D-Index & Metrics Best Publications

D-Index & Metrics

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 76 Citations 17,476 137 World Ranking 399 National Ranking 203
Genetics and Molecular Biology D-index 60 Citations 14,466 127 World Ranking 2329 National Ranking 1152
Medicine D-index 78 Citations 18,464 155 World Ranking 10490 National Ranking 5614

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Genetics
  • DNA

Kenneth M. Kaufman mainly focuses on Immunology, Genetics, Single-nucleotide polymorphism, Lupus erythematosus and Haplotype. He combines subjects such as Genetic predisposition, Genotype and Candidate gene with his study of Immunology. His study in Genetic association, Genome-wide association study, Locus, Regulation of gene expression and FCGR2A is done as part of Genetics.

His study in Genome-wide association study is interdisciplinary in nature, drawing from both Human leukocyte antigen, Disease and IL12A. His Haplotype research is within the category of Allele. His Autoimmune disease study incorporates themes from Rheumatoid arthritis, X chromosome and Anti-SSA/Ro autoantibodies.

His most cited work include:

  • Human polymorphism at microRNAs and microRNA target sites. (1284 citations)
  • Genome-wide association scan in women with systemic lupus erythematosus identifies susceptibility variants in ITGAM, PXK, KIAA1542 and other loci. (1069 citations)
  • Genome-wide association scan in women with systemic lupus erythematosus identifies susceptibility variants in ITGAM, PXK, KIAA1542 and other loci. (1069 citations)

What are the main themes of his work throughout his whole career to date?

His scientific interests lie mostly in Immunology, Genetics, Single-nucleotide polymorphism, Systemic lupus erythematosus and Genome-wide association study. The concepts of his Immunology study are interwoven with issues in Haplotype and Genotype. As a member of one scientific family, Kenneth M. Kaufman mostly works in the field of Single-nucleotide polymorphism, focusing on Odds ratio and, on occasion, Lupus nephritis.

His studies in Systemic lupus erythematosus integrate themes in fields like Autoantibody, Antibody, Molecular biology, Genetic linkage and Malar rash. In his research on the topic of Genetic association, Connective tissue disease is strongly related with Genetic predisposition. His research investigates the connection between Lupus erythematosus and topics such as Human leukocyte antigen that intersect with issues in FCGR2A.

He most often published in these fields:

  • Immunology (115.35%)
  • Genetics (82.57%)
  • Single-nucleotide polymorphism (71.37%)

What were the highlights of his more recent work (between 2014-2020)?

  • Genetics (82.57%)
  • Immunology (115.35%)
  • Genome-wide association study (44.40%)

In recent papers he was focusing on the following fields of study:

The scientist’s investigation covers issues in Genetics, Immunology, Genome-wide association study, Allele and Locus. As a part of the same scientific study, Kenneth M. Kaufman usually deals with the Genetics, concentrating on Systemic lupus erythematosus and frequently concerns with B cell. His Immunology research is multidisciplinary, relying on both Case-control study and Genetic predisposition.

His Genome-wide association study study introduces a deeper knowledge of Single-nucleotide polymorphism. His work on SNP is typically connected to Taxane as part of general Single-nucleotide polymorphism study, connecting several disciplines of science. His Genotype research focuses on Lupus erythematosus and how it relates to Rheumatoid arthritis and Anti-SSA/Ro autoantibodies.

Between 2014 and 2020, his most popular works were:

  • Transancestral mapping and genetic load in systemic lupus erythematosus (143 citations)
  • High-density genotyping of immune-related loci identifies new SLE risk variants in individuals with Asian ancestry (133 citations)
  • Transcription factors operate across disease loci, with EBNA2 implicated in autoimmunity (124 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • DNA
  • Genetics

Kenneth M. Kaufman mostly deals with Genome-wide association study, Lupus erythematosus, Immunology, Genetics and Single-nucleotide polymorphism. His study explores the link between Genome-wide association study and topics such as Genetic architecture that cross with problems in Arthritis and Gene polymorphism. His Lupus erythematosus research is multidisciplinary, incorporating perspectives in Human leukocyte antigen, Rheumatoid arthritis, Genotype, Autoimmunity and Anti-SSA/Ro autoantibodies.

His research investigates the connection between Human leukocyte antigen and topics such as Haplotype that intersect with problems in C4A and Sex characteristics. His study looks at the intersection of Immunology and topics like Allele with Genetic predisposition. As a member of one scientific family, Kenneth M. Kaufman mostly works in the field of Locus, focusing on Imputation and, on occasion, Expression quantitative trait loci, Gene mapping, Genetic association and Beta Karyopherins.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

Human polymorphism at microRNAs and microRNA target sites.

Liuqing Yang;Chunru Lin;Chunyu Jin;Joy C. Yang.
Frontiers in Genetics (2013)

2058 Citations

Genome-wide association scan in women with systemic lupus erythematosus identifies susceptibility variants in ITGAM, PXK, KIAA1542 and other loci.

John B. Harley;John B. Harley;John B. Harley;Marta E. Alarcón-Riquelme;Lindsey A. Criswell;Chaim O. Jacob.
Nature Genetics (2008)

1284 Citations

An increased prevalence of Epstein-Barr virus infection in young patients suggests a possible etiology for systemic lupus erythematosus.

J. A. James;K. M. Kaufman;A. D. Farris;E. Taylor-Albert.
Journal of Clinical Investigation (1997)

574 Citations

A functional variant in FCRL3 , encoding Fc receptor-like 3, is associated with rheumatoid arthritis and several autoimmunities

Yuta Kochi;Ryo Yamada;Akari Suzuki;John B. Harley;John B. Harley;John B. Harley.
Nature Genetics (2005)

427 Citations

Variants at multiple loci implicated in both innate and adaptive immune responses are associated with Sjogren's syndrome.

Christopher J. Lessard;Christopher J. Lessard;He Li;He Li;Indra Adrianto;John A. Ice.
Nature Genetics (2013)

375 Citations

A nonsynonymous functional variant in integrin-alpha(M) (encoded by ITGAM) is associated with systemic lupus erythematosus.

Swapan K. Nath;Shizhong Han;Xana Kim-Howard;Jennifer A. Kelly.
Nature Genetics (2008)

320 Citations

Genetic susceptibility to SLE: new insights from fine mapping and genome-wide association studies.

Isaac T. W. Harley;Kenneth M. Kaufman;Kenneth M. Kaufman;Carl D. Langefeld;John B. Harley;John B. Harley.
Nature Reviews Genetics (2009)

297 Citations

Association of a functional variant downstream of TNFAIP3 with systemic lupus erythematosus

Indra Adrianto;Feng Wen;Amanda Templeton;Graham B. Wiley.
Nature Genetics (2011)

250 Citations

Identification of IRAK1 as a risk gene with critical role in the pathogenesis of systemic lupus erythematosus

Chaim O. Jacob;Jiankun Zhu;Don L. Armstrong;Don L. Armstrong;Mei Yan.
Proceedings of the National Academy of Sciences of the United States of America (2009)

249 Citations

Sex-specific association of X-linked Toll-like receptor 7 (TLR7) with male systemic lupus erythematosus

Nan Shen;Qiong Fu;Qiong Fu;Yun Deng;Yun Deng;Xiaoxia Qian.
Proceedings of the National Academy of Sciences of the United States of America (2010)

232 Citations

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