His primary areas of study are Immunology, Cell biology, Immunity, Candida albicans and Indoleamine 2,3-dioxygenase. Paolo Puccetti combines subjects such as In vitro and In vivo with his study of Immunology. His biological study spans a wide range of topics, including Antigen-presenting cell, Dendritic cell, CD8, Immune tolerance and Antigen presentation.
His research investigates the connection with Antigen-presenting cell and areas like Natural killer T cell which intersect with concerns in Cancer research. His Candida albicans study combines topics in areas such as Interleukin 22 and Macrophage. The various areas that Paolo Puccetti examines in his Indoleamine 2,3-dioxygenase study include Phenotype, T cell and Antigen.
His primary scientific interests are in Immunology, Immune system, Cell biology, Microbiology and Candida albicans. His Immunology research integrates issues from In vitro and In vivo. His work in Immune system addresses issues such as Cancer research, which are connected to fields such as Immunogenicity.
He usually deals with Cell biology and limits it to topics linked to Indoleamine 2,3-dioxygenase and Aryl hydrocarbon receptor. His studies deal with areas such as Macrophage and Virulence as well as Microbiology. His study looks at the relationship between Immunity and fields such as Inflammation, as well as how they intersect with chemical problems.
Paolo Puccetti mainly focuses on Cell biology, Immunology, Immune system, Aryl hydrocarbon receptor and Indoleamine 2,3-dioxygenase. His Cell biology study incorporates themes from Regulator, Phenotype and ARG1. As part of his studies on Immunology, he often connects relevant areas like Signal transduction.
The study incorporates disciplines such as Antigen and Function in addition to Immune system. His Aryl hydrocarbon receptor research integrates issues from Catabolism, Receptor and Metabolic pathway. His research in Indoleamine 2,3-dioxygenase intersects with topics in Kynurenine pathway, Kynurenine, Immune tolerance, Immunity and Proinflammatory cytokine.
His primary areas of study are Immunology, Indoleamine 2,3-dioxygenase, Immune system, Biochemistry and Immune tolerance. His biological study spans a wide range of topics, including NFKB1 and NFAT. His Indoleamine 2,3-dioxygenase research incorporates themes from Tumor microenvironment, Signal transduction, Immunity and Autoimmunity.
His research investigates the connection between Signal transduction and topics such as Nod that intersect with problems in Cell biology. His work deals with themes such as Microbiology and Candida albicans, which intersect with Immunity. His Immune system research includes themes of Cancer research, Antibiotics and Kynurenine.
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Modulation of tryptophan catabolism by regulatory T cells.
Francesca Fallarino;Ursula Grohmann;Kwang Woo Hwang;Ciriana Orabona.
Nature Immunology (2003)
CTLA-4-Ig regulates tryptophan catabolism in vivo.
Ursula Grohmann;Ciriana Orabona;Francesca Fallarino;Carmine Vacca.
Nature Immunology (2002)
Tryptophan Catabolites from Microbiota Engage Aryl Hydrocarbon Receptor and Balance Mucosal Reactivity via Interleukin-22
Teresa Zelante;Rossana G. Iannitti;Cristina Cunha;Antonella De Luca.
Immunity (2013)
The Combined Effects of Tryptophan Starvation and Tryptophan Catabolites Down-Regulate T Cell Receptor ζ-Chain and Induce a Regulatory Phenotype in Naive T Cells
Francesca Fallarino;Ursula Grohmann;Sylvaine You;Barbara C. McGrath.
Journal of Immunology (2006)
T cell apoptosis by tryptophan catabolism.
Francesca Fallarino;Ursula Grohmann;Carmine Vacca;Roberta Bianchi.
Cell Death & Differentiation (2002)
Tolerance, DCs and tryptophan: much ado about IDO
Ursula Grohmann;Francesca Fallarino;Paolo Puccetti.
Trends in Immunology (2003)
Natural Killer Cells: Characteristics and Regulation of Activity
Ronald B. Herberman;Ronald B. Herberman;Ronald B. Herberman;Julie Y. Djeu;Julie Y. Djeu;Julie Y. Djeu;H. David Kay;H. David Kay;H. David Kay;John R. Ortaldo;John R. Ortaldo;John R. Ortaldo.
Immunological Reviews (1979)
IL-23 and the Th17 pathway promote inflammation and impair antifungal immune resistance.
Teresa Zelante;Antonella De Luca;Pierluigi Bonifazi;Claudia Montagnoli.
European Journal of Immunology (2007)
Defective tryptophan catabolism underlies inflammation in mouse chronic granulomatous disease
Luigina Romani;Francesca Fallarino;Antonella De Luca;Claudia Montagnoli.
Nature (2008)
Indoleamine 2,3-dioxygenase is a signaling protein in long-term tolerance by dendritic cells
Maria T Pallotta;Ciriana Orabona;Claudia Volpi;Carmine Vacca.
Nature Immunology (2011)
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