His primary areas of investigation include HMGB1, Tumor necrosis factor alpha, Immunology, Cell biology and Inflammation. His HMGB1 research incorporates elements of Sepsis, Intensive care, Signal transduction, Systemic inflammation and p38 mitogen-activated protein kinases. His research in Tumor necrosis factor alpha intersects with topics in Macrophage migration inhibitory factor, Cytokine, Lipopolysaccharide, Receptor and Proinflammatory cytokine.
His Cytokine research integrates issues from Monocyte and TLR4. His Immunology research is multidisciplinary, incorporating elements of Mediator and Pharmacology. His Cell biology study integrates concerns from other disciplines, such as High-mobility group, Inflammasome, Biochemistry and Anaerobic glycolysis.
Haichao Wang mainly focuses on HMGB1, Immunology, Sepsis, Proinflammatory cytokine and Tumor necrosis factor alpha. His HMGB1 research incorporates themes from Extracellular, Mediator, Cell biology, Molecular biology and Systemic inflammation. His study brings together the fields of Lung injury and Immunology.
The study incorporates disciplines such as Intensive care, Antagonist and Pharmacology in addition to Sepsis. His Proinflammatory cytokine study frequently draws connections to other fields, such as Monocyte. His studies in Tumor necrosis factor alpha integrate themes in fields like Lipopolysaccharide and Macrophage.
Haichao Wang spends much of his time researching Sepsis, HMGB1, Cell biology, Immunology and Antibody. The various areas that Haichao Wang examines in his Sepsis study include Pyroptosis, Immunity, Innate immune system, Macrophage and Pharmacology. His Pharmacology study incorporates themes from Tumor necrosis factor alpha, Cancer, Receptor and In vivo.
His HMGB1 study improves the overall literature in Inflammation. His biological study spans a wide range of topics, including Inflammasome and Necroptosis. His Antibody study combines topics in areas such as Secretion and Cancer research.
Haichao Wang mostly deals with Cell biology, HMGB1, Sepsis, Inflammation and Inflammasome. His Cell biology research is multidisciplinary, relying on both Pyroptosis and Proinflammatory cytokine. The concepts of his Proinflammatory cytokine study are interwoven with issues in Serum amyloid A, Mediator and In vivo.
His HMGB1 research includes themes of Cancer research, Extracellular, Hippo signaling pathway, Carcinogenesis and Internalization. His Sepsis study is concerned with Immunology in general. The concepts of his Inflammation study are interwoven with issues in Toxicity, Reactive oxygen species, Mitochondrion and Mitochondrial DNA.
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Guidelines for the use and interpretation of assays for monitoring autophagy
Daniel J. Klionsky;Fabio C. Abdalla;Hagai Abeliovich;Robert T. Abraham.
Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)
Daniel J. Klionsky;Kotb Abdelmohsen;Akihisa Abe;Joynal Abedin.
Vagus nerve stimulation attenuates the systemic inflammatory response to endotoxin
Lyudmila V. Borovikova;Svetlana Ivanova;Minghuang Zhang;Huan Yang.
HMG-1 as a Late Mediator of Endotoxin Lethality in Mice
Haichao Wang;Ona Bloom;Minghuang Zhang;Jaideep M. Vishnubhakat.
Nicotinic acetylcholine receptor alpha7 subunit is an essential regulator of inflammation.
Hong Wang;Man Yu;Mahendar Ochani;Carol Ann Amella.
High mobility group 1 protein (HMG-1) stimulates proinflammatory cytokine synthesis in human monocytes.
Ulf Andersson;Haichao Wang;Karin Palmblad;Ann-Charlotte Aveberger.
Journal of Experimental Medicine (2000)
Reversing established sepsis with antagonists of endogenous high-mobility group box 1
Huan Yang;Mahendar Ochani;Jianhua Li;Xiaoling Qiang.
Proceedings of the National Academy of Sciences of the United States of America (2004)
Cholinergic agonists inhibit HMGB1 release and improve survival in experimental sepsis.
Hong Wang;Hong Liao;Mahendar Ochani;Marilou Justiniani.
Nature Medicine (2004)
HMG-1 as a mediator of acute lung inflammation.
Edward Abraham;John Arcaroli;Aaron Carmody;Haichao Wang.
Journal of Immunology (2000)
Methylation of histone H4 at arginine 3 facilitating transcriptional activation by nuclear hormone receptor
Hengbin Wang;Zhi-Qing Huang;Li Xia;Qin Feng.
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