D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 125 Citations 52,500 368 World Ranking 96 National Ranking 66
Medicine D-index 132 Citations 56,452 442 World Ranking 941 National Ranking 561

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • Immune system

James G. Krueger mainly investigates Immunology, Psoriasis, Immune system, Interleukin 17 and Tumor necrosis factor alpha. Immunology is closely attributed to Disease in his work. James G. Krueger interconnects Surgery, Internal medicine, Risankizumab and Keratinocyte in the investigation of issues within Psoriasis.

His work in Keratinocyte addresses subjects such as Dermis, which are connected to disciplines such as Molecular biology. His Immune system research includes themes of Myeloid and Genetic predisposition. His Interleukin 17 research is multidisciplinary, relying on both Interleukin, Interleukin 22, Keratin 16 and Interferon gamma.

His most cited work include:

  • Pathogenesis and therapy of psoriasis (1274 citations)
  • Brodalumab, an Anti–Interleukin-17–Receptor Antibody for Psoriasis (826 citations)
  • Psoriasis vulgaris lesions contain discrete populations of Th1 and Th17 T cells. (791 citations)

What are the main themes of his work throughout his whole career to date?

Immunology, Psoriasis, Atopic dermatitis, Immune system and Pathology are his primary areas of study. Immunology is frequently linked to Disease in his study. James G. Krueger has researched Psoriasis in several fields, including Tumor necrosis factor alpha, Internal medicine and Interleukin 17.

His work carried out in the field of Internal medicine brings together such families of science as Gastroenterology, Endocrinology and Oncology. His research on Immune system focuses in particular on Dendritic cell. His Pathology study often links to related topics such as Keratinocyte.

He most often published in these fields:

  • Immunology (48.95%)
  • Psoriasis (45.47%)
  • Atopic dermatitis (19.69%)

What were the highlights of his more recent work (between 2018-2021)?

  • Psoriasis (45.47%)
  • Immunology (48.95%)
  • Atopic dermatitis (19.69%)

In recent papers he was focusing on the following fields of study:

James G. Krueger mostly deals with Psoriasis, Immunology, Atopic dermatitis, Internal medicine and Dermatology. His study focuses on the intersection of Psoriasis and fields such as Interferon with connections in the field of Tyrosine kinase 2. His Immune system, Cytokine, Dendritic cell, Chemokine and Filaggrin study are his primary interests in Immunology.

His Atopic dermatitis research incorporates elements of Biomarker, T cell, Systemic inflammation and SCORAD. His studies in Internal medicine integrate themes in fields like Gastroenterology and Oncology. In his study, Adalimumab and Brodalumab is inextricably linked to Hidradenitis suppurativa, which falls within the broad field of Dermatology.

Between 2018 and 2021, his most popular works were:

  • Dupilumab progressively improves systemic and cutaneous abnormalities in patients with atopic dermatitis (152 citations)
  • Atopic dermatitis endotypes and implications for targeted therapeutics. (122 citations)
  • Risankizumab compared with adalimumab in patients with moderate-to-severe plaque psoriasis (IMMvent): a randomised, double-blind, active-comparator-controlled phase 3 trial. (69 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • Cancer

His primary areas of study are Immunology, Atopic dermatitis, Psoriasis, Internal medicine and Inflammation. His works in Cytokine, Immune system, Dendritic cell, Interleukin and Filaggrin are all subjects of inquiry into Immunology. James G. Krueger has included themes like Immunohistochemistry, SCORAD, Disease, Biomarker and Immunoglobulin E in his Atopic dermatitis study.

James G. Krueger studies Psoriasis Area and Severity Index which is a part of Psoriasis. His Internal medicine research focuses on subjects like Gastroenterology, which are linked to Area under the curve and Keratin 16. The study incorporates disciplines such as Skin biopsy, Endothelium and Pathogenesis in addition to Inflammation.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Pathogenesis and therapy of psoriasis

Michelle A. Lowes;Anne M. Bowcock;James G. Krueger.
Nature (2007)

1889 Citations

Psoriasis vulgaris lesions contain discrete populations of Th1 and Th17 T cells.

Michelle A. Lowes;Toyoko Kikuchi;Judilyn Fuentes-Duculan;Irma Cardinale.
Journal of Investigative Dermatology (2008)

1151 Citations

Immunology of Psoriasis

Michelle A Lowes;Mayte Suárez-Fariñas;James G Krueger.
Annual Review of Immunology (2014)

1032 Citations

Brodalumab, an Anti–Interleukin-17–Receptor Antibody for Psoriasis

Kim A Papp;Craig Leonardi;Alan Menter;Jean-Paul Ortonne.
The New England Journal of Medicine (2012)

1017 Citations

Increased Expression of Interleukin 23 p19 and p40 in Lesional Skin of Patients with Psoriasis Vulgaris

Edmund Jd Lee;William L. Trepicchio;Judith L Oestreicher;Debra D Pittman.
Journal of Experimental Medicine (2004)

1004 Citations

Interleukin 6 is expressed in high levels in psoriatic skin and stimulates proliferation of cultured human keratinocytes.

Rachel M. Grossman;James Krueger;Debra Yourish;Angela Granelli-Piperno.
Proceedings of the National Academy of Sciences of the United States of America (1989)

973 Citations

The immunologic basis for the treatment of psoriasis with new biologic agents

James G. Krueger.
Journal of The American Academy of Dermatology (2002)

925 Citations

Th17 cytokines interleukin (IL)-17 and IL-22 modulate distinct inflammatory and keratinocyte-response pathways.

K.E. Nograles;L.C. Zaba;E. Guttman-Yassky;J. Fuentes-Duculan.
British Journal of Dermatology (2008)

776 Citations

CTLA4Ig-mediated blockade of T cell costimulation in patients with psoriasis vulgaris

Judith R. Abrams;Mark G. Lebwohl;Cynthia A. Guzzo;Brian V. Jegasothy.
Journal of Clinical Investigation (1999)

738 Citations

Psoriasis pathophysiology: current concepts of pathogenesis

J G Krueger;A Bowcock.
Annals of the Rheumatic Diseases (2005)

725 Citations

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