1982 - Fellow of the American Association for the Advancement of Science (AAAS)
1973 - Fellow of John Simon Guggenheim Memorial Foundation
His main research concerns Biochemistry, Internal medicine, Cell biology, Inflammation and Enzyme. His research in Extracellular, Superoxide, Degranulation, Liposome and Zymosan are components of Biochemistry. In vivo is closely connected to Endocrinology in his research, which is encompassed under the umbrella topic of Internal medicine.
His work carried out in the field of Cell biology brings together such families of science as Mannose 6-phosphate receptor, Chemotaxis, Cell adhesion and Colchicine. His study with Inflammation involves better knowledge in Immunology. The various areas that Gerald Weissmann examines in his Enzyme study include Cytoplasm and Lysis.
Gerald Weissmann mainly investigates Biochemistry, Cell biology, Inflammation, Immunology and Enzyme. Gerald Weissmann works mostly in the field of Biochemistry, limiting it down to topics relating to Calcium and, in certain cases, Biophysics, as a part of the same area of interest. His studies in Cell biology integrate themes in fields like Receptor, Chemotaxis, Secretion and Cytochalasin.
As part of his studies on Inflammation, Gerald Weissmann frequently links adjacent subjects like Arthritis. His Immunology research focuses on Rheumatoid arthritis and Immune system. His research brings together the fields of Cytoplasm and Enzyme.
Immunology, Art history, Biochemistry, Classics and Rheumatoid arthritis are his primary areas of study. Immunology is closely attributed to Internal medicine in his study. His Art history study typically links adjacent topics like Environmental ethics.
Gerald Weissmann works in the field of Biochemistry, focusing on Hydrolase in particular. His research links Periodontitis with Rheumatoid arthritis. His Inflammation study incorporates themes from Pathogenesis and Antigen.
His primary areas of study are Immunology, Rheumatoid arthritis, Immune system, Classics and Inflammation. His studies deal with areas such as Internal medicine and Rheumatology as well as Immunology. In the field of Internal medicine, his study on Disease overlaps with subjects such as Polysubstance dependence.
His research integrates issues of Periodontitis, Porphyromonas gingivalis and Pathogenesis in his study of Rheumatoid arthritis. His Inflammation study integrates concerns from other disciplines, such as Disease free survival and Antigen. His Antigen study combines topics in areas such as Receptor, Anaphylatoxin and Monoclonal antibody.
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A mechanism for the antiinflammatory effects of corticosteroids: the glucocorticoid receptor regulates leukocyte adhesion to endothelial cells and expression of endothelial-leukocyte adhesion molecule 1 and intercellular adhesion molecule 1.
Cronstein Bn;Kimmel Sc;Levin Ri;Martiniuk F.
Proceedings of the National Academy of Sciences of the United States of America (1992)
Complement and immunoglobulins stimulate superoxide production by human leukocytes independently of phagocytosis.
I M Goldstein;D Roos;H B Kaplan;G Weissmann.
Journal of Clinical Investigation (1975)
Release of inflammatory mediators from stimulated neutrophils.
Gerald Weissmann;James E. Smolen;Helen M. Korchak.
The New England Journal of Medicine (1980)
Mechanisms of Lysosomal Enzyme Release from Human Leukocytes II. EFFECTS OF cAMP AND cGMP, AUTONOMIC AGONISTS, AND AGENTS WHICH AFFECT MICROTUBULE FUNCTION
Robert B. Zurier;Gerald Weissmann;Sylvia Hoffstein;Sandra Kammerman.
Journal of Clinical Investigation (1974)
The mechanisms of action of nonsteroidal antiinflammatory drugs.
Steven B. Abramson;Gerald Weissmann.
Arthritis & Rheumatism (1989)
Adenosine: a physiological modulator of superoxide anion generation by human neutrophils.
B N Cronstein;S B Kramer;G Weissmann;R Hirschhorn.
Journal of Experimental Medicine (1983)
The action of steroids and streptolysin S on the permeability of phospholipid structures to cations.
A.D. Bangham;M.M. Standish;G. Weissmann.
Journal of Molecular Biology (1965)
Mechanisms of lysosomal enzyme release from leukocytes exposed to immune complexes and other particles.
Gerald Weissmann;Robert B. Zurier;Paul J. Spieler;Ira M. Goldstein.
Journal of Experimental Medicine (1971)
Adenosine: an endogenous inhibitor of neutrophil-mediated injury to endothelial cells.
B N Cronstein;R I Levin;J Belanoff;G Weissmann.
Journal of Clinical Investigation (1986)
Phospholipid spherules (liposomes) as a model for biological membranes.
Grazia Sessa;Gerald Weissmann.
Journal of Lipid Research (1968)
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