What is he best known for?
The fields of study he is best known for:
- Gene
- Cytokine
- Immune system
His primary areas of study are Molecular biology, Signal transduction, Chemokine, Immunology and Gene expression.
His Molecular biology study combines topics from a wide range of disciplines, such as Tumor necrosis factor alpha, NFKB1, Interferon gamma, Regulation of gene expression and Transcription.
His Signal transduction research is multidisciplinary, relying on both I-Kappa-B Kinase, Cancer research and Neutrophilia.
His Chemokine research is multidisciplinary, incorporating elements of Messenger RNA, In situ hybridization and Cell biology.
His work on Immune system and Cytokine as part of general Immunology study is frequently linked to Encephalomyelitis, bridging the gap between disciplines.
His Gene expression study incorporates themes from Complementary DNA, Open reading frame, Nucleic acid sequence and Homology.
His most cited work include:
- TLR4, but not TLR2, mediates IFN-beta-induced STAT1alpha/beta-dependent gene expression in macrophages. (716 citations)
- Astrocyte expression of mRNA encoding cytokines IP-10 and JE/MCP-1 in experimental autoimmune encephalomyelitis. (455 citations)
- The adaptor Act1 is required for interleukin 17–dependent signaling associated with autoimmune and inflammatory disease (422 citations)
What are the main themes of his work throughout his whole career to date?
His primary areas of investigation include Molecular biology, Gene expression, Cell biology, Chemokine and Messenger RNA.
The Molecular biology study combines topics in areas such as Lipopolysaccharide and Transcription, Gene, MRNA stabilization.
His research on Gene expression also deals with topics like
- Regulation of gene expression which is related to area like NFKB1,
- Tumor necrosis factor alpha which intersects with area such as Interferon gamma,
- STAT6 that intertwine with fields like Intracellular.
His Cell biology research focuses on subjects like Chemokine receptor, which are linked to CXCL10.
His work investigates the relationship between Chemokine and topics such as Cell type that intersect with problems in In situ hybridization.
His studies deal with areas such as Proinflammatory cytokine and CXCL1 as well as Messenger RNA.
He most often published in these fields:
- Molecular biology (50.00%)
- Gene expression (33.02%)
- Cell biology (30.19%)
What were the highlights of his more recent work (between 2016-2020)?
- Cell biology (30.19%)
- Cancer research (11.32%)
- Inflammation (14.15%)
In recent papers he was focusing on the following fields of study:
Thomas A. Hamilton focuses on Cell biology, Cancer research, Inflammation, Chemokine and Pathology.
His Cell biology study focuses on Signal transducing adaptor protein in particular.
His work carried out in the field of Signal transducing adaptor protein brings together such families of science as Messenger RNA, Untranslated region and Stem cell.
His work deals with themes such as CXCL1, Macrophage and Immunotherapy, which intersect with Cancer research.
His studies in Inflammation integrate themes in fields like Amyotrophic lateral sclerosis and Neuron.
His Chemokine research includes elements of Proinflammatory cytokine, Translation, P-bodies and MRNA stabilization.
Between 2016 and 2020, his most popular works were:
- Myeloid-Derived Suppressor Cell Subset Accumulation in Renal Cell Carcinoma Parenchyma Is Associated with Intratumoral Expression of IL1β, IL8, CXCL5, and Mip-1α. (70 citations)
- IL-17R-EGFR axis links wound healing to tumorigenesis in Lrig1 + stem cells (36 citations)
- IL-17-receptor-associated adaptor Act1 directly stabilizes mRNAs to mediate IL-17 inflammatory signaling. (32 citations)
In his most recent research, the most cited papers focused on:
- Gene
- Cytokine
- Immune system
His primary areas of investigation include Cell biology, Carcinogenesis, Population, Wound healing and Signal transducing adaptor protein.
The various areas that Thomas A. Hamilton examines in his Cell biology study include P-bodies, Messenger RNA, Untranslated region, MRNA stabilization and Proinflammatory cytokine.
His Proinflammatory cytokine study often links to related topics such as Chemokine.
His work on CXC chemokine receptors as part of general Chemokine research is frequently linked to Myeloid-derived Suppressor Cell, bridging the gap between disciplines.
The concepts of his Carcinogenesis study are interwoven with issues in Progenitor cell, Cancer research, Colitis and Epidermis.
His work in Progenitor cell addresses issues such as Pathology, which are connected to fields such as CXCL5.
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