2022 - Research.com Best Female Scientist Award
2021 - Distinguished Fellows of the American Association of Immunologists (AAI)
2010 - Fellow of the American Association for the Advancement of Science (AAAS)
The scientist’s investigation covers issues in Receptor, Molecular biology, TLR4, Signal transduction and Cell biology. Her Receptor research incorporates themes from Intestinal mucosa, Gene expression, Lipopolysaccharide, Immunity and Pharmacology. Her research integrates issues of Tumor necrosis factor alpha, Cell culture, Tyrosine phosphorylation, Point mutation and Activator in her study of Molecular biology.
Her TLR4 research includes themes of Cytokine and Toll-like receptor, Lymphocyte antigen 96. Her Signal transduction research is multidisciplinary, incorporating elements of Kinase, Death domain and Myeloid Differentiation Factor 88. Her work on TIRAP as part of her general Cell biology study is frequently connected to Interferon-Stimulated Gene Factor 3, thereby bridging the divide between different branches of science.
Stefanie N. Vogel mostly deals with Molecular biology, Cell biology, Immunology, Signal transduction and TLR4. Her Molecular biology research integrates issues from Gene expression, Cytokine, Lipopolysaccharide, Receptor and Macrophage. Stefanie N. Vogel has included themes like Biochemistry and Transcription factor in her Cell biology study.
As a part of the same scientific family, Stefanie N. Vogel mostly works in the field of Immunology, focusing on Virology and, on occasion, Bacterial vaccine and Immunization. Her study in Signal transduction is interdisciplinary in nature, drawing from both HEK 293 cells, Cancer research, Kinase and Myeloid Differentiation Factor 88. Her TLR4 study combines topics from a wide range of disciplines, such as Toll-like receptor, TRIF, CD14 and Transfection.
The scientist’s investigation covers issues in Immunology, Cell biology, Receptor, TLR4 and Innate immune system. When carried out as part of a general Immunology research project, her work on Immune system, Immunity and Cotton rat is frequently linked to work in Cytokine storm, therefore connecting diverse disciplines of study. Her Cell biology research includes elements of Interferon and Macrophage.
Her Receptor research is multidisciplinary, relying on both Molecular biology, Cancer research, Gene and Interleukin 4. Her work on TIRAP as part of general TLR4 research is often related to SNP, thus linking different fields of science. Stefanie N. Vogel does research in Signal transduction, focusing on TLR2 specifically.
Stefanie N. Vogel spends much of her time researching Immunology, Cell biology, TLR4, Receptor and Signal transduction. In the subject of general Immunology, her work in Immunity, Acquired immune system, Inflammation and Monophosphoryl Lipid A is often linked to Cytokine storm, thereby combining diverse domains of study. The study incorporates disciplines such as TRIF and Stimulator of interferon genes in addition to Cell biology.
Her studies in TLR4 integrate themes in fields like Proinflammatory cytokine, Lung injury and Cytokine. Her Receptor study incorporates themes from Molecular biology and Gene. Her work carried out in the field of Molecular biology brings together such families of science as Tumor necrosis factor alpha, STAT2, Cancer research and Gene expression.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Macrophage Activation and Polarization: Nomenclature and Experimental Guidelines
Peter J Murray;Judith E Allen;Subhra K Biswas;Edward A Fisher.
Cutting Edge: Repurification of Lipopolysaccharide Eliminates Signaling Through Both Human and Murine Toll-Like Receptor 2
Matthew Hirschfeld;Ying Ma;John H. Weis;Stefanie N. Vogel.
Journal of Immunology (2000)
The AIM2 inflammasome is essential for host defense against cytosolic bacteria and DNA viruses.
Vijay A.K. Rathinam;Zhaozhao Jiang;Stephen N. Waggoner;Shrutie Sharma.
Nature Immunology (2010)
TLR4, but not TLR2, mediates IFN-beta-induced STAT1alpha/beta-dependent gene expression in macrophages.
Vladimir Toshchakov;Bryan W Jones;Pin-Yu Perera;Karen Thomas.
Nature Immunology (2002)
Signaling by toll-like receptor 2 and 4 agonists results in differential gene expression in murine macrophages.
Matthew Hirschfeld;Janis J. Weis;Vladimir Toshchakov;Cindy A. Salkowski.
Infection and Immunity (2001)
Toll receptors, CD14, and macrophage activation and deactivation by LPS
Marina A. Dobrovolskaia;Stefanie N. Vogel.
Microbes and Infection (2002)
Inhibition of Lipopolysaccharide-Induced Signal Transduction in Endotoxin-Tolerized Mouse Macrophages: Dysregulation of Cytokine, Chemokine, and Toll-Like Receptor 2 and 4 Gene Expression
Andrei E. Medvedev;Karen M. Kopydlowski;Stefanie N. Vogel.
Journal of Immunology (2000)
Genetic and physical mapping of the Lps locus: identification of the toll-4 receptor as a candidate gene in the critical region.
Alexander Poltorak;Irina Smirnova;Xiaolong He;Mu Ya Liu.
Blood Cells Molecules and Diseases (1998)
CD11b/CD18 Acts in Concert with CD14 and Toll-Like Receptor (TLR) 4 to Elicit Full Lipopolysaccharide and Taxol-Inducible Gene Expression
Pin-Yu Perera;Tanya N. Mayadas;Osamu Takeuchi;Shizuo Akira.
Journal of Immunology (2001)
Gliadin induces an increase in intestinal permeability and zonulin release by binding to the chemokine receptor CXCR3.
Karen M. Lammers;Ruliang Lu;Julie Brownley;Bao Lu.
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