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Biology and Biochemistry

D-Index
66
Citations
21708
World Ranking
8489
National Ranking
3818

Overview

Matthew J. Fenton is affiliated with the National Institute of Allergy and Infectious Diseases in the United States. Their research primarily spans the field of medicine with a focus on surgery, biomedical engineering, cardiology and cardiovascular medicine, transplantation, and oncology.

The scientist has contributed extensively to topics including transplantation methods and outcomes, mechanical circulatory support devices, renal transplantation outcomes and treatments, cardiac structural anomalies and repair, cardiomyopathy and myosin studies, cardiovascular function and risk factors, and polyomavirus and related diseases.

Frequent publication venues for Fenton include:

  • The Journal of Heart and Lung Transplantation
  • Pediatric Transplantation
  • Perfusion
  • CHEST Journal
  • Open Heart

Some of the recent papers authored or co-authored by Matthew J. Fenton are:

  • ISHLT consensus statement on donor organ acceptability and management in pediatric heart transplantation, 2020, The Journal of Heart and Lung Transplantation
  • Cardiorespiratory Progression Over 5 Years and Role of Corticosteroids in Duchenne Muscular Dystrophy, 2020, CHEST Journal
  • Pediatric heart transplantation following donation after circulatory death, distant procurement, and ex-situ perfusion, 2022, The Journal of Heart and Lung Transplantation
  • Surveillance for cardiac allograft vasculopathy: Practice variations among 50 pediatric heart transplant centers, 2020, The Journal of Heart and Lung Transplantation
  • Cardiac care of children with dystrophinopathy and females carrying DMD-gene variations, 2022, Open Heart

Frequent co-authors collaborating with Matthew J. Fenton include:

  • Michael Burch
  • Richard Issitt
  • Jennifer Conway
  • Richard Crook
  • Craig Laurence

Best Publications

  • Reversing established sepsis with antagonists of endogenous high-mobility group box 1

    Huan Yang;Mahendar Ochani;Jianhua Li;Xiaoling Qiang

  • TLR4, but not TLR2, mediates IFN-beta-induced STAT1alpha/beta-dependent gene expression in macrophages.

    Vladimir Toshchakov;Bryan W Jones;Pin-Yu Perera;Karen Thomas

  • Human Toll-Like Receptors Mediate Cellular Activation by Mycobacterium tuberculosis

    Terry K. Means;Shuyan Wang;Egil Lien;Atsutoshi Yoshimura

  • HMGB1 signals through toll-like receptor (TLR) 4 and TLR2.

    Man Yu;Haichao Wang;Aihao Ding;Douglas T Golenbock

  • Toll-like receptor 4 imparts ligand-specific recognition of bacterial lipopolysaccharide

    Egil Lien;Terry K. Means;Holger Heine;Atsutoshi Yoshimura

  • Hyaluronan Fragments Act as an Endogenous Danger Signal by Engaging TLR2

    Kara A. Scheibner;Michael A. Lutz;Sada Boodoo;Matthew J. Fenton

  • Infection by Mycobacterium tuberculosis promotes human alveolar macrophage apoptosis.

    J Keane;M K Balcewicz-Sablinska;H G Remold;G L Chupp

  • LPS-binding proteins and receptors

    Matthew J. Fenton;Douglas T. Golenbock

  • ATLAS b-jet identification performance and efficiency measurement with tt¯ events in pp collisions at √s = 13 TeV

    Georges Aad;Alexander Kupco;Samuel Webb;Timo Dreyer

  • The CD14 Ligands Lipoarabinomannan and Lipopolysaccharide Differ in Their Requirement for Toll-Like Receptors

    Terry K. Means;Egil Lien;Atsutoshi Yoshimura;Shuyan Wang

  • Different Toll-like receptor agonists induce distinct macrophage responses

    Bryan W. Jones;Terry K. Means;Kurt A. Heldwein;Marc A. Keen

  • TLR4/MyD88/PI3K interactions regulate TLR4 signaling

    Michelle H. W. Laird;Sang Hoon Rhee;Darren J. Perkins;Andrei E. Medvedev

  • Immunopathology of tuberculosis: roles of macrophages and monocytes

    M J Fenton;M W Vermeulen

  • Aspergillus fumigatus Evades Immune Recognition during Germination through Loss of Toll-Like Receptor-4-Mediated Signal Transduction

    Mihai G. Netea;Adilia Warris;Jos W. M. Van der Meer;Matthew J. Fenton

  • The biology of Toll-like receptors

    Terry K. Means;Douglas T. Golenbock;Matthew J. Fenton

  • Differential Effects of a Toll-Like Receptor Antagonist on Mycobacterium tuberculosis-Induced Macrophage Responses

    Terry K. Means;Bryan W. Jones;Andra B. Schromm;Beth A. Shurtleff

  • A Lipopolysaccharide-Specific Enhancer Complex Involving Ets, Elk-1, Sp1, and CREB Binding Protein and p300 Is Recruited to the Tumor Necrosis Factor Alpha Promoter In Vivo

    Eunice Y. Tsai;James V. Falvo;Alla V. Tsytsykova;Amy K. Barczak

  • TLRs: differential adapter utilization by toll-like receptors mediates TLR-specific patterns of gene expression.

    Stefanie N. Vogel;Katherine A. Fitzgerald;Matthew J. Fenton

  • TLR2 and TLR4 serve distinct roles in the host immune response against Mycobacterium bovis BCG

    Kurt A. Heldwein;Michael D. Liang;Tonje K. Andresen;Karen E. Thomas

  • Induction of gamma interferon production in human alveolar macrophages by Mycobacterium tuberculosis.

    Matthew J. Fenton;Mary W. Vermeulen;Sue Kim;Marie Burdick

Frequent Co-Authors

Douglas T. Golenbock
Douglas T. Golenbock University of Massachusetts Chan Medical School
Stefanie N. Vogel
Stefanie N. Vogel University of Maryland, Baltimore
Egil Lien
Egil Lien University of Massachusetts Chan Medical School
Salomon Amar
Salomon Amar New York Medical College
Kevin J. Tracey
Kevin J. Tracey Feinstein Institute for Medical Research
Raymond P. Donnelly
Raymond P. Donnelly US Food and Drug Administration
Hardy Kornfeld
Hardy Kornfeld University of Massachusetts Chan Medical School
Thomas A. Hamilton
Thomas A. Hamilton Cleveland Clinic
John T. Belisle
John T. Belisle Colorado State University
Haichao Wang
Haichao Wang Feinstein Institute for Medical Research

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