1963 - Fellow of the American Association for the Advancement of Science (AAAS)
The scientist’s investigation covers issues in Immunology, Transplantation, Pathology, Complement system and Internal medicine. William M. Baldwin has included themes like Peripheral blood mononuclear cell, Flow cytometry and Von Willebrand factor in his Transplantation study. The Pathology study combines topics in areas such as Platelet, Complication, Organ transplantation and Andrology.
His Internal medicine research is multidisciplinary, incorporating perspectives in Titer, Endocrinology and Complement factor I. The various areas that William M. Baldwin examines in his Immune system study include Cytotoxic T cell and Cytokine. His biological study spans a wide range of topics, including Grading, Chronic allograft nephropathy, Anatomical pathology and Kidney disease.
His main research concerns Immunology, Transplantation, Pathology, Antibody and Immune system. His Immunology research includes elements of Cytotoxic T cell and Kidney. His Transplantation research incorporates themes from Flow cytometry, Immunosuppression and Complement membrane attack complex.
As part of his studies on Pathology, William M. Baldwin frequently links adjacent subjects like Spleen. He interconnects Macrophage and Major histocompatibility complex in the investigation of issues within Antibody. His Immune system study integrates concerns from other disciplines, such as Platelet, Internal medicine, Cytokine and Endocrinology.
William M. Baldwin spends much of his time researching Immunology, Inflammation, Cytotoxic T cell, Antibody and Transplantation. In his study, Antibody titer and Bone marrow is inextricably linked to Heart transplantation, which falls within the broad field of Immunology. His studies in Inflammation integrate themes in fields like Complement system, Endocrinology and Macrophage.
His Cytotoxic T cell research incorporates elements of T cell, Endogeny, CD8 and Effector. As a member of one scientific family, William M. Baldwin mostly works in the field of Antibody, focusing on Tissue Graft and, on occasion, CCL2, Interferon gamma and Flow cytometry. His research in Transplantation focuses on subjects like Pathology, which are connected to Kidney transplantation.
His primary scientific interests are in Immunology, Transplantation, Inflammation, Cytotoxic T cell and Pathology. Immunology is closely attributed to Cancer research in his study. His work in Cancer research covers topics such as T cell which are related to areas like Antigen.
His Transplantation research is multidisciplinary, relying on both Perfusion and Sensitization. His studies deal with areas such as Alternative complement pathway, Endocrinology and Bioinformatics as well as Inflammation. His research investigates the connection with Pathology and areas like Kidney transplantation which intersect with concerns in CD20, Monoclonal antibody and Platelet.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Banff 07 Classification of Renal Allograft Pathology: Updates and Future Directions
K. Solez;R. B. Colvin;L. C. Racusen;M. Haas.
American Journal of Transplantation (2008)
Banff '09 meeting report: Antibody mediated graft deterioration and implementation of Banff working groups
B. Sis;M. Mengel;M. Haas;R. B. Colvin.
American Journal of Transplantation (2010)
Identification of Hepatocarcinoma-Intestine-Pancreas/Pancreatitis-associated Protein I as a Biomarker for Pancreatic Ductal Adenocarcinoma by Protein Biochip Technology
Christophe Rosty;Laurence Christa;Scott Kuzdzal;William M. Baldwin.
Cancer Research (2002)
The effect of soluble complement receptor type 1 on hyperacute rejection of porcine xenografts
Scott K. Pruitt;Allan D. Kirk;R. Randal Bollinger;Henry C. Marsh.
Cytokines, adhesion molecules, and the pathogenesis of chronic rejection of rat renal allografts.
Wayne Hancock;W. Whitley;Stefan Tullius;Uwe Heemann.
Complement in organ transplantation. Contributions to inflammation, injury, and rejection.
William M. Baldwin;Scott K. Pruitt;Robert B. Brauer;Mohamed R. Daha.
B Cell Deficiency Confers Protection from Renal Ischemia Reperfusion Injury
Melissa J. Burne-Taney;Dolores B. Ascon;Frank Daniels;Lorraine Racusen.
Journal of Immunology (2003)
The effect of soluble complement receptor type 1 on hyperacute xenograft rejection.
Scott K. Pruitt;William M. Baldwin;Henry C. Marsh;Shu S. Lin.
Antibody-mediated rejection in human cardiac allografts: evaluation of immunoglobulins and complement activation products C4d and C3d as markers.
E. R. Rodriguez;Diane V. Skojec;Carmela D. Tan;Andrea A. Zachary.
American Journal of Transplantation (2005)
Use of C6-deficient rats to evaluate the mechanism of hyperacute rejection of discordant cardiac xenografts.
Robert B. Brauer;William M. Baldwin;Mohamed R. Daha;Scott K. Pruitt.
Journal of Immunology (1993)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below: