Kenji Kabashima mostly deals with Immunology, Immune system, Cell biology, Atopic dermatitis and Inflammation. His Immunology study frequently intersects with other fields, such as Keratinocyte. His Immune system study incorporates themes from Melanoma and Lymph.
The study incorporates disciplines such as Cytotoxic T cell, IL-2 receptor, Chemokine, Receptor and Dermis in addition to Cell biology. His Atopic dermatitis study combines topics from a wide range of disciplines, such as Psoriasis, Internal medicine, Pathogenesis and T helper cell. His Inflammation study combines topics in areas such as Anatomy and Cutaneous immunity.
His scientific interests lie mostly in Immunology, Dermatology, Immune system, Pathology and Atopic dermatitis. His research in Inflammation, Antigen, Psoriasis, Pathogenesis and Immunoglobulin E are components of Immunology. His study on Immune system is mostly dedicated to connecting different topics, such as Cell biology.
His Atopic dermatitis study frequently links to other fields, such as Internal medicine.
His primary areas of study are Dermatology, Atopic dermatitis, Immune system, Immunology and Inflammation. In his study, Melanoma is strongly linked to Nivolumab, which falls under the umbrella field of Dermatology. His biological study spans a wide range of topics, including Internal medicine, Randomized controlled trial and Clinical endpoint.
His Immune system research is multidisciplinary, incorporating perspectives in Cell type, Intravital Imaging and Pathology. His studies in Thymic stromal lymphopoietin, Immunoglobulin E, Filaggrin, Acquired immune system and Pathogenesis are all subfields of Immunology research. His work carried out in the field of Inflammation brings together such families of science as Dendritic cell, Antibody, Innate lymphoid cell and Cell biology.
His primary scientific interests are in Atopic dermatitis, Immunology, Immune system, Inflammation and Dermatology. His Atopic dermatitis research includes elements of Internal medicine, Quality of life, Clinical trial and Interleukin 15. His study in Incidence extends to Immunology with its themes.
His research ties Cytotoxic T cell and Immune system together. His Inflammation research incorporates themes from Antigen, Antibody, Innate lymphoid cell, In vivo and Histamine. Kenji Kabashima has included themes like Epidermolysis bullosa acquisita, Spirometry, Pemphigoid and Intravenous Immunoglobulins in his Dermatology study.
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Prostaglandin D2 as a mediator of allergic asthma.
Toshiyuki Matsuoka;Masakazu Hirata;Hiroyuki Tanaka;Yoshimasa Takahashi.
Tumor cell expression of programmed cell death-1 ligand 1 is a prognostic factor for malignant melanoma†
Ryosuke Hino;Kenji Kabashima;Kenji Kabashima;Yu Kato;Hiroaki Yagi.
Possible pathogenic role of Th17 cells for atopic dermatitis.
Chizuko Koga;Kenji Kabashima;Noriko Shiraishi;Miwa Kobayashi.
Journal of Investigative Dermatology (2008)
The prostaglandin receptor EP4 suppresses colitis, mucosal damage and CD4 cell activation in the gut
Kenji Kabashima;Tomomi Saji;Takahiko Murata;Miyako Nagamachi.
Journal of Clinical Investigation (2002)
Anti-PD-1 and Anti-CTLA-4 Therapies in Cancer: Mechanisms of Action, Efficacy, and Limitations.
Judith A. Seidel;Atsushi Otsuka;Kenji Kabashima;Kenji Kabashima.
Frontiers in Oncology (2018)
Anti-interleukin-31 receptor a antibody for atopic dermatitis
Thomas Ruzicka;Jon M. Hanifin;Masutaka Furue;Grazyna Pulka.
The New England Journal of Medicine (2017)
Prostaglandin E2-EP4 signaling initiates skin immune responses by promoting migration and maturation of Langerhans cells.
Kenji Kabashima;Daiji Sakata;Miyako Nagamachi;Yoshiki Miyachi.
Nature Medicine (2003)
Intrinsic Lymphotoxin-β Receptor Requirement for Homeostasis of Lymphoid Tissue Dendritic Cells
Kenji Kabashima;Theresa A. Banks;K. Mark Ansel;Theresa T. Lu.
New concept of the pathogenesis of atopic dermatitis: interplay among the barrier, allergy, and pruritus as a trinity.
Journal of Dermatological Science (2013)
The Asian atopic dermatitis phenotype combines features of atopic dermatitis and psoriasis with increased TH17 polarization.
Shinji Noda;Mayte Suárez-Fariñas;Benjamin Ungar;Benjamin Ungar;Soo Jung Kim.
The Journal of Allergy and Clinical Immunology (2015)
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