His primary areas of investigation include Human skin, Psoriasis, Internal medicine, Molecular biology and Endocrinology. His studies deal with areas such as Photoaging, Biochemistry, Connective tissue, Cell biology and Retinoic acid as well as Human skin. His Psoriasis study improves the overall literature in Immunology.
His Internal medicine study which covers Gastroenterology that intersects with Urinary system, Urine and Hydrocortisone. His Molecular biology study combines topics from a wide range of disciplines, such as Receptor, Epidermal growth factor, Cell surface receptor, Keratinocyte and Messenger RNA. The study incorporates disciplines such as Erythromycin, Liver transplantation and Epidermis in addition to Endocrinology.
John J. Voorhees mainly focuses on Psoriasis, Dermatology, Human skin, Molecular biology and Internal medicine. He has researched Psoriasis in several fields, including Genetics, Disease and Epidermis. His research integrates issues of Clinical trial, Tretinoin and Chemotherapy in his study of Dermatology.
His Human skin research integrates issues from Dermis, Pathology, Extracellular matrix, Cell biology and In vivo. His work in Molecular biology addresses subjects such as Retinoic acid, which are connected to disciplines such as Retinoid X receptor. His biological study spans a wide range of topics, including Gastroenterology and Endocrinology.
His main research concerns Human skin, Psoriasis, Cell biology, Extracellular matrix and Immunology. His Human skin study incorporates themes from Type I collagen, Dermis, Pathology, Fibroblast and In vivo. His Psoriasis research is multidisciplinary, incorporating perspectives in Genetics, Genome-wide association study, Transcriptome, Interleukin 17 and Disease.
His research in Extracellular matrix intersects with topics in Wound healing, Dermatology, Gene knockdown and Epidermis. His study in Immunology is interdisciplinary in nature, drawing from both Collagenase and Keratinocyte. John J. Voorhees has included themes like Tretinoin and Retinoic acid in his Endocrinology study.
John J. Voorhees mainly investigates Psoriasis, Genetics, Human skin, Immunology and Cell biology. He interconnects Odds ratio, Genome-wide association study, Interleukin 17, Gene and Disease in the investigation of issues within Psoriasis. The study incorporates disciplines such as Endocrinology, Dermis, Biopsy, Pathology and Extracellular matrix in addition to Human skin.
His Pathology study incorporates themes from Photoaging and Skin repair. His study looks at the intersection of Photoaging and topics like Treatment options with Dermatology. His biological study spans a wide range of topics, including Fibroblast, Fragmentation, Connective tissue and Molecular biology.
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Mechanisms of photoaging and chronological skin aging
Gary J. Fisher;Sewon Kang;James Varani;Zsuzsanna Bata-Csorgo.
Archives of Dermatology (2002)
Pathophysiology of Premature Skin Aging Induced by Ultraviolet Light
Gary J. Fisher;Zeng Quan Wang;Subhash C. Datta;James Varani.
The New England Journal of Medicine (1997)
Molecular basis of sun-induced premature skin ageing and retinoid antagonism
Gary J. Fisher;Subhash C. Datta;Harvinder S. Talwar;Zeng Quan Wang.
Nature (1996)
Genome-wide scan reveals association of psoriasis with IL-23 and NF-κB pathways
Rajan P. Nair;Kristina Callis Duffin;Cynthia Helms;Jun Ding.
Nature Genetics (2009)
Identification of 15 new psoriasis susceptibility loci highlights the role of innate immunity
Lam C. Tsoi;Sarah L. Spain;Sarah L. Spain;Jo Knight;Eva Ellinghaus;Eva Ellinghaus.
Nature Genetics (2012)
Induction of Pemphigus in Neonatal Mice by Passive Transfer of IgG from Patients with the Disease
Grant J. Anhalt;Ramzy S. Labib;John J. Voorhees;Theodore F. Beals.
The New England Journal of Medicine (1982)
Decreased collagen production in chronologically aged skin: roles of age-dependent alteration in fibroblast function and defective mechanical stimulation.
James Varani;Michael K. Dame;Laure Rittie;Suzanne E.G. Fligiel.
American Journal of Pathology (2006)
Vitamin A antagonizes decreased cell growth and elevated collagen-degrading matrix metalloproteinases and stimulates collagen accumulation in naturally aged human skin.
James Varani;Roscoe L. Warner;Mehrnaz Gharaee-Kermani;Sem H. Phan.
Journal of Investigative Dermatology (2000)
Sequence and Haplotype Analysis Supports HLA-C as the Psoriasis Susceptibility 1 Gene
Rajan P. Nair;Philip E. Stuart;Ioana Nistor;Ravi Hiremagalore.
American Journal of Human Genetics (2006)
Topical tretinoin improves photoaged skin. A double-blind vehicle-controlled study.
Jonathan S. Weiss;Charles N. Ellis;John T. Headington;Theresa Tincoff.
JAMA (1988)
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