Robert L. Fairchild spends much of his time researching Immunology, Chemokine, T cell, Transplantation and Immune system. Immunology is closely attributed to Cytotoxic T cell in his study. His Chemokine study incorporates themes from Antiserum and Chemotaxis.
He has researched T cell in several fields, including Priming and Cellular infiltration. The concepts of his Transplantation study are interwoven with issues in Kidney, Cytokine, Macrophage inflammatory protein and Pathology. In his study, Sensitization, Allergic contact dermatitis, Effector and CD28 is inextricably linked to Hapten, which falls within the broad field of Immune system.
His primary areas of study are Immunology, T cell, Cytotoxic T cell, Transplantation and CD8. Immunology is a component of his Chemokine, Immune system, Priming, IL-2 receptor and Major histocompatibility complex studies. His work deals with themes such as Cancer research, Dendritic cell, CD40 and Sensitization, which intersect with T cell.
His studies deal with areas such as Molecular biology and Cell biology as well as Cytotoxic T cell. The Transplantation study combines topics in areas such as Inflammation, Reperfusion injury and Pathology. His CD8 research includes elements of Adoptive cell transfer, Hapten, Interferon gamma and Antigen presentation.
Robert L. Fairchild mainly investigates Immunology, T cell, Transplantation, Inflammation and Cytotoxic T cell. His biological study spans a wide range of topics, including Kidney and Kidney transplantation. Robert L. Fairchild combines subjects such as Interleukin, Cancer research, Bone marrow and Immunosuppression with his study of T cell.
The study incorporates disciplines such as Endogeny, Cytokine, Immunity and Effector in addition to Transplantation. His work carried out in the field of Inflammation brings together such families of science as Adoptive cell transfer, MHC class I, Antigen and Cross reactive antibodies. His Cytotoxic T cell research is multidisciplinary, incorporating elements of Cardiac allograft, Mannan-binding lectin, Blockade, CD8 and Sensitization.
His scientific interests lie mostly in Immunology, T cell, Cytotoxic T cell, Transplantation and Kidney. As part of the same scientific family, Robert L. Fairchild usually focuses on Immunology, concentrating on Kidney transplantation and intersecting with Sensitization. His work in T cell addresses subjects such as Cancer research, which are connected to disciplines such as IL-2 receptor, Intestinal mucosa, Chemokine and Peripheral blood mononuclear cell.
In the field of Cytotoxic T cell, his study on Costimulatory blockade overlaps with subjects such as Complement. His Transplantation research incorporates elements of Immunosuppression, Human leukocyte antigen, Histocompatibility and Pharmacology. His Kidney study incorporates themes from Downregulation and upregulation, Receptor, Erythropoietin receptor, Transplant rejection and Antibody.
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Interferon action and apoptosis are defective in mice devoid of 2′,5′‐oligoadenylate‐dependent RNase L
Aimin Zhou;Aimin Zhou;Jayashree Paranjape;Thomas L. Brown;Huiqin Nie.
The EMBO Journal (1997)
T cell populations primed by hapten sensitization in contact sensitivity are distinguished by polarized patterns of cytokine production: interferon gamma-producing (Tc1) effector CD8+ T cells and interleukin (Il) 4/Il-10-producing (Th2) negative regulatory CD4+ T cells.
Hui Xu;Nancy A. DiIulio;Robert Fairchild.
Journal of Experimental Medicine (1996)
The Toll–Interleukin-1 Receptor Member SIGIRR Regulates Colonic Epithelial Homeostasis, Inflammation, and Tumorigenesis
Hui Xiao;Muhammet Fatih Gulen;Muhammet Fatih Gulen;Jinzhong Qin;Jinzhong Qin;Jianhong Yao.
Neutralization of Groα and macrophage inflammatory protein-2 attenuates renal ischemia/reperfusion injury
Masayoshi Miura;Xi Fu;Qi-Wei Zhang;Daniel G. Remick.
American Journal of Pathology (2001)
Urinary-Cell mRNA Profile and Acute Cellular Rejection in Kidney Allografts
Manikkam Suthanthiran;Joseph E. Schwartz;Ruchuang Ding;Michael Abecassis.
The New England Journal of Medicine (2013)
TLR engagement prevents transplantation tolerance
L. Chen;T. Wang;P. Zhou;L. Ma.
American Journal of Transplantation (2006)
Monokine Induced by IFN-γ Is a Dominant Factor Directing T Cells into Murine Cardiac Allografts During Acute Rejection
Masayoshi Miura;Ken Morita;Hirohito Kobayashi;Thomas A. Hamilton.
Journal of Immunology (2001)
Chemokines: directing leukocyte infiltration into allografts.
Tarek El-Sawy;Nader M Fahmy;Robert L Fairchild.
Current Opinion in Immunology (2002)
Early Chemokine Cascades in Murine Cardiac Grafts Regulate T Cell Recruitment and Progression of Acute Allograft Rejection
Ken Morita;Masayoshi Miura;David R. Paolone;Tara M. Engeman.
Journal of Immunology (2001)
T Cell Infiltration into Class II MHC-Disparate Allografts and Acute Rejection Is Dependent on the IFN-γ-Induced Chemokine Mig
Shoji Koga;Michael B. Auerbach;Tara M. Engeman;Andrew C. Novick.
Journal of Immunology (1999)
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