What is she best known for?
The fields of study she is best known for:
- Immune system
- Gene
- Enzyme
Her main research concerns Immunology, Microbiology, Immunity, Immune system and Candida albicans.
Her work in Inflammation, Aspergillosis, Cytokine, Cellular immunity and Interleukin 10 is related to Immunology.
Her Microbiology research integrates issues from Dendritic cell, Priming, Antigen and Adoptive cell transfer.
Her Immunity research incorporates elements of Acquired immune system, Attenuated vaccine, IL-2 receptor and Effector.
Luigina Romani works mostly in the field of Immune system, limiting it down to concerns involving Interleukin 12 and, occasionally, Disease and Immunopathology.
Her studies in Candida albicans integrate themes in fields like Cell, In vitro, T helper cell and Interleukin 4.
Her most cited work include:
- Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition) (4475 citations)
- Immunity to fungal infections (1404 citations)
- Tryptophan Catabolites from Microbiota Engage Aryl Hydrocarbon Receptor and Balance Mucosal Reactivity via Interleukin-22 (1023 citations)
What are the main themes of her work throughout her whole career to date?
Luigina Romani mostly deals with Immunology, Immune system, Microbiology, Immunity and Candida albicans.
Inflammation, Aspergillosis, Cytokine, Antigen and Innate immune system are subfields of Immunology in which her conducts study.
Her research on Immune system frequently connects to adjacent areas such as Effector.
Her Microbiology research incorporates themes from Host, Interleukin 10 and Virulence.
Her Immunity research includes themes of Pathogen, Transplantation and Indoleamine 2,3-dioxygenase.
She has researched Candida albicans in several fields, including Interleukin, Interferon gamma and Interleukin 4.
She most often published in these fields:
- Immunology (61.12%)
- Immune system (33.26%)
- Microbiology (29.27%)
What were the highlights of her more recent work (between 2017-2021)?
- Immune system (33.26%)
- Immunology (61.12%)
- Inflammation (14.99%)
In recent papers she was focusing on the following fields of study:
Her primary scientific interests are in Immune system, Immunology, Inflammation, Cell biology and Cystic fibrosis.
Her work deals with themes such as Microbiome, Pathogen, Microbiology, Host and Aspergillosis, which intersect with Immune system.
The concepts of her Immunology study are interwoven with issues in Kynurenine pathway and Candida albicans.
As part of the same scientific family, she usually focuses on Candida albicans, concentrating on Cytokine and intersecting with Epitope and Human gastrointestinal tract.
Her study in Inflammation is interdisciplinary in nature, drawing from both Cancer research, Pathogenesis, Virology, Off-label use and Commensalism.
Her work carried out in the field of Cell biology brings together such families of science as Autophagy, Inflammasome, Aryl hydrocarbon receptor, Transcription factor and Innate immune system.
Between 2017 and 2021, her most popular works were:
- Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition) (202 citations)
- Deficiency of immunoregulatory indoleamine 2,3-dioxygenase 1in juvenile diabetes (45 citations)
- The contribution of mast cells to bacterial and fungal infection immunity (44 citations)
In her most recent research, the most cited papers focused on:
- Immune system
- Gene
- Enzyme
Luigina Romani mainly focuses on Immune system, Immunology, Cell biology, Inflammation and Microbiology.
Her Immune system research includes themes of Mast cell, BECN1, Aryl hydrocarbon receptor and Intestinal mucosa.
The Immunology study combines topics in areas such as Peripheral blood mononuclear cell and Candida albicans.
Her studies deal with areas such as Inflammasome, Innate immune system, Cystic fibrosis and Cysteamine as well as Cell biology.
Her Inflammation research incorporates themes from Off-label use, Cachexia, MEDLINE and Virology.
Her Microbiology study integrates concerns from other disciplines, such as Host, Severe acute respiratory syndrome coronavirus 2, Coronavirus disease 2019 and Nose.
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