D-Index & Metrics Best Publications

D-Index & Metrics D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines.

Discipline name D-index D-index (Discipline H-index) only includes papers and citation values for an examined discipline in contrast to General H-index which accounts for publications across all disciplines. Citations Publications World Ranking National Ranking
Microbiology D-index 96 Citations 27,082 292 World Ranking 290 National Ranking 15
Medicine D-index 104 Citations 31,862 400 World Ranking 4231 National Ranking 154

Overview

What is he best known for?

The fields of study he is best known for:

  • Gene
  • Enzyme
  • Internal medicine

His primary areas of study are Immunology, Molecular biology, Biochemistry, Cell biology and NADPH oxidase. His Immunology study combines topics in areas such as Endothelial stem cell and Genotype. His work deals with themes such as Platelet and Monoclonal antibody, which intersect with Molecular biology.

His Biochemistry research incorporates elements of Phagocytosis, Granulocyte and Histamine. His studies deal with areas such as Inflammation, Chemokine, Umbilical vein and Fluorescence microscope as well as Cell biology. His NADPH oxidase study combines topics from a wide range of disciplines, such as Phagocyte, Western blot, Superoxide and Chronic granulomatous disease.

His most cited work include:

  • FcγRIIIa-158V/F Polymorphism Influences the Binding of IgG by Natural Killer Cell FcγRIIIa, Independently of the FcγRIIIa-48L/R/H Phenotype (676 citations)
  • Complement and immunoglobulins stimulate superoxide production by human leukocytes independently of phagocytosis. (639 citations)
  • Chronic Granulomatous Disease: The European Experience (475 citations)

What are the main themes of his work throughout his whole career to date?

His main research concerns Immunology, Molecular biology, Biochemistry, Chronic granulomatous disease and NADPH oxidase. His Immunology research incorporates themes from Receptor, Internal medicine and Disease. His Molecular biology study incorporates themes from Cytochrome, Monoclonal antibody and Antigen.

The study incorporates disciplines such as Phagocytosis and Cell biology in addition to Biochemistry. The various areas that Dirk Roos examines in his Chronic granulomatous disease study include Mutation, CYBB, Nicotinamide adenine dinucleotide phosphate and X chromosome. His biological study deals with issues like Superoxide, which deal with fields such as Hydrogen peroxide.

He most often published in these fields:

  • Immunology (35.70%)
  • Molecular biology (23.34%)
  • Biochemistry (22.20%)

What were the highlights of his more recent work (between 2007-2021)?

  • Immunology (35.70%)
  • Chronic granulomatous disease (21.97%)
  • NADPH oxidase (18.31%)

In recent papers he was focusing on the following fields of study:

His primary areas of study are Immunology, Chronic granulomatous disease, NADPH oxidase, Genetics and Gene. His study looks at the relationship between Immunology and topics such as Disease, which overlap with Gene mutation. He has included themes like Mutation, Medical microbiology, Nicotinamide adenine dinucleotide phosphate, CYBB and Allele in his Chronic granulomatous disease study.

His NADPH oxidase study integrates concerns from other disciplines, such as Oxidase test, Phagocyte, Superoxide and Microbiology. As a part of the same scientific family, Dirk Roos mostly works in the field of Innate immune system, focusing on Chemokine receptor and, on occasion, Cell biology. His Reactive oxygen species study is concerned with the field of Biochemistry as a whole.

Between 2007 and 2021, his most popular works were:

  • Chronic Granulomatous Disease: The European Experience (475 citations)
  • Neutrophils: Between host defence, immune modulation, and tissue injury (329 citations)
  • Invasive fungal infection and impaired neutrophil killing in human CARD9 deficiency. (201 citations)

In his most recent research, the most cited papers focused on:

  • Gene
  • Enzyme
  • Internal medicine

Dirk Roos mainly focuses on Immunology, Chronic granulomatous disease, NADPH oxidase, Microbiology and Cell biology. His research on Immunology often connects related areas such as Candida albicans. Dirk Roos interconnects Inflammasome, Staphylococcus aureus, CYBB and Primary immunodeficiency, Disease in the investigation of issues within Chronic granulomatous disease.

His study in NADPH oxidase is interdisciplinary in nature, drawing from both Nicotinamide adenine dinucleotide phosphate, Oxidase test, Aspergillus fumigatus, Burkholderia and Superoxide. As part of one scientific family, Dirk Roos deals mainly with the area of Microbiology, narrowing it down to issues related to the Receptor, and often Laminarin and Zymosan. His study looks at the relationship between Cell biology and fields such as Chemokine receptor, as well as how they intersect with chemical problems.

This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.

Best Publications

FcγRIIIa-158V/F Polymorphism Influences the Binding of IgG by Natural Killer Cell FcγRIIIa, Independently of the FcγRIIIa-48L/R/H Phenotype

H. R. Koene;M. Kleijer;J. Algra;D. Roos.
Blood (1997)

1146 Citations

Complement and immunoglobulins stimulate superoxide production by human leukocytes independently of phagocytosis.

I M Goldstein;D Roos;H B Kaplan;G Weissmann.
Journal of Clinical Investigation (1975)

916 Citations

Chronic Granulomatous Disease: The European Experience

J. Merlijn van den Berg;Elsbeth van Koppen;Anders Åhlin;Bernd H. Belohradsky.
PLOS ONE (2009)

677 Citations

Neutrophils: Between host defence, immune modulation, and tissue injury

Philipp Kruger;Mona Saffarzadeh;Alexander N. R. Weber;Nikolaus Rieber.
PLOS Pathogens (2015)

613 Citations

Chronic granulomatous disease

D. Roos;J.T. Curnutte.
Primary immunodeficiency diseases, a molecular and genetic approach (1999)

574 Citations

Human neutrophils lose their surface Fc gamma RIII and acquire Annexin V binding sites during apoptosis in vitro

C. H. E. Homburg;M. De Haas;A. E. G. K. Von Dem Borne;A. J. Verhoeven.
Blood (1995)

572 Citations

The Pi-linked receptor FcRIII is released on stimulation of neutrophils

T. W. Huizinga;C. E. van der Schoot;C. Jost;R. Klaassen.
Nature (1988)

501 Citations

Membrane surface antigen expression on neutrophils: a reappraisal of the use of surface markers for neutrophil activation.

T.W. Kuijpers;A.T.J. Tool;C.E. van der Schoot;L.A. Ginsel.
Blood (1991)

490 Citations

Human neutrophil immunodeficiency syndrome is associated with an inhibitory Rac2 mutation

Daniel R. Ambruso;Cindy Knall;Amy N. Abell;Julie Panepinto.
Proceedings of the National Academy of Sciences of the United States of America (2000)

480 Citations

Mutations in the X-linked and autosomal recessive forms of chronic granulomatous disease

D Roos;M de Boer;F Kuribayashi;C Meischl.
Blood (1996)

480 Citations

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