D-Index & Metrics Best Publications
Research.com 2022 Best Female Scientist Award Badge

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Biology and Biochemistry D-index 101 Citations 37,041 286 World Ranking 657 National Ranking 423
Best female scientists D-index 106 Citations 37,611 336 World Ranking 681 National Ranking 422

Research.com Recognitions

Awards & Achievements

2022 - Research.com Best Female Scientist Award

2021 - Distinguished Fellows of the American Association of Immunologists (AAI)

2010 - Fellow of the American Association for the Advancement of Science (AAAS)

Overview

What is she best known for?

The fields of study she is best known for:

  • Gene
  • Enzyme
  • Cytokine

The scientist’s investigation covers issues in Receptor, Molecular biology, TLR4, Signal transduction and Cell biology. Her Receptor research incorporates themes from Intestinal mucosa, Gene expression, Lipopolysaccharide, Immunity and Pharmacology. Her research integrates issues of Tumor necrosis factor alpha, Cell culture, Tyrosine phosphorylation, Point mutation and Activator in her study of Molecular biology.

Her TLR4 research includes themes of Cytokine and Toll-like receptor, Lymphocyte antigen 96. Her Signal transduction research is multidisciplinary, incorporating elements of Kinase, Death domain and Myeloid Differentiation Factor 88. Her work on TIRAP as part of her general Cell biology study is frequently connected to Interferon-Stimulated Gene Factor 3, thereby bridging the divide between different branches of science.

Her most cited work include:

  • Macrophage Activation and Polarization: Nomenclature and Experimental Guidelines (3032 citations)
  • Cutting Edge: Repurification of Lipopolysaccharide Eliminates Signaling Through Both Human and Murine Toll-Like Receptor 2 (1073 citations)
  • The AIM2 inflammasome is essential for host defense against cytosolic bacteria and DNA viruses. (954 citations)

What are the main themes of her work throughout her whole career to date?

Stefanie N. Vogel mostly deals with Molecular biology, Cell biology, Immunology, Signal transduction and TLR4. Her Molecular biology research integrates issues from Gene expression, Cytokine, Lipopolysaccharide, Receptor and Macrophage. Stefanie N. Vogel has included themes like Biochemistry and Transcription factor in her Cell biology study.

As a part of the same scientific family, Stefanie N. Vogel mostly works in the field of Immunology, focusing on Virology and, on occasion, Bacterial vaccine and Immunization. Her study in Signal transduction is interdisciplinary in nature, drawing from both HEK 293 cells, Cancer research, Kinase and Myeloid Differentiation Factor 88. Her TLR4 study combines topics from a wide range of disciplines, such as Toll-like receptor, TRIF, CD14 and Transfection.

She most often published in these fields:

  • Molecular biology (33.63%)
  • Cell biology (28.57%)
  • Immunology (25.89%)

What were the highlights of her more recent work (between 2013-2021)?

  • Immunology (25.89%)
  • Cell biology (28.57%)
  • Receptor (21.13%)

In recent papers she was focusing on the following fields of study:

The scientist’s investigation covers issues in Immunology, Cell biology, Receptor, TLR4 and Innate immune system. When carried out as part of a general Immunology research project, her work on Immune system, Immunity and Cotton rat is frequently linked to work in Cytokine storm, therefore connecting diverse disciplines of study. Her Cell biology research includes elements of Interferon and Macrophage.

Her Receptor research is multidisciplinary, relying on both Molecular biology, Cancer research, Gene and Interleukin 4. Her work on TIRAP as part of general TLR4 research is often related to SNP, thus linking different fields of science. Stefanie N. Vogel does research in Signal transduction, focusing on TLR2 specifically.

Between 2013 and 2021, her most popular works were:

  • Macrophage Activation and Polarization: Nomenclature and Experimental Guidelines (3032 citations)
  • CD14 dependence of TLR4 endocytosis and TRIF signaling displays ligand specificity and is dissociable in endotoxin tolerance (89 citations)
  • TLR4 antagonist FP7 inhibits LPS-induced cytokine production and glycolytic reprogramming in dendritic cells, and protects mice from lethal influenza infection (81 citations)

In her most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Immune system

Stefanie N. Vogel spends much of her time researching Immunology, Cell biology, TLR4, Receptor and Signal transduction. In the subject of general Immunology, her work in Immunity, Acquired immune system, Inflammation and Monophosphoryl Lipid A is often linked to Cytokine storm, thereby combining diverse domains of study. The study incorporates disciplines such as TRIF and Stimulator of interferon genes in addition to Cell biology.

Her studies in TLR4 integrate themes in fields like Proinflammatory cytokine, Lung injury and Cytokine. Her Receptor study incorporates themes from Molecular biology and Gene. Her work carried out in the field of Molecular biology brings together such families of science as Tumor necrosis factor alpha, STAT2, Cancer research and Gene expression.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Macrophage Activation and Polarization: Nomenclature and Experimental Guidelines

Peter J Murray;Judith E Allen;Subhra K Biswas;Edward A Fisher.
Immunity (2014)

3001 Citations

Cutting Edge: Repurification of Lipopolysaccharide Eliminates Signaling Through Both Human and Murine Toll-Like Receptor 2

Matthew Hirschfeld;Ying Ma;John H. Weis;Stefanie N. Vogel.
Journal of Immunology (2000)

1291 Citations

The AIM2 inflammasome is essential for host defense against cytosolic bacteria and DNA viruses.

Vijay A.K. Rathinam;Zhaozhao Jiang;Stephen N. Waggoner;Shrutie Sharma.
Nature Immunology (2010)

1236 Citations

TLR4, but not TLR2, mediates IFN-beta-induced STAT1alpha/beta-dependent gene expression in macrophages.

Vladimir Toshchakov;Bryan W Jones;Pin-Yu Perera;Karen Thomas.
Nature Immunology (2002)

1129 Citations

Signaling by toll-like receptor 2 and 4 agonists results in differential gene expression in murine macrophages.

Matthew Hirschfeld;Janis J. Weis;Vladimir Toshchakov;Cindy A. Salkowski.
Infection and Immunity (2001)

855 Citations

Toll receptors, CD14, and macrophage activation and deactivation by LPS

Marina A. Dobrovolskaia;Stefanie N. Vogel.
Microbes and Infection (2002)

649 Citations

Inhibition of Lipopolysaccharide-Induced Signal Transduction in Endotoxin-Tolerized Mouse Macrophages: Dysregulation of Cytokine, Chemokine, and Toll-Like Receptor 2 and 4 Gene Expression

Andrei E. Medvedev;Karen M. Kopydlowski;Stefanie N. Vogel.
Journal of Immunology (2000)

617 Citations

Genetic and physical mapping of the Lps locus: identification of the toll-4 receptor as a candidate gene in the critical region.

Alexander Poltorak;Irina Smirnova;Xiaolong He;Mu Ya Liu.
Blood Cells Molecules and Diseases (1998)

499 Citations

CD11b/CD18 Acts in Concert with CD14 and Toll-Like Receptor (TLR) 4 to Elicit Full Lipopolysaccharide and Taxol-Inducible Gene Expression

Pin-Yu Perera;Tanya N. Mayadas;Osamu Takeuchi;Shizuo Akira.
Journal of Immunology (2001)

477 Citations

Gliadin induces an increase in intestinal permeability and zonulin release by binding to the chemokine receptor CXCR3.

Karen M. Lammers;Ruliang Lu;Julie Brownley;Bao Lu.
Gastroenterology (2008)

475 Citations

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