2022 - Research.com Immunology in Germany Leader Award
2016 - German National Academy of Sciences Leopoldina - Deutsche Akademie der Naturforscher Leopoldina – Nationale Akademie der Wissenschaften Microbiology and Immunology
His primary scientific interests are in Inflammasome, Cell biology, Immunology, Innate immune system and Inflammation. In his research on the topic of Inflammasome, Regulation of gene expression is strongly related with In vivo. Eicke Latz has included themes like DNA, Receptor, Toll-like receptor and Immune system in his Cell biology study.
As a member of one scientific family, Eicke Latz mostly works in the field of Immunology, focusing on Disease and, on occasion, Reprogramming. He has researched Innate immune system in several fields, including Acquired immune system, Neuroscience and Immunity. His biological study deals with issues like Cholesterol, which deal with fields such as Transcriptional regulation.
His main research concerns Inflammasome, Cell biology, Immunology, Inflammation and Innate immune system. His research integrates issues of Interleukin, Programmed cell death and Microglia in his study of Inflammasome. His Cell biology research incorporates themes from Receptor, Toll-like receptor and Immune system.
His studies in Immunology integrate themes in fields like Cancer research and Disease. His work in Inflammation addresses subjects such as Fibrosis, which are connected to disciplines such as Kidney. Eicke Latz interconnects Macrophage, Cell, Neuroscience and In vivo in the investigation of issues within Innate immune system.
His primary areas of study are Inflammasome, Cell biology, Innate immune system, Inflammation and Immune system. Inflammasome is a subfield of Receptor that he tackles. His Cell biology research includes themes of Microvesicles, Internalization and Toll-like receptor.
The Innate immune system study combines topics in areas such as Macrophage and Neuroscience. His Inflammation research also covers Immunology and Internal medicine studies. His work on Proinflammatory cytokine, Immunity and AIM2 as part of general Immunology research is frequently linked to Dried blood, bridging the gap between disciplines.
Eicke Latz mostly deals with Inflammasome, Inflammation, Innate immune system, Immunology and Cell biology. His work deals with themes such as Gene family, Microglia, HEK 293 cells, Neuroinflammation and Proinflammatory cytokine, which intersect with Inflammasome. His studies in Inflammation integrate themes in fields like Endocrinology, Blood pressure, Renal function and Fibrosis, Renal fibrosis.
Eicke Latz combines subjects such as Platelet, Caspase 1 and Immunity with his study of Innate immune system. Eicke Latz studies Immune system, a branch of Immunology. Eicke Latz works mostly in the field of Cell biology, limiting it down to concerns involving Receptor and, occasionally, Humanized mouse, Cell and Extracellular.
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NLRP3 inflammasomes are required for atherogenesis and activated by cholesterol crystals
Peter Duewell;Hajime Kono;Katey J. Rayner;Katey J. Rayner;Cherilyn M. Sirois.
Neuroinflammation in Alzheimer's disease
Michael T Heneka;Monica J Carson;Joseph El Khoury;Gary E Landreth.
Lancet Neurology (2015)
IKKepsilon and TBK1 are essential components of the IRF3 signaling pathway.
Katherine A. Fitzgerald;Sarah M. McWhirter;Kerrie L. Faia;Daniel C. Rowe.
Nature Immunology (2003)
Silica crystals and aluminum salts activate the NALP3 inflammasome through phagosomal destabilization
Veit Hornung;Franz G. Bauernfeind;Annett Halle;Eivind O. Samstad;Eivind O. Samstad.
Nature Immunology (2008)
Activation and regulation of the inflammasomes
Eicke Latz;T. Sam Xiao;Andrea Stutz.
Nature Reviews Immunology (2013)
AIM2 recognizes cytosolic dsDNA and forms a caspase-1 activating inflammasome with ASC
Veit Hornung;Veit Hornung;Andrea Ablasser;Andrea Ablasser;Marie Charrel-Dennis;Franz G. Bauernfeind;Franz G. Bauernfeind.
The NALP3 inflammasome is involved in the innate immune response to amyloid-beta.
Annett Halle;Veit Hornung;Gabor C Petzold;Cameron R Stewart.
Nature Immunology (2008)
Cutting edge: NF-kappaB activating pattern recognition and cytokine receptors license NLRP3 inflammasome activation by regulating NLRP3 expression.
Franz G. Bauernfeind;Gabor Horvath;Andrea Stutz;Emad S. Alnemri.
Journal of Immunology (2009)
TLR9 signals after translocating from the ER to CpG DNA in the lysosome
Eicke Latz;Annett Schoenemeyer;Alberto Visintin;Katherine A. Fitzgerald.
Nature Immunology (2004)
NLRP3 is activated in Alzheimer´s disease and contributes to pathology in APP/PS1 mice
Michael T. Heneka;Markus P. Kummer;Andrea Stutz;Andrea Delekate.
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