2012 - Distinguished Fellowship Award, American College of Cardiology (ACC)
2003 - Gifted Teacher Award, American College of Cardiology (ACC)
The scientist’s investigation covers issues in Internal medicine, Cardiology, Myocardial infarction, Pathology and Endocrinology. His research brings together the fields of Surgery and Internal medicine. As a part of the same scientific study, Prediman K. Shah usually deals with the Myocardial infarction, concentrating on Vulnerable plaque and frequently concerns with Mass screening.
His biological study spans a wide range of topics, including Inflammation, Thrombosis and Matrix metalloproteinase. His Endocrinology study integrates concerns from other disciplines, such as Apolipoprotein E and Neointima. Prediman K. Shah has included themes like Immunology, Aorta and Recombinant DNA in his Apolipoprotein B study.
Internal medicine, Cardiology, Myocardial infarction, Immunology and Endocrinology are his primary areas of study. Prediman K. Shah regularly links together related areas like Surgery in his Internal medicine studies. His work in Cardiology addresses subjects such as Anesthesia, which are connected to disciplines such as Stroke volume.
His Myocardial infarction study frequently links to related topics such as Radionuclide ventriculography. His Immunology research focuses on Apolipoprotein B and how it relates to Peptide, Apolipoprotein E and Vascular disease. His study looks at the relationship between Inflammation and topics such as Thrombosis, which overlap with Pathology and Thrombus.
His primary areas of investigation include Internal medicine, Immunology, Cardiology, Endocrinology and Immune system. His Internal medicine study frequently draws parallels with other fields, such as Diabetes mellitus. In his study, which falls under the umbrella issue of Immunology, Adeno-associated virus, Aorta and Transplantation is strongly linked to Apolipoprotein E.
As part of his studies on Cardiology, he often connects relevant areas like Surgery. His Immune system study also includes
The scientist’s investigation covers issues in Internal medicine, Immune system, Immunology, Endocrinology and Cardiology. His work on Myocardial infarction, Cholesterol and Hazard ratio as part of general Internal medicine research is often related to Pressure overload, thus linking different fields of science. The Acute coronary syndrome and Unstable angina research Prediman K. Shah does as part of his general Myocardial infarction study is frequently linked to other disciplines of science, such as Adrenergic beta-Antagonists, therefore creating a link between diverse domains of science.
In his research, Apolipoprotein E is intimately related to Cytotoxic T cell, which falls under the overarching field of Immunology. While the research belongs to areas of Endocrinology, he spends his time largely on the problem of Gastroenterology, intersecting his research to questions surrounding Coronary atherosclerosis. His research links Adventitia with Cardiology.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Coronary Plaque Disruption
Erling Falk;Prediman K. Shah;Valentin Fuster.
Circulation (1995)
From vulnerable plaque to vulnerable patient: a call for new definitions and risk assessment strategies: Part II.
Morteza Naghavi;Peter Libby;Erling Falk;S. Ward Casscells;S. Ward Casscells.
Circulation (2003)
Effects of Dalcetrapib in Patients with a Recent Acute Coronary Syndrome
Gregory G. Schwartz;Anders G. Olsson;Markus Abt;Christie M. Ballantyne.
The New England Journal of Medicine (2012)
Pravastatin Treatment Increases Collagen Content and Decreases Lipid Content, Inflammation, Metalloproteinases, and Cell Death in Human Carotid Plaques Implications for Plaque Stabilization
Milita Crisby;Gunilla Nordin-Fredriksson;Prediman K. Shah;Juliana Yano.
Circulation (2001)
From Vulnerable Plaque to Vulnerable Patient
Morteza Naghavi;Peter Libby;Erling Falk;S. Ward Casscells.
Circulation (2003)
The VIVA Trial Vascular Endothelial Growth Factor in Ischemia for Vascular Angiogenesis
Timothy D. Henry;Brian H. Annex;George R. McKendall;Michael A. Azrin.
Circulation (2003)
Lack of Toll-like receptor 4 or myeloid differentiation factor 88 reduces atherosclerosis and alters plaque phenotype in mice deficient in apolipoprotein E
Kathrin S. Michelsen;Michelle H. Wong;Prediman K. Shah;Wenxuan Zhang.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Human monocyte-derived macrophages induce collagen breakdown in fibrous caps of atherosclerotic plaques. Potential role of matrix-degrading metalloproteinases and implications for plaque rupture.
P K Shah;E Falk;J J Badimon;A Fernandez-Ortiz.
Circulation (1995)
Effect of Vasodilator Therapy on Mortality in Chronic Congestive Heart Failure. Results of a Veterans Administration Cooperative Study
J. Cohn;D. Archibald;S. Ziesche;J. Franciosa.
Survey of Anesthesiology (1987)
Toll-Like Receptor-4 Is Expressed by Macrophages in Murine and Human Lipid-Rich Atherosclerotic Plaques and Upregulated by Oxidized LDL
Xiaoou Helen Xu;Prediman K. Shah;Emmanuelle Faure;Ozlem Equils.
Circulation (2001)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
Lund University
Cedars-Sinai Medical Center
Cedars-Sinai Medical Center
University of California, Los Angeles
University of California, Los Angeles
Cedars-Sinai Medical Center
Cedars-Sinai Medical Center
Cedars-Sinai Medical Center
Baylor College of Medicine
University of Southern California
Colorado State University
Google (United States)
Korea Advanced Institute of Science and Technology
Universitat Politècnica de València
National Institutes of Health
Max Planck Society
University of Tokyo
University of Minnesota
Ghent University
Wake Forest University
Vrije Universiteit Amsterdam
Brock University
Duke University
Wake Forest University
World Health Organization
Maastricht University