D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Immunology D-index 121 Citations 49,248 429 World Ranking 114 National Ranking 80
Medicine D-index 125 Citations 53,481 501 World Ranking 1235 National Ranking 742

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Internal medicine
  • DNA

John B. Harley spends much of his time researching Immunology, Lupus erythematosus, Genetics, Autoimmune disease and Systemic lupus erythematosus. His Immunology study which covers Allele that intersects with Locus. His studies deal with areas such as Twin study, Immune system, Internal medicine, Genetic association and Anti-SSA/Ro autoantibodies as well as Lupus erythematosus.

His work on Genetics deals in particular with Genetic linkage, Candidate gene, Human leukocyte antigen, Genome-wide association study and Genetic predisposition. The Autoimmune disease study combines topics in areas such as Epigenetics and X chromosome. John B. Harley has included themes like Virus and Epstein–Barr virus in his Systemic lupus erythematosus study.

His most cited work include:

  • Development of autoantibodies before the clinical onset of systemic lupus erythematosus. (1775 citations)
  • Human polymorphism at microRNAs and microRNA target sites. (1284 citations)
  • Genome-wide association scan in women with systemic lupus erythematosus identifies susceptibility variants in ITGAM, PXK, KIAA1542 and other loci. (1069 citations)

What are the main themes of his work throughout his whole career to date?

His primary areas of study are Immunology, Genetics, Systemic lupus erythematosus, Lupus erythematosus and Autoantibody. His research on Immunology frequently connects to adjacent areas such as Single-nucleotide polymorphism. John B. Harley combines subjects such as Nephritis, Family aggregation, Gene and Rheumatology with his study of Systemic lupus erythematosus.

The study incorporates disciplines such as Internal medicine, Connective tissue disease and Genetic predisposition in addition to Lupus erythematosus. His Autoantibody research focuses on Molecular biology and how it connects with RNA and Ribonucleoprotein. His Allele research is multidisciplinary, incorporating elements of Human leukocyte antigen and Genotype.

He most often published in these fields:

  • Immunology (102.32%)
  • Genetics (65.98%)
  • Systemic lupus erythematosus (44.54%)

What were the highlights of his more recent work (between 2015-2021)?

  • Genetics (65.98%)
  • Genome-wide association study (25.68%)
  • Immunology (102.32%)

In recent papers he was focusing on the following fields of study:

John B. Harley focuses on Genetics, Genome-wide association study, Immunology, Disease and Internal medicine. His research integrates issues of Genotyping, Linkage disequilibrium, Genetic association and Genetic architecture in his study of Genome-wide association study. His study in Autoantibody, Autoimmunity, Autoimmune disease, Immune system and Human leukocyte antigen is carried out as part of his studies in Immunology.

The various areas that he examines in his Autoantibody study include Systemic lupus erythematosus, Immune dysregulation, B-cell activating factor and Rheumatology. His study in Internal medicine is interdisciplinary in nature, drawing from both Lupus erythematosus, Endocrinology and Oncology. His Lupus erythematosus study combines topics in areas such as Connective tissue disease and Polymorphism.

Between 2015 and 2021, his most popular works were:

  • Transancestral mapping and genetic load in systemic lupus erythematosus (143 citations)
  • High-density genotyping of immune-related loci identifies new SLE risk variants in individuals with Asian ancestry (133 citations)
  • Transcription factors operate across disease loci, with EBNA2 implicated in autoimmunity (124 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Internal medicine
  • DNA

The scientist’s investigation covers issues in Immunology, Disease, Genome-wide association study, Lupus erythematosus and Genetics. His Immunology and Autoantibody, Autoimmune disease, Autoimmunity, Anti-SSA/Ro autoantibodies and Pathogenesis investigations all form part of his Immunology research activities. His research in Disease intersects with topics in Diagnosis code, Immune system and Cohort.

His Genome-wide association study study combines topics from a wide range of disciplines, such as Medical record, Genetic predisposition, Genetic association and Genetic architecture. His Lupus erythematosus research includes elements of Regulation of gene expression, Rheumatoid arthritis and Polymorphism. In his study, Serology is strongly linked to Single-nucleotide polymorphism, which falls under the umbrella field of Internal medicine.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Development of autoantibodies before the clinical onset of systemic lupus erythematosus.

Melissa R Arbuckle;Micah T McClain;Mark V Rubertone;R Hal Scofield.
The New England Journal of Medicine (2003)

2453 Citations

Human polymorphism at microRNAs and microRNA target sites.

Liuqing Yang;Chunru Lin;Chunyu Jin;Joy C. Yang.
Frontiers in Genetics (2013)

2058 Citations

Genome-wide association scan in women with systemic lupus erythematosus identifies susceptibility variants in ITGAM, PXK, KIAA1542 and other loci.

John B. Harley;John B. Harley;John B. Harley;Marta E. Alarcón-Riquelme;Lindsey A. Criswell;Chaim O. Jacob.
Nature Genetics (2008)

1284 Citations

NIH conference. The idiopathic hypereosinophilic syndrome. Clinical, pathophysiologic, and therapeutic considerations.

Anthony S. Fauci;John B. Harley;William C. Roberts;Victor J. Ferrans.
Annals of Internal Medicine (1982)

1063 Citations

A regulatory polymorphism in PDCD1 is associated with susceptibility to systemic lupus erythematosus in humans

Ludmila Prokunina;Casimiro Castillejo-López;Fredrik Öberg;Iva Gunnarsson.
Nature Genetics (2002)

903 Citations

Changes in the pattern of DNA methylation associate with twin discordance in systemic lupus erythematosus

Biola M. Javierre;Agustin F. Fernandez;Julia Richter;Fatima Al-Shahrour.
Genome Research (2010)

613 Citations

An increased prevalence of Epstein-Barr virus infection in young patients suggests a possible etiology for systemic lupus erythematosus.

J. A. James;K. M. Kaufman;A. D. Farris;E. Taylor-Albert.
Journal of Clinical Investigation (1997)

574 Citations

Genome scan of human systemic lupus erythematosus: Evidence for linkage on chromosome 1q in African-American pedigrees

Kathy L. Moser;Barbara R. Neas;Barbara R. Neas;Jane E. Salmon;Hua Yu.
Proceedings of the National Academy of Sciences of the United States of America (1998)

533 Citations

High serum IFN-alpha activity is a heritable risk factor for systemic lupus erythematosus.

T B Niewold;J Hua;T J A Lehman;J B Harley.
Genes and Immunity (2007)

480 Citations

Early events in lupus humoral autoimmunity suggest initiation through molecular mimicry.

Micah T McClain;Latisha D Heinlen;Latisha D Heinlen;Gregory J Dennis;Jon Roebuck.
Nature Medicine (2005)

446 Citations

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