath;Dominik Rückerl

Frequent Co-Authors

Ronald M. Evans
Ronald M. Evans Salk Institute for Biological Studies
Peter J. A. Davies
Peter J. A. Davies Texas A&M University
Sascha Sauer
Sascha Sauer Max Delbrück Center for Molecular Medicine
John W. R. Schwabe
John W. R. Schwabe University of Leicester
Peter Tontonoz
Peter Tontonoz University of California, Los Angeles
Éva Rajnavölgyi
Éva Rajnavölgyi University of Debrecen
Zoltán Szekanecz
Zoltán Szekanecz University of Debrecen
Christos C. Zouboulis
Christos C. Zouboulis Medizinische Hochschule Brandenburg Theodor Fontane
Bénédicte Chazaud
Bénédicte Chazaud Inserm : Institut national de la santé et de la recherche médicale
Xianlin Han
Xianlin Han The University of Texas Health Science Center at San Antonio

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