Sarah L. Gaffen mostly deals with Immunology, Interleukin 17, Cytokine, Immune system and Interleukin. She has included themes like Periodontitis, Receptor, Signal transduction and Candida albicans in her Immunology study. Her studies deal with areas such as Chemokine, T cell, Virology and Cell biology as well as Interleukin 17.
Her Cytokine research incorporates elements of Inflammation and Transcription factor. Sarah L. Gaffen works mostly in the field of Immune system, limiting it down to concerns involving Interleukin 12 and, occasionally, Corpus albicans and Oropharyngeal Candidiasis. In her study, Bone cell is inextricably linked to Tumor necrosis factor alpha, which falls within the broad field of Interleukin.
Sarah L. Gaffen mainly focuses on Immunology, Interleukin 17, Cytokine, Candida albicans and Signal transduction. Her work in Immunology is not limited to one particular discipline; it also encompasses Oropharyngeal Candidiasis. Her Interleukin 17 research is multidisciplinary, incorporating elements of T cell, Chemokine, Proinflammatory cytokine, Interleukin and Kidney.
Her work deals with themes such as Tumor necrosis factor alpha, Adipogenesis, Autoimmunity and Pathogenesis, which intersect with Cytokine. In general Candida albicans study, her work on Corpus albicans and Disseminated Candidiasis often relates to the realm of Candidalysin, thereby connecting several areas of interest. She combines subjects such as Receptor, Transcription factor, Kinase and Molecular biology with her study of Signal transduction.
Sarah L. Gaffen mainly investigates Immunology, Candida albicans, Interleukin 17, Cytokine and Corpus albicans. Specifically, her work in Immunology is concerned with the study of Inflammation. Her biological study spans a wide range of topics, including Cancer research and Immunity.
Her research in Interleukin 17 intersects with topics in Tumor necrosis factor alpha, Molecular biology, Signal transduction, Proinflammatory cytokine and Interleukin. Her Cytokine study combines topics in areas such as Autoimmunity, Pathogenesis and Cell biology. Her research integrates issues of Autoantibody, Chronic Candidiasis, Immune system and Autoimmune regulator in her study of Corpus albicans.
Her scientific interests lie mostly in Corpus albicans, Immunology, Candida albicans, Candidalysin and Microbiology. Her Corpus albicans study frequently draws parallels with other fields, such as Immune system. Her Immunology study deals with Regulation of gene expression intersecting with Cytokine.
The study incorporates disciplines such as Toxin, Secretion and Immunity in addition to Candida albicans. In her study, which falls under the umbrella issue of Microbiology, Acquired immune system is strongly linked to Innate immune system. Her Interleukin 17 study combines topics from a wide range of disciplines, such as Signal transduction and Disease.
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IL-23 and IL-17 in the establishment of protective pulmonary CD4 + T cell responses after vaccination and during Mycobacterium tuberculosis challenge
Shabaana A Khader;Guy K Bell;John E Pearl;Jeffrey J Fountain.
Nature Immunology (2007)
Structure and signalling in the IL-17 receptor family.
Sarah L. Gaffen.
Nature Reviews Immunology (2009)
The IL-23-IL-17 immune axis: from mechanisms to therapeutic testing
Sarah L. Gaffen;Renu Jain;Abhishek V. Garg;Daniel J. Cua.
Nature Reviews Immunology (2014)
Th17 cells and IL-17 receptor signaling are essential for mucosal host defense against oral candidiasis
Heather R. Conti;Fang Shen;Namrata Nayyar;Eileen Stocum.
Journal of Experimental Medicine (2009)
Interleukin-17 and its target genes: mechanisms of interleukin-17 function in disease.
Reiko M Onishi;Sarah L Gaffen;Sarah L Gaffen.
Overview of interleukin-2 function, production and clinical applications.
Sarah L. Gaffen;Kathleen D. Liu.
An overview of IL-17 function and signaling.
Sarah L. Gaffen.
Th17 cells at the crossroads of innate and adaptive immunity against infectious diseases at the mucosa
Shabaana A. Khader;Sarah L. Gaffen;Jay K. Kolls.
Mucosal Immunology (2009)
1,25-Dihydroxyvitamin D 3 ameliorates Th17 autoimmunity via transcriptional modulation of interleukin-17A
Sneha Joshi;Luiz Carlos Pantalena;Xikui K. Liu;Sarah L. Gaffen;Sarah L. Gaffen.
Molecular and Cellular Biology (2011)
Functional Cooperation between Interleukin-17 and Tumor Necrosis Factor-α Is Mediated by CCAAT/Enhancer-binding Protein Family Members
Matthew J. Ruddy;Grace C. Wong;Xikui K. Liu;Hiroyasu Yamamoto.
Journal of Biological Chemistry (2004)
Profile was last updated on December 6th, 2021.
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