D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 78 Citations 20,137 430 World Ranking 1108 National Ranking 581
Medicine D-index 78 Citations 20,716 489 World Ranking 12932 National Ranking 6729

Research.com Recognitions

Awards & Achievements

2005 - Polish Academy of Science

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Immune system

Reperfusion injury, Immunology, Transplantation, Ischemia and Heme oxygenase are his primary areas of study. His study in Reperfusion injury is interdisciplinary in nature, drawing from both Inflammation, TLR4, Endocrinology and Liver transplantation. His Immunology study combines topics in areas such as Cancer research and Organ transplantation.

His Transplantation research is multidisciplinary, relying on both Cytoprotection, Apoptosis, Endothelium and Bioinformatics. In his research on the topic of Ischemia, Isograft is strongly related with Pathology. His Heme oxygenase research incorporates themes from Andrology, Liver function and Pharmacology.

His most cited work include:

  • Upregulation of heme oxygenase-1 protects genetically fat Zucker rat livers from ischemia/reperfusion injury. (463 citations)
  • Upregulation of heme oxygenase-1 protects genetically fat Zucker rat livers from ischemia/reperfusion injury. (463 citations)
  • Cutting Edge: TLR4 Activation Mediates Liver Ischemia/Reperfusion Inflammatory Response via IFN Regulatory Factor 3-Dependent MyD88-Independent Pathway (398 citations)

What are the main themes of his work throughout his whole career to date?

Jerzy W. Kupiec-Weglinski spends much of his time researching Immunology, Transplantation, Reperfusion injury, Ischemia and Immune system. His Transplantation study integrates concerns from other disciplines, such as T lymphocyte, Immune tolerance and Heme oxygenase. His research investigates the link between Reperfusion injury and topics such as Liver transplantation that cross with problems in Viaspan.

His study in Ischemia is interdisciplinary in nature, drawing from both Ex vivo, Liver function and Pathology. His Immune system study combines topics from a wide range of disciplines, such as Antigen and Cell biology. His work carried out in the field of Internal medicine brings together such families of science as Hepatocyte and Cardiology.

He most often published in these fields:

  • Immunology (57.06%)
  • Transplantation (36.81%)
  • Reperfusion injury (30.47%)

What were the highlights of his more recent work (between 2011-2021)?

  • Reperfusion injury (30.47%)
  • Cancer research (14.11%)
  • Immunology (57.06%)

In recent papers he was focusing on the following fields of study:

Jerzy W. Kupiec-Weglinski focuses on Reperfusion injury, Cancer research, Immunology, Ischemia and Internal medicine. The concepts of his Reperfusion injury study are interwoven with issues in Liver transplantation, Transplantation, Inflammation, Proinflammatory cytokine and Pharmacology. His Transplantation research includes elements of Hindlimb, Endothelial stem cell and Pathology.

Jerzy W. Kupiec-Weglinski combines subjects such as Protein kinase B, Cytoprotection, T cell and PI3K/AKT/mTOR pathway, PTEN with his study of Cancer research. His work on Immune system, Cytokine, Immunity and Adoptive cell transfer as part of general Immunology study is frequently linked to Hepatoprotection, bridging the gap between disciplines. As a part of the same scientific family, he mostly works in the field of Internal medicine, focusing on Endocrinology and, on occasion, Necrosis.

Between 2011 and 2021, his most popular works were:

  • Ischaemia-reperfusion injury in liver transplantation--from bench to bedside. (363 citations)
  • ASC/caspase‐1/IL‐1β signaling triggers inflammatory responses by promoting HMGB1 induction in liver ischemia/reperfusion injury (115 citations)
  • ATF6 Mediates a Pro‐Inflammatory Synergy Between ER Stress and TLR Activation in the Pathogenesis of Liver Ischemia‐Reperfusion Injury (92 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Immune system

Jerzy W. Kupiec-Weglinski mainly investigates Reperfusion injury, Cancer research, Immunology, Inflammation and Proinflammatory cytokine. The Reperfusion injury study combines topics in areas such as Endocrinology, Endoplasmic reticulum and TLR4. His Immunology research integrates issues from Apoptosis and Liver transplantation, Transplantation.

His research integrates issues of Innate immune system, Immune system and Macrophage, Macrophage polarization in his study of Inflammation. His Proinflammatory cytokine research focuses on Liver injury and how it connects with Inflammasome and Pyrin domain. The various areas that Jerzy W. Kupiec-Weglinski examines in his Ischemia study include Autophagy and Pharmacology.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Upregulation of heme oxygenase-1 protects genetically fat Zucker rat livers from ischemia/reperfusion injury.

Farin Amersi;Roland Buelow;Hirohisa Kato;Bibo Ke.
Journal of Clinical Investigation (1999)

637 Citations

Ischaemia-reperfusion injury in liver transplantation--from bench to bedside.

Yuan Zhai;Henrik Petrowsky;Johnny C. Hong;Ronald W. Busuttil.
Nature Reviews Gastroenterology & Hepatology (2013)

585 Citations

Hepatic ischemia/reperfusion injury—a fresh look

Constantino Fondevila;Ronald W Busuttil;Jerzy W Kupiec-Weglinski.
Experimental and Molecular Pathology (2003)

563 Citations

Cutting Edge: TLR4 Activation Mediates Liver Ischemia/Reperfusion Inflammatory Response via IFN Regulatory Factor 3-Dependent MyD88-Independent Pathway

Yuan Zhai;Xiu Da Shen;Ryan O'Connell;Feng Gao.
Journal of Immunology (2004)

515 Citations

Liver ischemia and reperfusion injury: new insights into mechanisms of innate-adaptive immune-mediated tissue inflammation.

Yuan Zhai;Ronald W. Busuttil;Jerzy W. Kupiec-Weglinski.
American Journal of Transplantation (2011)

444 Citations

National conference to assess antibody-mediated rejection in solid organ transplantation.

Steven K. Takemoto;Adriana Zeevi;Sandy Feng;Robert B. Colvin.
American Journal of Transplantation (2004)

410 Citations

Chronic rejection--an undefined conundrum.

Tilney Nl;Whitley Wd;Kupiec-Weglinski Jw.
Transplantation (1991)

403 Citations

Ex vivo exposure to carbon monoxide prevents hepatic ischemia/reperfusion injury through p38 MAP kinase pathway.

Farin Amersi;Xiu-Da Shen;Dean Anselmo;Judy Melinek.
Hepatology (2002)

331 Citations

Migration patterns of dendritic cells in the mouse. Homing to T cell-dependent areas of spleen, and binding within marginal zone.

Jonathan M. Austyn;Jerzy W. Kupiec-Weglinski;Deborah F. Hankins;Peter J. Morris.
Journal of Experimental Medicine (1988)

323 Citations

Ischemia and reperfusion injury in liver transplantation.

J.W. Kupiec-Weglinski;R.W. Busuttil.
International Congress on Immunosuppression (2005)

312 Citations

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