Moshe Arditi mainly investigates Toll-like receptor, TLR4, Immunology, Signal transduction and Cell biology. His Toll-like receptor study combines topics from a wide range of disciplines, such as Proinflammatory cytokine and Macrophage. His TLR4 research is multidisciplinary, relying on both Tumor necrosis factor alpha and Intestinal epithelium.
His work is connected to Immune system and Inflammation, as a part of Immunology. His Signal transduction research incorporates themes from Apoptosis, Interleukin 18 and Death domain. His Cell biology research integrates issues from Lipopolysaccharide, Transfection, Myeloid Differentiation Factor 88 and Lymphocyte antigen 96.
His primary areas of study are Immunology, Inflammation, Immune system, Innate immune system and Cell biology. In the field of Immunology, his study on Pathogenesis and TLR4 overlaps with subjects such as Chlamydophila pneumoniae. Moshe Arditi combines subjects such as Pattern recognition receptor and Pharmacology with his study of Inflammation.
Moshe Arditi interconnects Microbiology and Virology in the investigation of issues within Immune system. His research in Innate immune system is mostly concerned with Toll-like receptor. His Cell biology research includes elements of Lipopolysaccharide and Inflammasome.
His primary areas of investigation include Immunology, Kawasaki disease, Vasculitis, Inflammasome and Inflammation. His work on Immune system and Superantigen as part of general Immunology research is frequently linked to 2019-20 coronavirus outbreak, bridging the gap between disciplines. The study incorporates disciplines such as Murine model, Myocarditis and Pathogenesis in addition to Vasculitis.
His Inflammasome study incorporates themes from Autophagy, Pathogen, Cell biology, Interleukin and In vivo. His Cell biology research is multidisciplinary, incorporating perspectives in Cell, Downregulation and upregulation and Cholesterol. His studies in Inflammation integrate themes in fields like Interferon, Hormone, Peritoneal cavity and Pharmacology.
His main research concerns Immunology, Vasculitis, Immune system, Kawasaki disease and Heart disease. His Immunology study frequently draws connections between adjacent fields such as Shock. His Immune system research is mostly focused on the topic Innate immune system.
As a part of the same scientific family, Moshe Arditi mostly works in the field of Innate immune system, focusing on Interleukin and, on occasion, Inflammasome and Cell biology. His studies deal with areas such as Anakinra, Disease, Myocarditis and Systemic vasculitis as well as Kawasaki disease. His research in Inflammation intersects with topics in Barrier function, Intestinal permeability and Pathogenesis.
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Oxidized Mitochondrial DNA Activates the NLRP3 Inflammasome during Apoptosis
Kenichi Shimada;Timothy R. Crother;Justin Karlin;Jargalsaikhan Dagvadorj.
Lack of Toll-like receptor 4 or myeloid differentiation factor 88 reduces atherosclerosis and alters plaque phenotype in mice deficient in apolipoprotein E
Kathrin S. Michelsen;Michelle H. Wong;Prediman K. Shah;Wenxuan Zhang.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Decreased Expression of Toll-Like Receptor-4 and MD-2 Correlates with Intestinal Epithelial Cell Protection Against Dysregulated Proinflammatory Gene Expression in Response to Bacterial Lipopolysaccharide
Maria T. Abreu;Puja Vora;Emmanuelle Faure;Lisa S. Thomas.
Journal of Immunology (2001)
Toll-Like Receptor-4 Is Expressed by Macrophages in Murine and Human Lipid-Rich Atherosclerotic Plaques and Upregulated by Oxidized LDL
Xiaoou Helen Xu;Prediman K. Shah;Emmanuelle Faure;Ozlem Equils.
Bacterial Lipopolysaccharide Activates NF-κB through Toll-like Receptor 4 (TLR-4) in Cultured Human Dermal Endothelial Cells DIFFERENTIAL EXPRESSION OF TLR-4 AND TLR-2 IN ENDOTHELIAL CELLS
Emmanuelle Faure;Ozlem Equils;Peter A. Sieling;Lisa Thomas.
Journal of Biological Chemistry (2000)
TLR Signaling in the Gut in Health and Disease
Maria T. Abreu;Masayuki Fukata;Moshe Arditi.
Journal of Immunology (2005)
Bacterial Lipopolysaccharide Activates Nuclear Factor-κB through Interleukin-1 Signaling Mediators in Cultured Human Dermal Endothelial Cells and Mononuclear Phagocytes
Frank X. Zhang;Carsten J. Kirschning;Roberta Mancinelli;Xiao Ping Xu.
Journal of Biological Chemistry (1999)
Cooperation of Toll-like receptor 2 and 6 for cellular activation by soluble tuberculosis factor and Borrelia burgdorferi outer surface protein A lipoprotein: role of Toll-interacting protein and IL-1 receptor signaling molecules in Toll-like receptor 2 signaling.
Yonca Bulut;Emmanuelle Faure;Lisa Thomas;Ozlem Equils.
Journal of Immunology (2001)
Toll-like receptor-4 is required for intestinal response to epithelial injury and limiting bacterial translocation in a murine model of acute colitis
Masayuki Fukata;Kathrin S. Michelsen;Rajaraman Eri;Lisa S. Thomas.
American Journal of Physiology-gastrointestinal and Liver Physiology (2005)
Bacterial Lipopolysaccharide and IFN-γ Induce Toll-Like Receptor 2 and Toll-Like Receptor 4 Expression in Human Endothelial Cells: Role of NF-κB Activation
Emmanuelle Faure;Lisa Thomas;Helen Xu;Andrei E. Medvedev.
Journal of Immunology (2001)
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