Ajay M. Shah focuses on Internal medicine, Endocrinology, NADPH oxidase, Reactive oxygen species and Cell biology. His research is interdisciplinary, bridging the disciplines of Cardiology and Internal medicine. His research in Endocrinology focuses on subjects like Endothelial stem cell, which are connected to Vascular disease.
The various areas that he examines in his NADPH oxidase study include Oxidase test and Molecular biology. His research integrates issues of Nicotinamide adenine dinucleotide phosphate, Cytochrome c and Superoxide, Enzyme in his study of Reactive oxygen species. His Cell biology research is multidisciplinary, incorporating elements of Oxidative phosphorylation and Antioxidant.
The scientist’s investigation covers issues in Internal medicine, Endocrinology, Cardiology, NADPH oxidase and Heart failure. His study in Nitric oxide, Endothelium, Angiotensin II, Endothelial dysfunction and Pressure overload is carried out as part of his Internal medicine studies. Insulin resistance, Muscle hypertrophy, Oxidative stress, Nitric oxide synthase and Myocyte are among the areas of Endocrinology where Ajay M. Shah concentrates his study.
In general Cardiology, his work in Ejection fraction and Myocardial infarction is often linked to In patient linking many areas of study. His biological study focuses on NOX4. His research ties Heart disease and Heart failure together.
Internal medicine, Heart failure, Cardiology, Emergency medicine and Cell biology are his primary areas of study. In his study, Volume overload is strongly linked to Endocrinology, which falls under the umbrella field of Internal medicine. His Heart failure study incorporates themes from Disease and Comorbidity.
As a part of the same scientific study, he usually deals with the Emergency medicine, concentrating on Cohort and frequently concerns with Incidence. His study in Cell biology concentrates on NADPH oxidase and Mitochondrion. As a part of the same scientific family, Ajay M. Shah mostly works in the field of Clinical endpoint, focusing on Diabetes mellitus and, on occasion, Intensive care unit.
Ajay M. Shah mainly investigates Internal medicine, Heart failure, Cell biology, Confidence interval and Emergency medicine. His study in Internal medicine is interdisciplinary in nature, drawing from both Endocrinology and Cardiology. His Endocrinology study combines topics from a wide range of disciplines, such as Exercise capacity and Exercise performance.
His Heart failure research incorporates themes from Immunology, Pathogenesis, Cohort, Energy metabolism and Comorbidity. The Cell biology study combines topics in areas such as Downregulation and upregulation and Calcium. His Confidence interval study integrates concerns from other disciplines, such as Severe disease, Retrospective cohort study and Cohort study.
This overview was generated by a machine learning system which analysed the scientist’s body of work. If you have any feedback, you can contact us here.
The effect of digoxin on mortality and morbidity in patients with heart failure
G Perry;E Brown;R Thornton;T Shiva.
The New England Journal of Medicine (1997)
Plasma MicroRNA Profiling Reveals Loss of Endothelial MiR-126 and Other MicroRNAs in Type 2 Diabetes
Anna Zampetaki;Stefan Kiechl;Ignat Drozdov;Peter Willeit.
Circulation Research (2010)
Endothelial cell superoxide generation: regulation and relevance for cardiovascular pathophysiology
Jian-Mei Li;Ajay M Shah.
American Journal of Physiology-regulatory Integrative and Comparative Physiology (2004)
Pivotal role of a gp91(phox)-containing NADPH oxidase in angiotensin II-induced cardiac hypertrophy in mice.
Jennifer K. Bendall;Alison C. Cave;Christophe Heymes;Nicholas Gall.
Circulation (2002)
NADPH oxidases in cardiovascular health and disease.
Alison C Cave;Alison C Brewer;Anilkumar Narayanapanicker;Robin Ray.
Antioxidants & Redox Signaling (2006)
Increased myocardial NADPH oxidase activity in human heart failure.
Christophe Heymes;Jennifer K Bendall;Philippe Ratajczak;Alison C Cave.
Journal of the American College of Cardiology (2003)
The oxygen-rich postnatal environment induces cardiomyocyte cell-cycle arrest through DNA damage response.
Bao N. Puente;Wataru Kimura;Shalini A. Muralidhar;Jesung Moon.
Cell (2014)
Nox4 Is a Protective Reactive Oxygen Species Generating Vascular NADPH Oxidase
Katrin Schröder;Min Zhang;Sebastian Benkhoff;Anja Mieth.
Circulation Research (2012)
Oxidative stress and redox signalling in cardiac hypertrophy and heart failure
Mike Seddon;Yee H Looi;Ajay M Shah.
Heart (2007)
Activation of NADPH Oxidase During Progression of Cardiac Hypertrophy to Failure
Jian-Mei Li;Nick P. Gall;David J. Grieve;Mingyou Chen.
Hypertension (2002)
If you think any of the details on this page are incorrect, let us know.
We appreciate your kind effort to assist us to improve this page, it would be helpful providing us with as much detail as possible in the text box below:
Goethe University Frankfurt
King's College London
Inserm
Innsbruck Medical University
University of Oxford
King's College London
University of Edinburgh
University of Göttingen
University of Manchester
Ludwig Maximilian University of Munich
Nanyang Technological University
University of Arkansas at Fayetteville
University of Siena
Kyoto University
Bhabha Atomic Research Centre
National Tsing Hua University
Zoological Society of London
American Museum of Natural History
University of Naples Federico II
Université Libre de Bruxelles
Pfizer (Canada)
Paul Sabatier University
Vanderbilt University Medical Center
Northwestern University
University Medical Center Groningen
University of California, Berkeley