2009 - Member of Academia Europaea
2003 - Royal Netherlands Academy of Arts and Sciences
Immunology, Proinflammatory cytokine, Innate immune system, Immune system and Cytokine are his primary areas of study. In Immunology, Jos W. M. van der Meer works on issues like Candida albicans, which are connected to Toll-like receptor. His Proinflammatory cytokine research is multidisciplinary, incorporating elements of Interleukin 18, Interferon gamma, Microbiology and Signal transduction, Cell biology.
His Innate immune system study combines topics from a wide range of disciplines, such as Acquired immune system, Epigenetics and Immunity. As a part of the same scientific family, Jos W. M. van der Meer mostly works in the field of Immune system, focusing on Monocyte and, on occasion, Reprogramming. His research investigates the connection with Cytokine and areas like Receptor antagonist which intersect with concerns in Interleukin-21 receptor and Molecular biology.
Jos W. M. van der Meer spends much of his time researching Immunology, Cytokine, Internal medicine, Microbiology and Innate immune system. His study in Immune system, Proinflammatory cytokine, Inflammation, Tumor necrosis factor alpha and Immunity is carried out as part of his Immunology studies. Jos W. M. van der Meer usually deals with Cytokine and limits it to topics linked to Receptor and Cell biology.
His study looks at the relationship between Internal medicine and fields such as Placebo, as well as how they intersect with chemical problems. Candida albicans is the focus of his Microbiology research. His study brings together the fields of Acquired immune system and Innate immune system.
Jos W. M. van der Meer focuses on Immunology, Internal medicine, Chronic fatigue syndrome, Cytokine and Immune system. His work is connected to Inflammation, Innate immune system, Immunity, Q fever and Proinflammatory cytokine, as a part of Immunology. His study explores the link between Innate immune system and topics such as Acquired immune system that cross with problems in Neuroscience.
His biological study spans a wide range of topics, including Gastroenterology, Placebo and Endocrinology. His Cytokine research integrates issues from Tumor necrosis factor alpha, Pathogenesis, Periodic fever syndrome, Peripheral blood mononuclear cell and TLR4. His T cell and Pattern recognition receptor study, which is part of a larger body of work in Immune system, is frequently linked to Functional genomics, bridging the gap between disciplines.
His primary areas of investigation include Immunology, Immune system, Inflammation, Immunity and Innate immune system. His Immunology research is multidisciplinary, relying on both Receptor and Cell biology. The various areas that he examines in his Immune system study include Missense mutation, Medical history, Interferon and Pathogenesis.
His research integrates issues of Bioinformatics, Bradykinin, Icatibant, Angioedema and Pulmonary edema in his study of Inflammation. His studies in Immunity integrate themes in fields like Myeloid and Vaccination. His study in Innate immune system is interdisciplinary in nature, drawing from both Acquired immune system, Genome-wide association study and Epigenetics.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Discovery, research, and development of new antibiotics: the WHO priority list of antibiotic-resistant bacteria and tuberculosis
Evelina Tacconelli;Elena Carrara;Alessia Savoldi;Stephan Harbarth.
Lancet Infectious Diseases (2017)
Treating inflammation by blocking interleukin-1 in a broad spectrum of diseases
Charles A. Dinarello;Anna Simon;Jos W. M. van der Meer.
Nature Reviews Drug Discovery (2012)
Chronic fatigue syndrome
Judith B Prins;Jos W M van der Meer;Gijs Bleijenberg.
The Lancet (2006)
Dimensional assessment of chronic fatigue syndrome
Jan H.M.M. Vercoulen;Caroline M.A. Swanink;Jan F.M. Fennis;Joep M.D. Galama.
Journal of Psychosomatic Research (1994)
mTOR- and HIF-1α–mediated aerobic glycolysis as metabolic basis for trained immunity
Shih-Chin Cheng;Jessica Quintin;Robert A. Cramer;Kelly M. Shepardson.
Bacille Calmette-Guérin induces NOD2-dependent nonspecific protection from reinfection via epigenetic reprogramming of monocytes
Johanneke Kleinnijenhuis;Jessica Quintin;Frank Preijers;Leo A. B. Joosten.
Proceedings of the National Academy of Sciences of the United States of America (2012)
Epigenetic programming of monocyte-to-macrophage differentiation and trained innate immunity
Sadia Saeed;Jessica Quintin;Hindrik H. D. Kerstens;Nagesha A. Rao.
Innate Immunity to Mycobacterium tuberculosis
Reinout van Crevel;Tom H M Ottenhoff;Jos W M van der Meer.
Clinical Microbiology Reviews (2002)
Trained Immunity: A Memory for Innate Host Defense
Mihai G. Netea;Jessica Quintin;Jos W.M. van der Meer.
Cell Host & Microbe (2011)
Immune sensing of Candida albicans requires cooperative recognition of mannans and glucans by lectin and Toll-like receptors
Mihai G. Netea;Neil A.R. Gow;Carol A. Munro;Steven Bates.
Journal of Clinical Investigation (2006)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: