Hugo C. Castro-Faria-Neto spends much of his time researching Immunology, Sepsis, Cytokine, Inflammation and Biochemistry. The study incorporates disciplines such as Apoptosis and Pharmacology in addition to Immunology. Hugo C. Castro-Faria-Neto interconnects Cerebral Malaria, Encephalopathy, Neuroinflammation, Chloroquine and Plasmodium berghei in the investigation of issues within Sepsis.
His Cytokine research incorporates elements of Proinflammatory cytokine, Dengue virus, Intensive care and Platelet activation. His study in Inflammation is interdisciplinary in nature, drawing from both Macrophage, P-selectin and Dengue fever. The concepts of his Biochemistry study are interwoven with issues in Platelet-activating factor and Cell biology.
Hugo C. Castro-Faria-Neto mainly focuses on Immunology, Internal medicine, Inflammation, Endocrinology and Pharmacology. His works in Eosinophil, Sepsis, Cytokine, Lipopolysaccharide and Eotaxin are all subjects of inquiry into Immunology. His work carried out in the field of Cytokine brings together such families of science as Macrophage and Intensive care.
In general Internal medicine study, his work on Community-acquired pneumonia often relates to the realm of NEFA, thereby connecting several areas of interest. His work on Chemokine as part of general Inflammation study is frequently linked to Low level laser therapy, therefore connecting diverse disciplines of science. The various areas that Hugo C. Castro-Faria-Neto examines in his Endocrinology study include Receptor, Leukotriene B4, Peritoneal cavity and Neutrophilia.
His scientific interests lie mostly in Inflammation, Internal medicine, Immunology, Endocrinology and Mesenchymal stem cell. His studies in Inflammation integrate themes in fields like Tumor necrosis factor alpha, Cytokine, Sepsis, Macrophage and Synaptophysin. His Internal medicine research is multidisciplinary, relying on both Gastroenterology and Oleic acid.
His Immunology study frequently intersects with other fields, such as Lung. As part of the same scientific family, he usually focuses on Endocrinology, concentrating on CXCL1 and intersecting with Stimulation, Lipid droplet and Ex vivo. His study on Mesenchymal stem cell also encompasses disciplines like
Hugo C. Castro-Faria-Neto mostly deals with Inflammation, Endocrinology, Internal medicine, Sepsis and Immunology. His research integrates issues of Tumor necrosis factor alpha, CD18, Pharmacology and Peritoneal Infection in his study of Inflammation. His study looks at the relationship between Endocrinology and fields such as Peritoneal cavity, as well as how they intersect with chemical problems.
Hugo C. Castro-Faria-Neto works on Internal medicine which deals in particular with Neuroinflammation. His research on Sepsis frequently links to adjacent areas such as Cytokine. His research in Immunology intersects with topics in Oxidative stress and Tyrosine-kinase inhibitor.
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Cytokine profiles as markers of disease severity in sepsis: a multiplex analysis
Fernando A. Bozza;Fernando A. Bozza;Jorge I F Salluh;André M Japiassú;André M Japiassú;Marcio Soares.
Critical Care (2007)
Uptake of apoptotic cells drives the growth of a pathogenic trypanosome in macrophages.
C. G. Freire-de-Lima;D. O. Nascimento;M. B. P. Soares;P. T. Bozza.
Low-Level Laser Therapy Induces Dose-Dependent Reduction of TNFα Levels in Acute Inflammation
F. Aimbire;R. Albertini;M.T.T. Pacheco;H.C. Castro-Faria-Neto.
Photomedicine and Laser Surgery (2006)
Mycobacterium bovis bacillus Calmette-Guérin induces TLR2-mediated formation of lipid bodies: intracellular domains for eicosanoid synthesis in vivo.
Heloisa D’Avila;Rossana C. N. Melo;Gleydes G. Parreira;Eduardo Werneck-Barroso.
Journal of Immunology (2006)
Inflammatory Platelet-activating Factor-like Phospholipids in Oxidized Low Density Lipoproteins Are Fragmented Alkyl Phosphatidylcholines
Gopal K. Marathe;Sean S. Davies;Kathleen A. Harrison;Adriana R. Silva.
Journal of Biological Chemistry (1999)
MACROPHAGE MIGRATION INHIBITORY FACTOR LEVELS CORRELATE WITH FATAL OUTCOME IN SEPSIS
Fernando A. Bozza;Rachel N. Gomes;André M. Japiassu;Marcio Soares.
Mycobacterium bovis Bacillus Calmette-Guérin Infection Induces TLR2-Dependent Peroxisome Proliferator-Activated Receptor γ Expression and Activation: Functions in Inflammation, Lipid Metabolism, and Pathogenesis
Patrícia E. Almeida;Adriana R. Silva;Clarissa M. Maya-Monteiro;Dániel Töröcsik.
Journal of Immunology (2009)
Effects of different protocol doses of low power gallium–aluminum–arsenate (Ga–Al–As) laser radiation (650 nm) on carrageenan induced rat paw ooedema
R Albertini;F.S.C Aimbire;F.I Correa;W Ribeiro.
Journal of Photochemistry and Photobiology B-biology (2004)
Lipopolysaccharide-Induced Leukocyte Lipid Body Formation In Vivo: Innate Immunity Elicited Intracellular Loci Involved in Eicosanoid Metabolism
Patrícia Pacheco;Fernando A. Bozza;Rachel N. Gomes;Marcelo Bozza.
Journal of Immunology (2002)
Leptin Induces Macrophage Lipid Body Formation by a Phosphatidylinositol 3-Kinase- and Mammalian Target of Rapamycin-dependent Mechanism
Clarissa M. Maya-Monteiro;Patricia E. Almeida;Heloisa D'Ávila;Aline S. Martins.
Journal of Biological Chemistry (2008)
Profile was last updated on December 6th, 2021.
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