D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 67 Citations 21,398 195 World Ranking 1760 National Ranking 859

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Immune system
  • Internal medicine

Moshe Arditi mainly investigates Toll-like receptor, TLR4, Immunology, Signal transduction and Cell biology. His Toll-like receptor study combines topics from a wide range of disciplines, such as Proinflammatory cytokine and Macrophage. His TLR4 research is multidisciplinary, relying on both Tumor necrosis factor alpha and Intestinal epithelium.

His work is connected to Immune system and Inflammation, as a part of Immunology. His Signal transduction research incorporates themes from Apoptosis, Interleukin 18 and Death domain. His Cell biology research integrates issues from Lipopolysaccharide, Transfection, Myeloid Differentiation Factor 88 and Lymphocyte antigen 96.

His most cited work include:

  • Oxidized Mitochondrial DNA Activates the NLRP3 Inflammasome during Apoptosis (1028 citations)
  • Lack of Toll-like receptor 4 or myeloid differentiation factor 88 reduces atherosclerosis and alters plaque phenotype in mice deficient in apolipoprotein E (851 citations)
  • Decreased Expression of Toll-Like Receptor-4 and MD-2 Correlates with Intestinal Epithelial Cell Protection Against Dysregulated Proinflammatory Gene Expression in Response to Bacterial Lipopolysaccharide (608 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are Immunology, Inflammation, Immune system, Innate immune system and Cell biology. In the field of Immunology, his study on Pathogenesis and TLR4 overlaps with subjects such as Chlamydophila pneumoniae. Moshe Arditi combines subjects such as Pattern recognition receptor and Pharmacology with his study of Inflammation.

Moshe Arditi interconnects Microbiology and Virology in the investigation of issues within Immune system. His research in Innate immune system is mostly concerned with Toll-like receptor. His Cell biology research includes elements of Lipopolysaccharide and Inflammasome.

He most often published in these fields:

  • Immunology (65.20%)
  • Inflammation (25.99%)
  • Immune system (22.47%)

What were the highlights of his more recent work (between 2017-2021)?

  • Immunology (65.20%)
  • Kawasaki disease (17.62%)
  • Vasculitis (18.06%)

In recent papers he was focusing on the following fields of study:

His primary areas of investigation include Immunology, Kawasaki disease, Vasculitis, Inflammasome and Inflammation. His work on Immune system and Superantigen as part of general Immunology research is frequently linked to 2019-20 coronavirus outbreak, bridging the gap between disciplines. The study incorporates disciplines such as Murine model, Myocarditis and Pathogenesis in addition to Vasculitis.

His Inflammasome study incorporates themes from Autophagy, Pathogen, Cell biology, Interleukin and In vivo. His Cell biology research is multidisciplinary, incorporating perspectives in Cell, Downregulation and upregulation and Cholesterol. His studies in Inflammation integrate themes in fields like Interferon, Hormone, Peritoneal cavity and Pharmacology.

Between 2017 and 2021, his most popular works were:

  • Superantigenic character of an insert unique to SARS-CoV-2 spike supported by skewed TCR repertoire in patients with hyperinflammation. (33 citations)
  • An insertion unique to SARS-CoV-2 exhibits superantigenic character strengthened by recent mutations. (30 citations)
  • Intestinal Permeability and IgA Provoke Immune Vasculitis Linked to Cardiovascular Inflammation (28 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Immune system
  • Internal medicine

His main research concerns Immunology, Vasculitis, Immune system, Kawasaki disease and Heart disease. His Immunology study frequently draws connections between adjacent fields such as Shock. His Immune system research is mostly focused on the topic Innate immune system.

As a part of the same scientific family, Moshe Arditi mostly works in the field of Innate immune system, focusing on Interleukin and, on occasion, Inflammasome and Cell biology. His studies deal with areas such as Anakinra, Disease, Myocarditis and Systemic vasculitis as well as Kawasaki disease. His research in Inflammation intersects with topics in Barrier function, Intestinal permeability and Pathogenesis.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Oxidized Mitochondrial DNA Activates the NLRP3 Inflammasome during Apoptosis

Kenichi Shimada;Timothy R. Crother;Justin Karlin;Jargalsaikhan Dagvadorj.
Immunity (2012)

1617 Citations

Lack of Toll-like receptor 4 or myeloid differentiation factor 88 reduces atherosclerosis and alters plaque phenotype in mice deficient in apolipoprotein E

Kathrin S. Michelsen;Michelle H. Wong;Prediman K. Shah;Wenxuan Zhang.
Proceedings of the National Academy of Sciences of the United States of America (2004)

1185 Citations

Decreased Expression of Toll-Like Receptor-4 and MD-2 Correlates with Intestinal Epithelial Cell Protection Against Dysregulated Proinflammatory Gene Expression in Response to Bacterial Lipopolysaccharide

Maria T. Abreu;Puja Vora;Emmanuelle Faure;Lisa S. Thomas.
Journal of Immunology (2001)

971 Citations

Toll-Like Receptor-4 Is Expressed by Macrophages in Murine and Human Lipid-Rich Atherosclerotic Plaques and Upregulated by Oxidized LDL

Xiaoou Helen Xu;Prediman K. Shah;Emmanuelle Faure;Ozlem Equils.
Circulation (2001)

837 Citations

Bacterial Lipopolysaccharide Activates NF-κB through Toll-like Receptor 4 (TLR-4) in Cultured Human Dermal Endothelial Cells DIFFERENTIAL EXPRESSION OF TLR-4 AND TLR-2 IN ENDOTHELIAL CELLS

Emmanuelle Faure;Ozlem Equils;Peter A. Sieling;Lisa Thomas.
Journal of Biological Chemistry (2000)

804 Citations

TLR Signaling in the Gut in Health and Disease

Maria T. Abreu;Masayuki Fukata;Moshe Arditi.
Journal of Immunology (2005)

756 Citations

Bacterial Lipopolysaccharide Activates Nuclear Factor-κB through Interleukin-1 Signaling Mediators in Cultured Human Dermal Endothelial Cells and Mononuclear Phagocytes

Frank X. Zhang;Carsten J. Kirschning;Roberta Mancinelli;Xiao Ping Xu.
Journal of Biological Chemistry (1999)

738 Citations

Cooperation of Toll-like receptor 2 and 6 for cellular activation by soluble tuberculosis factor and Borrelia burgdorferi outer surface protein A lipoprotein: role of Toll-interacting protein and IL-1 receptor signaling molecules in Toll-like receptor 2 signaling.

Yonca Bulut;Emmanuelle Faure;Lisa Thomas;Ozlem Equils.
Journal of Immunology (2001)

597 Citations

Toll-like receptor-4 is required for intestinal response to epithelial injury and limiting bacterial translocation in a murine model of acute colitis

Masayuki Fukata;Kathrin S. Michelsen;Rajaraman Eri;Lisa S. Thomas.
American Journal of Physiology-gastrointestinal and Liver Physiology (2005)

572 Citations

Bacterial Lipopolysaccharide and IFN-γ Induce Toll-Like Receptor 2 and Toll-Like Receptor 4 Expression in Human Endothelial Cells: Role of NF-κB Activation

Emmanuelle Faure;Lisa Thomas;Helen Xu;Andrei E. Medvedev.
Journal of Immunology (2001)

559 Citations

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