1941 - Fellow of the American Association for the Advancement of Science (AAAS)
James T. Elder focuses on Psoriasis, Genetics, Immunology, Gene and Genome-wide association study. His work carried out in the field of Psoriasis brings together such families of science as Internal medicine, Case-control study, Disease and Keratinocyte. His study in Locus, Single-nucleotide polymorphism, Allele, Human leukocyte antigen and Haplotype is carried out as part of his studies in Genetics.
His research investigates the link between Immunology and topics such as Cancer research that cross with problems in Transforming growth factor beta, Transforming growth factor, beta 3 and TGF alpha. As a part of the same scientific study, James T. Elder usually deals with the Gene, concentrating on Autoimmunity and frequently concerns with Genomics, DNA microarray, Autoantibody and Medical genetics. The concepts of his Genome-wide association study study are interwoven with issues in Genetic association and Arthritis.
His primary scientific interests are in Psoriasis, Genetics, Immunology, Molecular biology and Genome-wide association study. The study incorporates disciplines such as Tumor necrosis factor alpha, Internal medicine, Disease and Gene in addition to Psoriasis. His is involved in several facets of Genetics study, as is seen by his studies on Locus, Human leukocyte antigen, Allele, Haplotype and Genetic association.
In his study, which falls under the umbrella issue of Immunology, Transcription factor is strongly linked to Cancer research. He interconnects Gene expression, Keratinocyte, Receptor, Messenger RNA and Human skin in the investigation of issues within Molecular biology. His Genome-wide association study study is concerned with Single-nucleotide polymorphism in general.
James T. Elder mainly focuses on Psoriasis, Genome-wide association study, Genetic association, Internal medicine and Psoriatic arthritis. His Psoriasis research incorporates elements of Genetics, Meta-analysis, Disease, Atopic dermatitis and Genetic architecture. Atopic dermatitis is a subfield of Immunology that he investigates.
His Genome-wide association study research is multidisciplinary, incorporating elements of Odds ratio and Immune system. His Genetic association research is multidisciplinary, relying on both Imputation, Human leukocyte antigen and Major histocompatibility complex. His Internal medicine study combines topics from a wide range of disciplines, such as Linkage disequilibrium and Oncology.
James T. Elder mainly investigates Psoriasis, Internal medicine, Psoriatic arthritis, Genome-wide association study and Atopic dermatitis. James T. Elder combines subjects such as Tyrosine kinase 2, Drug repositioning, CD28, Molecular biology and Gene with his study of Psoriasis. Gene is closely attributed to Immunology in his work.
His research in Internal medicine intersects with topics in Genetic marker and Genetic architecture. James T. Elder has included themes like Genetics, Cohort study, Oncology, Genetic association and Cohort in his Psoriatic arthritis study. His Genome-wide association study study is focused on Single-nucleotide polymorphism in general.
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Genome-wide scan reveals association of psoriasis with IL-23 and NF-κB pathways
Rajan P. Nair;Kristina Callis Duffin;Cynthia Helms;Jun Ding.
Nature Genetics (2009)
Identification of 15 new psoriasis susceptibility loci highlights the role of innate immunity
Lam C. Tsoi;Sarah L. Spain;Sarah L. Spain;Jo Knight;Eva Ellinghaus;Eva Ellinghaus.
Nature Genetics (2012)
Sequence and Haplotype Analysis Supports HLA-C as the Psoriasis Susceptibility 1 Gene
Rajan P. Nair;Philip E. Stuart;Ioana Nistor;Ravi Hiremagalore.
American Journal of Human Genetics (2006)
Overexpression of transforming growth factor alpha in psoriatic epidermis
James T. Elder;Gary J. Fisher;Patricia B. Lindquist;Gregory L. Bennett.
Science (1989)
Cellular localization of interleukin-8 and its inducer, tumor necrosis factor-alpha in psoriasis.
B. J. Nickoloff;G. D. Karabin;J. N. W. N. Barker;C. E. M. Griffiths.
American Journal of Pathology (1991)
Evidence for Two Psoriasis Susceptibility Loci (HLA and 17q) and Two Novel Candidate Regions (16q and 20p) by Genome-Wide Scan
Rajan P. Nair;Tilo Henseler;Stefan Jenisch;Philip Stuart.
Human Molecular Genetics (1997)
Deletion of the late cornified envelope LCE3B and LCE3C genes as a susceptibility factor for psoriasis
Rafael De Cid;Eva Riveira-Munoz;Patrick L.J.M. Zeeuwen;Jason Robarge.
Nature Genetics (2009)
Pervasive sharing of genetic effects in autoimmune disease.
Chris Cotsapas;Benjamin F. Voight;Benjamin F. Voight;Elizabeth Rossin;Kasper Lage;Kasper Lage;Kasper Lage.
PLOS Genetics (2011)
Induction of IL-17+ T Cell Trafficking and Development by IFN-γ: Mechanism and Pathological Relevance in Psoriasis
Ilona Kryczek;Allen T. Bruce;Johann E. Gudjonsson;Andrew Johnston.
Journal of Immunology (2008)
Fine mapping of type 1 diabetes susceptibility loci and evidence for colocalization of causal variants with lymphoid gene enhancers.
Suna Onengut-Gumuscu;Wei-Min Chen;Oliver Burren;Nick J Cooper.
Nature Genetics (2015)
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