2014 - Fellow of the American Association for the Advancement of Science (AAAS)
Member of the Association of American Physicians
His primary areas of study are NADPH oxidase, Molecular biology, Chronic granulomatous disease, Biochemistry and Oxidase test. He has included themes like Respiratory burst and Superoxide in his NADPH oxidase study. Mary C. Dinauer works mostly in the field of Superoxide, limiting it down to concerns involving Nitric oxide and, occasionally, Superoxide dismutase and Peroxynitrite.
The Molecular biology study combines topics in areas such as Complementary DNA, Gene and Mutant. His research on Chronic granulomatous disease concerns the broader Immunology. His Oxidase test research includes elements of Phagocyte and Potassium channel.
The scientist’s investigation covers issues in NADPH oxidase, Immunology, Chronic granulomatous disease, Molecular biology and Oxidase test. His NADPH oxidase research is multidisciplinary, relying on both Respiratory burst and Superoxide. His Immunology research integrates issues from Haematopoiesis, Stem cell and Transplantation.
His Chronic granulomatous disease research is multidisciplinary, incorporating perspectives in Aspergillus fumigatus, Phagocyte, CYBB and Genetic enhancement, Gene. His research integrates issues of Mutation, Cytochrome, Mutant and Transfection in his study of Molecular biology. His work deals with themes such as Phagosome and COS cells, which intersect with Oxidase test.
His main research concerns NADPH oxidase, Immunology, Chronic granulomatous disease, Inflammation and Oxidase test. His research in NADPH oxidase intersects with topics in Molecular biology, Macrophage and Superoxide. His work carried out in the field of Superoxide brings together such families of science as Respiratory burst, Extracellular and Siderophore.
His studies in Immunology integrate themes in fields like Systemic lupus erythematosus and Haematopoiesis. His Chronic granulomatous disease study integrates concerns from other disciplines, such as CYBB, Immune system, Myeloperoxidase and Granulocyte. His Oxidase test research includes elements of Zymosan, Reactive oxygen species, Epitope and Antigen presentation.
Mary C. Dinauer mainly focuses on Immunology, NADPH oxidase, Innate immune system, Respiratory burst and Chronic granulomatous disease. His Immunology research incorporates themes from Haematopoiesis, Streptococcus pneumoniae, Progenitor cell, Microbiology and Bacterial pneumonia. His NADPH oxidase research is classified as research in Cell biology.
His Cell biology research is multidisciplinary, relying on both Oxidase test and Biochemistry. Within one scientific family, he focuses on topics pertaining to Superoxide under Respiratory burst, and may sometimes address concerns connected to Phagosome, Chloride channel, Reactive oxygen species and Siderophore. His Chronic granulomatous disease research incorporates elements of Chédiak–Higashi syndrome, Chemotaxis, Hyperimmunoglobulin E syndrome and Myeloperoxidase.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
Mouse model of X-linked chronic granulomatous disease, an inherited defect in phagocyte superoxide production
Jonathan D. Pollock;David A. Williams;David A. Williams;Mary A.C. Gifford;Ling Lin Li.
Nature Genetics (1995)
Functional analysis of Nox4 reveals unique characteristics compared to other NADPH oxidases
Kendra D. Martyn;Linda M. Frederick;Katharina von Loehneysen;Mary C. Dinauer.
Cellular Signalling (2006)
Salmonella pathogenicity island 2-dependent evasion of the phagocyte NADPH oxidase.
Andrés Vazquez-Torres;Yisheng Xu;Jessica Jones-Carson;David W. Holden.
Science (2000)
Phenotype of mice and macrophages deficient in both phagocyte oxidase and inducible nitric oxide synthase.
Michael U Shiloh;John D MacMicking;Susan Nicholson;Juliet E Brause.
Immunity (1999)
Deficiency of the hematopoietic cell-specific Rho family GTPase Rac2 is characterized by abnormalities in neutrophil function and host defense.
Andrew W. Roberts;Chaekyun Kim;Ling Zhen;John B. Lowe.
Immunity (1999)
Activation of antibacterial autophagy by NADPH oxidases
Ju Huang;Veronica Canadien;Grace Y. Lam;Benjamin E. Steinberg.
Proceedings of the National Academy of Sciences of the United States of America (2009)
Absence of Respiratory Burst in X-linked Chronic Granulomatous Disease Mice Leads to Abnormalities in Both Host Defense and Inflammatory Response to Aspergillus fumigatus
David E. Morgenstern;Mary A.C. Gifford;Ling Lin Li;Claire M. Doerschuk.
Journal of Experimental Medicine (1997)
The glycoprotein encoded by the X-linked chronic granulomatous disease locus is a component of the neutrophil cytochrome b complex
Mary C. Dinauer;Stuart H. Orkin;Stuart H. Orkin;Robin Brown;Robin Brown;Algirdas J. Jesaitis.
Nature (1987)
Periplasmic superoxide dismutase protects Salmonella from products of phagocyte NADPH-oxidase and nitric oxide synthase
Mary Ann De Groote;Urs A. Ochsner;Michael U. Shiloh;Carl Nathan.
Proceedings of the National Academy of Sciences of the United States of America (1997)
Ischemic Stroke Injury Is Reduced in Mice Lacking a Functional NADPH Oxidase
Claire E. Walder;Simon P. Green;Walter C. Darbonne;Joanne Mathias.
Stroke (1997)
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