His main research concerns Internal medicine, Pathology, Thrombosis, Thrombus and Cardiology. His studies in Internal medicine integrate themes in fields like Endocrinology and Type 2 Diabetes Mellitus. His work deals with themes such as Magnetic resonance imaging, Matrix metalloproteinase and Vascular disease, which intersect with Pathology.
The study incorporates disciplines such as Hemostasis, Platelet, Fibrin and Tissue factor in addition to Thrombosis. His Thrombus research includes themes of Artery, Inhalation, Antithrombotic, Heparin and Disease. His biological study spans a wide range of topics, including Restenosis and Surgery.
Juan J. Badimon mainly focuses on Internal medicine, Cardiology, Thrombosis, Thrombus and Platelet. His study brings together the fields of Endocrinology and Internal medicine. His work on Angioplasty, Unstable angina and Clopidogrel as part of his general Cardiology study is frequently connected to In patient, thereby bridging the divide between different branches of science.
Juan J. Badimon interconnects Artery and Pathology in the investigation of issues within Thrombosis. His Pathology study which covers Vascular disease that intersects with Magnetic resonance imaging. His Platelet research incorporates elements of Ex vivo, Pharmacology and Perfusion.
His primary areas of investigation include Internal medicine, Cardiology, Heart failure, Empagliflozin and Pharmacology. His Internal medicine study frequently links to other fields, such as Diabetes mellitus. His Heart failure research incorporates themes from Ketone bodies and Canagliflozin.
Juan J. Badimon combines subjects such as Agonist, Receptor, Platelet, Thrombin and Reperfusion injury with his study of Pharmacology. His work carried out in the field of Platelet brings together such families of science as Ex vivo and Intensive care medicine. The Antithrombotic study combines topics in areas such as Thrombosis, Thrombogenicity, Thrombus and Placebo.
Juan J. Badimon spends much of his time researching Internal medicine, Cardiology, Heart failure, Empagliflozin and Pharmacology. His research brings together the fields of Endocrinology and Internal medicine. His work on Ventricular remodeling, Thrombus and Antithrombotic as part of general Cardiology research is frequently linked to Macitentan, bridging the gap between disciplines.
His research integrates issues of Ketone bodies, Metabolism, Stent and Cholesterol, Lipoprotein in his study of Heart failure. Juan J. Badimon has included themes like Ejection fraction and Canagliflozin in his Empagliflozin study. His study in Pharmacology is interdisciplinary in nature, drawing from both Platelet activation, Platelet, Thrombin and Sodium Glucose Cotransporters.
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The pathogenesis of coronary artery disease and the acute coronary syndromes (1).
Valentin Fuster;Lina Badimon;Juan J. Badimon;James H. Chesebro.
The New England Journal of Medicine (1992)
From Vulnerable Plaque to Vulnerable Patient A Call for New Definitions and Risk Assessment Strategies: Part I
Morteza Naghavi;Peter Libby;Erling Falk;S. Ward Casscells;S. Ward Casscells.
From Vulnerable Plaque to Vulnerable Patient
Morteza Naghavi;Peter Libby;Erling Falk;S. Ward Casscells.
Blood-borne tissue factor: another view of thrombosis.
Peter L. A. Giesen;Ursula Rauch;Bernd Bohrmann;Dorothee Kling.
Proceedings of the National Academy of Sciences of the United States of America (1999)
Coronary Artery Disease: Pathogenesis and Acute Coronary Syndromes
Stephen G. Worthley;Julio I. Osende;Gérard Helft;Juan J. Badimon.
Mount Sinai Journal of Medicine (2001)
Regression of atherosclerotic lesions by high density lipoprotein plasma fraction in the cholesterol-fed rabbit.
J J Badimon;L Badimon;V Fuster.
Journal of Clinical Investigation (1990)
Atherothrombosis and high-risk plaque: part I: evolving concepts.
Valentin Fuster;Pedro R. Moreno;Zahi A. Fayad;Roberto Corti.
Journal of the American College of Cardiology (2005)
Human monocyte-derived macrophages induce collagen breakdown in fibrous caps of atherosclerotic plaques. Potential role of matrix-degrading metalloproteinases and implications for plaque rupture.
P. K. Shah;E. Falk;J. J. Badimon;A. Fernandez-Ortiz.
Syndromes of accelerated atherosclerosis: role of vascular injury and smooth muscle cell proliferation.
John H. Ip;Valentin Fuster;Lina Badimon;Juan Badimon.
Journal of the American College of Cardiology (1990)
Tissue Factor Modulates the Thrombogenicity of Human Atherosclerotic Plaques
V. Toschi;R. Gallo;M. Lettino;J. T. Fallon.
Journal of the American College of Cardiology
(Impact Factor: 27.203)
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